Vehicular emissions induce vascular MMP-9 expression and activity associated with endothelin-1-mediated pathways - Wikidata - awgldk - TriplyDB

Vehicular emissions induce vascular MMP-9 expression and activity associated with endothelin-1-mediated pathways

Vehicular emissions induce vascular MMP-9 expression and activity associated with endothelin-1-mediated pathways

http://www.wikidata.org/entity/Q33921009

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Nanoparticle inhalation impairs coronary microvascular reactivity via a local reactive oxygen species-dependent mechanism MMP-9-dependent serum-borne bioactivity caused by multiwalled carbon nanotube exposure induces vascular dysfunction via the CD36 scavenger receptor Effects of tempol and redox-cycling nitroxides in models of oxidative stress.Air particulate matter and cardiovascular disease: the epidemiological, biomedical and clinical evidence Mechanisms linking traffic-related air pollution and atherosclerosis Air Pollution-Induced Vascular Dysfunction: Potential Role of Endothelin-1 (ET-1) System Comparative effects of inhaled diesel exhaust and ambient fine particles on inflammation, atherosclerosis, and vascular dysfunction The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis Particulate matter and atherosclerosis: role of particle size, composition and oxidative stress Engine exhaust particulate and gas phase contributions to vascular toxicity Inhaled diesel emissions alter atherosclerotic plaque composition in ApoE(-/-) mice Formation of vascular S-nitrosothiols and plasma nitrates/nitrites following inhalation of diesel emissions Metal rich particulate matter impairs acetylcholine-mediated vasorelaxation of microvessels in mice Serum-borne bioactivity caused by pulmonary multiwalled carbon nanotubes induces neuroinflammation via blood-brain barrier impairment Biokinetically-based in vitro cardiotoxicity of residual oil fly ash: hazard identification and mechanisms of injury Environmental stressors and cardio-metabolic disease: part II-mechanistic insights.A comparison of vascular effects from complex and individual air pollutants indicates a role for monoxide gases and volatile hydrocarbons.Ultrastructural changes in atherosclerotic plaques following the instillation of airborne particulate matter into the lungs of rabbits.Altered gene transcription in human cells treated with Ludox® silica nanoparticles.Milano summer particulate matter (PM10) triggers lung inflammation and extra pulmonary adverse events in mice.Novel association between plasma matrix metalloproteinase-9 and risk of incident atrial fibrillation in a case-cohort study: the Atherosclerosis Risk in Communities study HIV Tat induces expression of ICAM-1 in HUVECs: implications for miR-221/-222 in HIV-associated cardiomyopathy.The oxidized low-density lipoprotein receptor mediates vascular effects of inhaled vehicle emissions Human immunodeficiency virus transgenic rats exhibit pulmonary hypertension Acrolein inhalation prevents vascular endothelial growth factor-induced mobilization of Flk-1+/Sca-1+ cells in mice.The mdx Mutation in the 129/Sv Background Results in a Milder Phenotype: Transcriptome Comparative Analysis Searching for the Protective Factors c-Src-dependent EGF receptor transactivation contributes to ET-1-induced COX-2 expression in brain microvascular endothelial cells Circulating levels of matrix metalloproteinase-9 and abdominal aortic pathology: from the Dallas Heart Study.Up-regulation of COX-2/PGE2 by endothelin-1 via MAPK-dependent NF-κB pathway in mouse brain microvascular endothelial cells Endothelial inflammatory transcriptional responses to an altered plasma exposome following inhalation of diesel emissions Hypercholesterolemia potentiates aortic endothelial response to inhaled diesel exhaust Identification of chemical components of combustion emissions that affect pro-atherosclerotic vascular responses in mice.MMP9 Rs3918242 Polymorphism Affects Tachycardia-Induced MMP9 Expression in Cultured Atrial-Derived Myocytes but Is Not a Risk Factor for Atrial Fibrillation among the Taiwanese How Can We Protect Susceptible Individuals from the Adverse Cardiovascular Effects of Air Pollution?Ischaemic heart disease mortality and years of work in trucking industry workers Metabolomic changes in murine serum following inhalation exposure to gasoline and diesel engine emissions.Impairment of coronary endothelial cell ET(B) receptor function after short-term inhalation exposure to whole diesel emissions.Exposure to vehicle emissions results in altered blood brain barrier permeability and expression of matrix metalloproteinases and tight junction proteins in mice.Surface area-dependence of gas-particle interactions influences pulmonary and neuroinflammatory outcomes.Changes in traffic exposure and the risk of incident myocardial infarction and all-cause mortality.

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P2860

Vehicular emissions induce vascular MMP-9 expression and activity associated with endothelin-1-mediated pathways

http://www.wikidata.org/entity/Q33921009

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2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009 թվականի հունվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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Vehicular emissions induce vas ...... endothelin-1-mediated pathways

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Vehicular emissions induce vas ...... endothelin-1-mediated pathways

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Vehicular emissions induce vas ...... endothelin-1-mediated pathways

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Jacob D McDonald

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Jean-Clare Seagrave

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JoAnn Lucero

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Matthew J Campen

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Michael C Madden

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Selitá Lucas

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Travis L Knuckles

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P2860

Cardiovascular mortality and long-term exposure to particulate air pollution: epidemiological evidence of general pathophysiological pathways of disease

NAD(P)H oxidase: role in cardiovascular biology and disease

Exposure to traffic and the onset of myocardial infarction

Long-term air pollution exposure and acceleration of atherosclerosis and vascular inflammation in an animal model

Particulate air pollution induces progression of atherosclerosis

Ischemic and thrombotic effects of dilute diesel-exhaust inhalation in men with coronary heart disease

Matrix metalloproteinases in vascular remodeling and atherogenesis: the good, the bad, and the ugly.

Dual role of matrix metalloproteinases (matrixins) in intimal thickening and atherosclerotic plaque rupture.

Reactive oxygen species produced by macrophage-derived foam cells regulate the activity of vascular matrix metalloproteinases in vitro. Implications for atherosclerotic plaque stability.

Respiratory damage in children exposed to urban pollution.

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