Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea. - Wikidata - awgldk - TriplyDB

Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea.

Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea.

http://www.wikidata.org/entity/Q33916216

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Role of Oxidative Stress in the Neurocognitive Dysfunction of Obstructive Sleep Apnea Syndrome Emerging co-morbidities of obstructive sleep apnea: cognition, kidney disease, and cancer Disrupted sleep without sleep curtailment induces sleepiness and cognitive dysfunction via the tumor necrosis factor-α pathway Chronic intermittent hypoxia-induced deficits in synaptic plasticity and neurocognitive functions: a role for brain-derived neurotrophic factor.Resveratrol attenuates intermittent hypoxia-induced macrophage migration to visceral white adipose tissue and insulin resistance in male mice.New insights on NOX enzymes in the central nervous system.Inflammatory markers and obstructive sleep apnea in obese children: the NANOS study.Sleep apnea: a redox edge with aging?The polymorphic and contradictory aspects of intermittent hypoxia Targeting NOX enzymes in the central nervous system: therapeutic opportunities.Exogenous erythropoietin administration attenuates intermittent hypoxia-induced cognitive deficits in a murine model of sleep apnea Long-term intermittent hypoxia elevates cobalt levels in the brain and injures white matter in adult mice.Chronic intermittent hypoxia increases rat sternohyoid muscle NADPH oxidase expression with attendant modest oxidative stress.Chronic intermittent hypoxia exerts CNS region-specific effects on rat microglial inflammatory and TLR4 gene expression.Influence of gonadal hormones on the behavioral effects of intermittent hypoxia in mice.Cognitive function in prepubertal children with obstructive sleep apnea: a modifying role for NADPH oxidase p22 subunit gene polymorphisms?Sleep fragmentation induces cognitive deficits via nicotinamide adenine dinucleotide phosphate oxidase-dependent pathways in mouse.The link between angiotensin II-mediated anxiety and mood disorders with NADPH oxidase-induced oxidative stress.Glucoregulatory consequences and cardiorespiratory parameters in rats exposed to chronic-intermittent hypoxia: effects of the duration of exposure and losartan.Human apolipoprotein E4 targeted replacement in mice reveals increased susceptibility to sleep disruption and intermittent hypoxia.Intermittent hypoxia causes NOX2-dependent remodeling of atrial connexins.Cerebrovascular consequences of obstructive sleep apnea P2X7 Receptor Antagonism Attenuates the Intermittent Hypoxia-induced Spatial Deficits in a Murine Model of Sleep Apnea Via Inhibiting Neuroinflammation and Oxidative Stress.Making sense of oxidative stress in obstructive sleep apnea: mediator or distracter?Adverse cognitive effects of high-fat diet in a murine model of sleep apnea are mediated by NADPH oxidase activity Pathogenesis of cognitive dysfunction in patients with obstructive sleep apnea: a hypothesis with emphasis on the nucleus tractus solitarius Which NADPH oxidase isoform is relevant for ischemic stroke? The case for nox 2.Cognition and biomarkers of oxidative stress in obstructive sleep apnea Association of sleep disordered breathing and cognitive deficit in APOE ε4 carriers.Urinary neurotransmitters are selectively altered in children with obstructive sleep apnea and predict cognitive morbidity Pathophysiologic mechanisms of cardiovascular disease in obstructive sleep apnea syndrome GRK5 deficiency leads to susceptibility to intermittent hypoxia-induced cognitive impairment.Growth hormone releasing hormone (GHRH) signaling modulates intermittent hypoxia-induced oxidative stress and cognitive deficits in mouse.Oxidative stress and psychological disorders.Protective effect of 3-(naphthalen-2-yl(propoxy)methyl)azetidine hydrochloride on hypoxia-induced toxicity by suppressing microglial activation in BV-2 cells.Sleep fragmentation in mice induces nicotinamide adenine dinucleotide phosphate oxidase 2-dependent mobilization, proliferation, and differentiation of adipocyte progenitors in visceral white adipose tissue.Dysfunctional nucleus tractus solitarius: its crucial role in promoting neuropathogenetic cascade of Alzheimer's dementia--a novel hypothesis.Death by a thousand cuts in Alzheimer's disease: hypoxia--the prodrome.Obstructive sleep apnea and delirium: exploring possible mechanisms.Chronic intermittent hypoxia and hypertension: a review of systemic inflammation and Chinese medicine.

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Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea.

http://www.wikidata.org/entity/Q33916216

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Ehab A Dayyat

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Shelley X Zhang

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Yang Wang

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P2860

Recombinant 47-kilodalton cytosol factor restores NADPH oxidase in chronic granulomatous disease

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

Chemistry and biochemistry of 4-hydroxynonenal, malonaldehyde and related aldehydes

Cytochrome b-558 alpha-subunit cloning and expression in rat aortic smooth muscle cells

The p47phox mouse knock-out model of chronic granulomatous disease

Impaired spatial working memory and altered choline acetyltransferase (CHAT) immunoreactivity and nicotinic receptor binding in rats exposed to intermittent hypoxia during sleep

T cells express a phagocyte-type NADPH oxidase that is activated after T cell receptor stimulation

Developments of a water-maze procedure for studying spatial learning in the rat

Validation of open:closed arm entries in an elevated plus-maze as a measure of anxiety in the rat

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