Detrusor overactivity is associated with downregulation of large-conductance calcium- and voltage-activated potassium channel protein. - Wikidata - awgldk - TriplyDB

Detrusor overactivity is associated with downregulation of large-conductance calcium- and voltage-activated potassium channel protein.

Detrusor overactivity is associated with downregulation of large-conductance calcium- and voltage-activated potassium channel protein.

http://www.wikidata.org/entity/Q33917816

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Regulation of urinary bladder function by protein kinase C in physiology and pathophysiology Research Findings on Overactive Bladder Optogenetic Modulation of Urinary Bladder Contraction for Lower Urinary Tract Dysfunction Increased detrusor collagen is associated with detrusor overactivity and decreased bladder compliance in men with benign prostatic obstruction.Large-conductance voltage- and Ca2+-activated K+ channel regulation by protein kinase C in guinea pig urinary bladder smooth muscle.Role of potassium ion channels in detrusor smooth muscle function and dysfunction The regulation of BK channel activity by pre- and post-translational modifications.Central role of the BK channel in urinary bladder smooth muscle physiology and pathophysiology.BK channel activators and their therapeutic perspectives Neurogenic detrusor overactivity is associated with decreased expression and function of the large conductance voltage- and Ca(2+)-activated K(+) channels Molecular mechanisms underlying the effect of the novel BK channel opener GoSlo: involvement of the S4/S5 linker and the S6 segment.Large-conductance voltage- and Ca2+-activated K+ channels regulate human detrusor smooth muscle function Large conductance Ca2+ -activated K+ channel activation with NS1619 decreases myogenic and neurogenic contractions of rat detrusor smooth muscle Protein kinase C modulates frequency of micturition and non-voiding contractions in the urinary bladder via neuronal and myogenic mechanisms.Effects of the novel BK (KCa 1.1) channel opener GoSlo-SR-5-130 are dependent on the presence of BKβ subunits.Suppression of human detrusor smooth muscle excitability and contractility via pharmacological activation of large conductance Ca2+-activated K+ channels.SK but not IK channels regulate human detrusor smooth muscle spontaneous and nerve-evoked contractions.Selective inhibition of phosphodiesterase 1 relaxes urinary bladder smooth muscle: role for ryanodine receptor-mediated BK channel activation.Constitutively active phosphodiesterase activity regulates urinary bladder smooth muscle function: critical role of KCa1.1 channel.Bladder smooth muscle organ culture preparation maintains the contractile phenotype.Spontaneous and evoked contractions are regulated by PKC-mediated signaling in detrusor smooth muscle: involvement of BK channels.GATA-6 and NF-κB activate CPI-17 gene transcription and regulate Ca2+ sensitization of smooth muscle contraction Single-channel biophysical and pharmacological characterizations of native human large-conductance calcium-activated potassium channels in freshly isolated detrusor smooth muscle cells.Functional BK channels facilitate the β3-adrenoceptor agonist-mediated relaxation of nerve-evoked contractions in rat urinary bladder smooth muscle isolated strips.Big-conductance Ca2+-activated K+ channels in physiological and pathophysiological urinary bladder smooth muscle cells.Functional effects of KCNQ K(+) channels in airway smooth muscle Original Research: Combined model of bladder detrusor smooth muscle and interstitial cells.Impact of partial urethral obstruction on bladder function: time-dependent changes and functional correlates of altered expression of Ca²⁺ signaling regulators.Inosine attenuates spontaneous activity in the rat neurogenic bladder through an A2B pathway.A Chinese Medicine Formula "Xian-Jia-Tang" for Treating Bladder Outlet Obstruction by Improving Urodynamics and Inhibiting Oxidative Stress through Potassium Channels.Large-conductance calcium-activated potassium channel activity, as determined by whole-cell patch clamp recording, is decreased in urinary bladder smooth muscle cells from male rats with partial urethral obstruction.Partial bladder outlet obstruction is associated with decreased expression and function of the small-conductance Ca2+-activated K+ channel in guinea pig detrusor smooth muscle.Effects of Rho-kinase inhibition on myosin light chain phosphorylation and obstruction-induced detrusor overactivity.Urothelial MaxiK-activity regulates mucosal and detrusor metabolism.Spontaneous Contractile Activity of the Detrusor Muscle and Its Role in the Pathogenesis of Overactive Bladder Syndrome.

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Detrusor overactivity is associated with downregulation of large-conductance calcium- and voltage-activated potassium channel protein.

http://www.wikidata.org/entity/Q33917816

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年学术文章

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2010年学术文章

@zh-cn

2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Detrusor overactivity is assoc ...... ted potassium channel protein.

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Detrusor overactivity is assoc ...... ted potassium channel protein.

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Detrusor overactivity is assoc ...... ted potassium channel protein.

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Detrusor overactivity is assoc ...... ted potassium channel protein.

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AJPRENAL.00595.2009

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American Journal of Physiology - Renal Physiology

P1476

Detrusor overactivity is assoc ...... ted potassium channel protein.

@en

P2093

Bruce Malkowicz

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Cristiano Mendes Gomes

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Jaber Alanzi

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James Marx

http://www.w3.org/2001/XMLSchema#string

Joseph A Hypolite

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Samuel Chacko

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Shaohua Chang

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Stephen A Zderic

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P2860

Urodynamic properties and neurotransmitter dependence of urinary bladder contractility in the BK channel deletion model of overactive bladder

Overactive bladder and incontinence in the absence of the BK large conductance Ca2+-activated K+ channel

Ca2+ sensitivity of smooth muscle and nonmuscle myosin II: modulated by G proteins, kinases, and myosin phosphatase

Signal transduction and regulation in smooth muscle.

Altered expression of calcium-activated K and Cl channels in detrusor overactivity of rats with partial bladder outlet obstruction.

Storage and voiding symptoms: pathophysiologic aspects.

Alteration of contractile and regulatory proteins following partial bladder outlet obstruction.

Effect of bladder outlet obstruction on the morphology, physiology, and pharmacology of the bladder.

Smooth muscle hypertrophy following partial bladder outlet obstruction is associated with overexpression of non-muscle caldesmon.

Effect of partial outlet obstruction on rabbit urinary bladder smooth muscle function.

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