Inhibition of Mac-1 (CD11b/CD18) enhances tumor response to radiation by reducing myeloid cell recruitment - Wikidata - awgldk - TriplyDB

Inhibition of Mac-1 (CD11b/CD18) enhances tumor response to radiation by reducing myeloid cell recruitment

Inhibition of Mac-1 (CD11b/CD18) enhances tumor response to radiation by reducing myeloid cell recruitment

http://www.wikidata.org/entity/Q33929607

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Host immune cellular reactions in corneal neovascularization The impact of the myeloid response to radiation therapy Radiation for Awakening the Dormant Immune System, a Promising Challenge to be Explored Tumor endothelial cells Vasculogenesis: a crucial player in the resistance of solid tumours to radiotherapy Role of immune escape mechanisms in Hodgkin's lymphoma development and progression: a whole new world with therapeutic implications Radiation as immunomodulator: implications for dendritic cell-based immunotherapy EDA-Fibronectin Originating from Osteoblasts Inhibits the Immune Response against Cancer Design and pharmacology of a highly specific dual FMS and KIT kinase inhibitor Radiation-induced immune responses: mechanisms and therapeutic perspectives Macrophage biology in development, homeostasis and disease Protective and pathogenic functions of macrophage subsets Functional significance of mononuclear phagocyte populations generated through adult hematopoiesis Enhanced cancer radiotherapy through immunosuppressive stromal cell destruction in tumors Emergence of spatial structure in the tumor microenvironment due to the Warburg effect Small molecule agonists of integrin CD11b/CD18 do not induce global conformational changes and are significantly better than activating antibodies in reducing vascular injury.The process of macrophage migration promotes matrix metalloproteinase-independent invasion by tumor cells.Small molecule-mediated activation of the integrin CD11b/CD18 reduces inflammatory disease.Validation of a device for the active manipulation of the tumor microenvironment during intravital imaging.Immunological Aspect of Radiation-Induced Pneumonitis, Current Treatment Strategies, and Future Prospects Macrophage infiltration and genetic landscape of undifferentiated uterine sarcomas.Efficient in vivo selection of a novel tumor-associated peptide from a phage display library.Neutrophils in the tumor microenvironment: trying to heal the wound that cannot heal.Melanoma cell expression of CD200 inhibits tumor formation and lung metastasis via inhibition of myeloid cell functions.Expression of NF-κB p50 in tumor stroma limits the control of tumors by radiation therapy.Head and neck cancer relapse after chemoradiotherapy correlates with CD163+ macrophages in primary tumour and CD11b+ myeloid cells in recurrences The tumor-immune microenvironment and response to radiation therapy Ionizing radiation induces tumor cell lysyl oxidase secretion.Combinations of immunotherapy and radiation in cancer therapy.Blood classical monocytes phenotype is not altered in primary non-small cell lung cancer.TH2-Polarized CD4(+) T Cells and Macrophages Limit Efficacy of Radiotherapy.Macrophages and cathepsin proteases blunt chemotherapeutic response in breast cancer.Immune microenvironments in solid tumors: new targets for therapy.Apurinic/Apyrimidinic endonuclease 1 regulates inflammatory response in macrophages.Prognostic Value of CD11b Expression Level for Acute Myeloid Leukemia Patients: A Meta-Analysis Dequalinium blocks macrophage-induced metastasis following local radiation.Targeting myeloid cells in the tumor microenvironment enhances vaccine efficacy in murine epithelial ovarian cancer.Differential macrophage programming in the tumor microenvironment.Increased Notch Signaling Enhances Radioresistance of Malignant Stromal Cells Induced by Glioma Stem/ Progenitor Cells Imaging tumor-stroma interactions during chemotherapy reveals contributions of the microenvironment to resistance.

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Inhibition of Mac-1 (CD11b/CD18) enhances tumor response to radiation by reducing myeloid cell recruitment

http://www.wikidata.org/entity/Q33929607

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Proceedings of the National Academy of Sciences of the United States of America

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Inhibition of Mac-1 (CD11b/CD1 ...... ucing myeloid cell recruitment

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Agnieszka Czechowicz

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Cho-Hwa Liao

http://www.w3.org/2001/XMLSchema#string

Diane Tseng

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G-One Ahn

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J Martin Brown

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Mary Jo Dorie

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P2860

Proinflammatory activities of S100: proteins S100A8, S100A9, and S100A8/A9 induce neutrophil chemotaxis and adhesion

Myeloid-related protein-8/14 is critical for the biological response to vascular injury

S100A9/S100A8: Myeloid representatives of the S100 protein family as prominent players in innate immunity

Rovelizumab (ICOS Corp)

Tumour-mediated upregulation of chemoattractants and recruitment of myeloid cells predetermines lung metastasis

Mac-1 (CD11b/CD18) is essential for Fc receptor-mediated neutrophil cytotoxicity and immunologic synapse formation.

Chemoattractant signaling between tumor cells and macrophages regulates cancer cell migration, metastasis and neovascularization.

Matrix metalloproteinase-9 is required for tumor vasculogenesis but not for angiogenesis: role of bone marrow-derived myelomonocytic cells

Increased serum levels of MRP-8/14 in type 1 diabetes induce an increased expression of CD11b and an enhanced adhesion of circulating monocytes to fibronectin.

Innovations in image-guided radiotherapy.

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43201508

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20404138

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2889597

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