Shutoff of HeLa cell protein synthesis by encephalomyocarditis virus and poliovirus: a comparative study.
about
Functional dissection of eukaryotic initiation factor 4F: the 4A subunit and the central domain of the 4G subunit are sufficient to mediate internal entry of 43S preinitiation complexesThe encephalomyocarditis virusEncephalomyocarditis virus 2A protein is required for viral pathogenesis and inhibition of apoptosisPresence of the cap-binding protein in initiation factor preparations from poliovirus-infected HeLa cellsTranslational elongation rate changes in encephalomyocarditis virus-infected and interferon-treated cellsInactivation of cap-binding proteins accompanies the shut-off of host protein synthesis by poliovirus.Cardiovirus 2A protein associates with 40S but not 80S ribosome subunits during infection.Translational control of protein synthesis after infection by vesicular stomatitis virus.Clinical and experimental aspects of viral myocarditis.Selection of an attenuated Coxsackievirus B3 variant, using a monoclonal antibody reactive to myocyte antigen.Evidence for the presence of an inhibitor on ribosomes in mouse L cells infected with mengovirus.Translational efficiency of poliovirus mRNA: mapping inhibitory cis-acting elements within the 5' noncoding regionLeader protein of foot-and-mouth disease virus is required for cleavage of the p220 component of the cap-binding protein complexShutoff of host translation by encephalomyocarditis virus infection does not involve cleavage of the eucaryotic initiation factor 4F polypeptide that accompanies poliovirus infectionInhibition of host translation in encephalomyocarditis virus-infected L cells: a novel mechanism.Poliovirus mutant that does not selectively inhibit host cell protein synthesis.Translation of polioviral mRNA is inhibited by cleavage of polypyrimidine tract-binding proteins executed by polioviral 3C(pro)Reversion by hypotonic medium of the shutoff of protein synthesis induced by encephalomyocarditis virus.Encephalomyocarditis viral RNA can be translated under conditions of poliovirus-induced translation shutoff in vivo.Eukaryotic translation initiation factor 4E availability controls the switch between cap-dependent and internal ribosomal entry site-mediated translation.Augmentation of pathogenesis of coxsackievirus B3 infections in mice by exogenous administration of interleukin-1 and interleukin-2.E6AP/UBE3A catalyzes encephalomyocarditis virus 3C protease polyubiquitylation and promotes its concentration reduction in virus-infected cells.VPg unlinkase/TDP2 in cardiovirus infected cells: Re-localization and proteolytic cleavage.Engineered Oncolytic Poliovirus PVSRIPO Subverts MDA5-Dependent Innate Immune Responses in Cancer Cells
P2860
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P2860
Shutoff of HeLa cell protein synthesis by encephalomyocarditis virus and poliovirus: a comparative study.
description
1980 nî lūn-bûn
@nan
1980 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1980 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1980年の論文
@ja
1980年論文
@yue
1980年論文
@zh-hant
1980年論文
@zh-hk
1980年論文
@zh-mo
1980年論文
@zh-tw
1980年论文
@wuu
name
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@ast
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@en
type
label
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@ast
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@en
prefLabel
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@ast
Shutoff of HeLa cell protein s ...... liovirus: a comparative study.
@en
P2093
P2860
P1433
P1476
Shutoff of HeLa cell protein s ...... oliovirus: a comparative study
@en
P2093
P2860
P304
P407
P577
1980-07-01T00:00:00Z