A major advance in the use of growth factors to enhance wound healing.
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Lens-specific expression of transforming growth factor beta1 in transgenic mice causes anterior subcapsular cataractsRegulation of HEF1 expression and phosphorylation by TGF-beta 1 and cell adhesionTumor necrosis factor-alpha inhibits collagen alpha1(I) gene expression and wound healing in a murine model of cachexiaInduction of specific microRNAs inhibits cutaneous wound healingDermal transforming growth factor-beta responsiveness mediates wound contraction and epithelial closure.Attenuation of the transforming growth factor beta-signaling pathway in chronic venous ulcersThe Effect of Bone-Marrow-Derived Stem Cells and Adipose-Derived Stem Cells on Wound Contraction and Epithelization.Current therapies for wound healing: electrical stimulation, biological therapeutics, and the potential for gene therapy.Hepatocyte growth factor is the most potent endogenous stimulant of rabbit gastric epithelial cell proliferation and migration in primary culture.Biological approaches to improve skeletal muscle healing after injury and diseaseCalreticulin enhances porcine wound repair by diverse biological effects.Beyond good and evil in the oral cavity: insights into host-microbe relationships derived from transcriptional profiling of gingival cellsTransforming growth factor-beta - and tumor necrosis factor-alpha -mediated induction and proteolytic activation of MMP-9 in human skin.Prevention and treatment of pressure sores.Transforming growth factor-beta type-II receptor signalling: intrinsic/associated casein kinase activity, receptor interactions and functional effects of blocking antibodies.Vesical urothelium proliferation in spinal cord injured persons: an immunohistochemical study of PCNA and MIB.1 labelling.Role of IL-17 and TGF-β in peritoneal adhesion formation after surgical trauma.Fibrosis linked to TGF-beta in yet another disease.TGF-beta signaling promotes survival and repair in rat alveolar epithelial type 2 cells during recovery after hyperoxic injury.The heparan sulphate deficient Hspg2 exon 3 null mouse displays reduced deposition of TGF-β1 in skin compared to C57BL/6 wild type mice.Transforming growth factor-beta isoforms differently stimulate proalpha2 (I) collagen gene expression during wound healing process in transgenic mice.
P2860
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P2860
A major advance in the use of growth factors to enhance wound healing.
description
1993 nî lūn-bûn
@nan
1993 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
A major advance in the use of growth factors to enhance wound healing.
@ast
A major advance in the use of growth factors to enhance wound healing.
@en
type
label
A major advance in the use of growth factors to enhance wound healing.
@ast
A major advance in the use of growth factors to enhance wound healing.
@en
prefLabel
A major advance in the use of growth factors to enhance wound healing.
@ast
A major advance in the use of growth factors to enhance wound healing.
@en
P2860
P356
P1476
A major advance in the use of growth factors to enhance wound healing.
@en
P2093
P2860
P304
P356
10.1172/JCI116868
P407
P577
1993-12-01T00:00:00Z