Molecular mechanisms in down-regulation of tumor necrosis factor expression. - Wikidata - awgldk - TriplyDB

Molecular mechanisms in down-regulation of tumor necrosis factor expression.

Molecular mechanisms in down-regulation of tumor necrosis factor expression.

http://www.wikidata.org/entity/Q33923066

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Lipopolysaccharide (LPS) partial structures inhibit responses to LPS in a human macrophage cell line without inhibiting LPS uptake by a CD14-mediated pathway Dissociation of lipopolysaccharide (LPS)-inducible gene expression in murine macrophages pretreated with smooth LPS versus monophosphoryl lipid A.Tolerance to endotoxin-induced expression of the interleukin-1 beta gene in blood neutrophils of humans with the sepsis syndrome.Induction of cytokine synthesis by flagella from gram-negative bacteria may be dependent on the activation or differentiation state of human monocytes.HLA-B27-transfected (Salmonella permissive) and HLA-A2-transfected (Salmonella nonpermissive) human monocytic U937 cells differ in their production of cytokines.S-adenosylmethionine decreases lipopolysaccharide-induced phosphodiesterase 4B2 and attenuates tumor necrosis factor expression via cAMP/protein kinase A pathway.p38 MAP kinase and MKK-1 co-operate in the generation of GM-CSF from LPS-stimulated human monocytes by an NF-kappa B-independent mechanism.Bacterial evasion of host immune defense: Yersinia enterocolitica encodes a suppressor for tumor necrosis factor alpha expression.Human macrophages acquire a hyporesponsive state of tumor necrosis factor alpha production in response to successive Mycobacterium avium serovar 4 stimulation Low-dose lipopolysaccharide (LPS) pretreatment of mouse macrophages modulates LPS-dependent interleukin-6 production in vitro.G Protein-coupled pH-sensing Receptor OGR1 Is a Regulator of Intestinal Inflammation Lipopolysaccharide-mediated transcriptional activation of the human tissue factor gene in THP-1 monocytic cells requires both activator protein 1 and nuclear factor kappa B binding sites.HCV-specific cytokine induction in monocytes of patients with different outcomes of hepatitis C.Misoprostol modulates cytokine expression through a cAMP pathway: Potential therapeutic implication for liver disease.Lipopolysaccharide-induced selective priming effects on tumor necrosis factor alpha and nitric oxide production in mouse peritoneal macrophages.A labile transcriptional repressor modulates endotoxin tolerance Proteasomal degradation of Nod2 protein mediates tolerance to bacterial cell wall components.Downregulation of CD40 ligand response in monocytes from sepsis patients.miR-146a is critical for endotoxin-induced tolerance: IMPLICATION IN INNATE IMMUNITY.TLR signaling in human antigen-presenting cells regulates MR1-dependent activation of MAIT cells.Constitutive nuclear NF-kappa B in cells of the monocyte lineage.Complex NF-kappaB interactions at the distal tumor necrosis factor promoter region in human monocytes.NF-kappaB1 (p50) is upregulated in lipopolysaccharide tolerance and can block tumor necrosis factor gene expression.Low endotoxic potential of Legionella pneumophila lipopolysaccharide due to failure of interaction with the monocyte lipopolysaccharide receptor CD14.Prevention of experimental endotoxin shock by a monocyte activator.Protection against lethal pneumococcal septicemia in pigs is associated with decreased levels of interleukin-6 in blood.Proteolytic processing of NF-kappa B/I kappa B in human monocytes. ATP-dependent induction by pro-inflammatory mediators.Repression of class II major histocompatibility complex genes by cyclic AMP is mediated by conserved promoter elements.Mechanism of endotoxin desensitization: involvement of interleukin 10 and transforming growth factor beta.Enhanced PDE4B expression augments LPS-inducible TNF expression in ethanol-primed monocytes: relevance to alcoholic liver disease Multiple mechanisms of reduced major histocompatibility complex class II expression in endotoxin tolerance.

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P2860

Molecular mechanisms in down-regulation of tumor necrosis factor expression.

http://www.wikidata.org/entity/Q33923066

description

1990 nî lūn-bûn

@nan

1990 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1990 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1990年の論文

@ja

1990年論文

@yue

1990年論文

@zh-hant

1990年論文

@zh-hk

1990年論文

@zh-mo

1990年論文

@zh-tw

1990年论文

@wuu

name

Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Proceedings of the National Academy of Sciences of the United States of America

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Molecular mechanisms in down-regulation of tumor necrosis factor expression.

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Baeuerle PA

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Haas JG

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Riethmüller G

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Ziegler-Heitbrock HW

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P2860

An endotoxin-induced serum factor that causes necrosis of tumors

Human lymphotoxin and tumor necrosis factor genes: structure, homology and chromosomal localization

Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease

A technique for radiolabeling DNA restriction endonuclease fragments to high specific activity

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

Activation of DNA-binding activity in an apparently cytoplasmic precursor of the NF-kappa B transcription factor

Regulation of tumor necrosis factor alpha transcription in macrophages: involvement of four kappa B-like motifs and of constitutive and inducible forms of NF-kappa B

CR3 (CD11b/CD18) expresses one binding site for Arg-Gly-Asp-containing peptides and a second site for bacterial lipopolysaccharide

Eukaryotic transcriptional regulatory proteins

Shock and tissue injury induced by recombinant human cachectin.

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