Unraveling a role for dopamine in Huntington's disease: the dual role of reactive oxygen species and D2 receptor stimulation - Wikidata - awgldk - TriplyDB

Unraveling a role for dopamine in Huntington's disease: the dual role of reactive oxygen species and D2 receptor stimulation

Unraveling a role for dopamine in Huntington's disease: the dual role of reactive oxygen species and D2 receptor stimulation

http://www.wikidata.org/entity/Q33922831

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Modulation of the glutamatergic transmission by Dopamine: a focus on Parkinson, Huntington and Addiction diseases Possible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's disease Multiple phenotypes in Huntington disease mouse neural stem cells.Conformational targeting of fibrillar polyglutamine proteins in live cells escalates aggregation and cytotoxicity.Dopamine D2 receptor stimulation potentiates PolyQ-Huntingtin-induced mouse striatal neuron dysfunctions via Rho/ROCK-II activation Seeking brain biomarkers for preventive therapy in Huntington disease.Tetrabenazine is neuroprotective in Huntington's disease mice.Differential vulnerability of neurons in Huntington's disease: the role of cell type-specific features.Proteostasis in striatal cells and selective neurodegeneration in Huntington's disease.Neuroprotective potential of CB1 receptor agonists in an in vitro model of Huntington's disease.Renal dopamine receptors, oxidative stress, and hypertension.Loss of the thyroid hormone-binding protein Crym renders striatal neurons more vulnerable to mutant huntingtin in Huntington's disease.Huntington's Disease and Striatal Signaling.Dopaminergic signaling and striatal neurodegeneration in Huntington's disease.COMT Val158Met Polymorphism Modulates Huntington's Disease Progression.CYP46A1, the rate-limiting enzyme for cholesterol degradation, is neuroprotective in Huntington's disease.Hypothalamic dysfunction and neuroendocrine and metabolic alterations in Huntington's disease: clinical consequences and therapeutic implications.MAP kinase phosphatase 1 (MKP-1/DUSP1) is neuroprotective in Huntington's disease via additive effects of JNK and p38 inhibition Cellular responses to reactive oxygen species-induced DNA damage and aging.The injured nervous system: a Darwinian perspective.Protein aggregates in Huntington's disease.White matter predicts functional connectivity in premanifest Huntington's disease.Antioxidant gene therapy against neuronal cell death.Dopamine D2 receptor relies upon PPM/PP2C protein phosphatases to dephosphorylate huntingtin protein Molecular mechanisms and potential therapeutical targets in Huntington's disease.Huntington's disease and Group I metabotropic glutamate receptors.Screening of therapeutic strategies for Huntington's disease in YAC128 transgenic mice.Dopamine imbalance in Huntington's disease: a mechanism for the lack of behavioral flexibility.Neuroprotective effects of psychotropic drugs in Huntington's disease The role of dopamine in Huntington's disease.Dopamine and Huntington's disease.Corticostriatal circuit dysfunction in Huntington's disease: intersection of glutamate, dopamine and calcium.Ventricular fibrillation cardiac arrest produces a chronic striatal hyperdopaminergic state that is worsened by methylphenidate treatment.Coupling of D2R Short but not D2R Long receptor isoform to the Rho/ROCK signaling pathway renders striatal neurons vulnerable to mutant huntingtin.Hydrogen generation by reaction of Si nanopowder with neutral water.Dopamine determines the vulnerability of striatal neurons to the N-terminal fragment of mutant huntingtin through the regulation of mitochondrial complex II.Adult neural progenitor cells from Huntington's disease mouse brain exhibit increased proliferation and migration due to enhanced calcium and ROS signals.Improvement of mitochondrial function by paliperidone attenuates quinolinic acid-induced behavioural and neurochemical alterations in rats: implications in Huntington's disease.Dopamine induces platelet production from megakaryocytes via oxidative stress-mediated signaling pathways.Mechanisms involved in the formation of dopamine-induced intracellular bodies within striatal neurons.

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Unraveling a role for dopamine in Huntington's disease: the dual role of reactive oxygen species and D2 receptor stimulation

http://www.wikidata.org/entity/Q33922831

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2005 nî lūn-bûn

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2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2005年の論文

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2005年論文

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2005年論文

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Unraveling a role for dopamine ...... es and D2 receptor stimulation

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Unraveling a role for dopamine ...... es and D2 receptor stimulation

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Unraveling a role for dopamine ...... es and D2 receptor stimulation

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Christiane Pagès

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Delphine Charvin

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Emilliana Borrelli

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Jocelyne Caboche

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Peter Vanhoutte

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P2860

Activation of MLK2-mediated signaling cascades by polyglutamine-expanded huntingtin

Dopamine receptors: from structure to function

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death

Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats

Parkinsonian-like locomotor impairment in mice lacking dopamine D2 receptors

Early motor dysfunction and striosomal distribution of huntingtin microaggregates in Huntington's disease knock-in mice

Inactivation of Hdh in the brain and testis results in progressive neurodegeneration and sterility in mice

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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16103364

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Huntington disease

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1189314

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