The upstream regulatory region of the human papilloma virus-16 contains an E2 protein-independent enhancer which is specific for cervical carcinoma cells and regulated by glucocorticoid hormones.
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Cloning and functional analysis of spliced isoforms of human nuclear factor I-X: interference with transcriptional activation by NFI/CTF in a cell-type specific mannerThe combined influence of oral contraceptives and human papillomavirus virus on cutaneous squamous cell carcinomaTwo novel promoters in the upstream regulatory region of human papillomavirus type 31b are negatively regulated by epithelial differentiationThe chromatin structure of the long control region of human papillomavirus type 16 represses viral oncoprotein expression.Human papillomavirus type 16 cooperates with activated ras and fos oncogenes in the hormone-dependent transformation of primary mouse cells.The HPV Cellular Transactivator Brn-3a Can Be Used to Predict Cervical Adenocarcinoma and Squamous Carcinoma Precancer Lesions in the Developed and Developing Worlds.Induction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationNovel binding sites for regulatory factors in the human papillomavirus type 18 enhancer and promoter identified by in vivo footprintingNuclear matrix attachment regions of human papillomavirus type 16 point toward conservation of these genomic elements in all genital papillomavirusesRole of the E1--E4 protein in the differentiation-dependent life cycle of human papillomavirus type 31.Nuclear matrix attachment regions of human papillomavirus type 16 repress or activate the E6 promoter, depending on the physical state of the viral DNA.Critical roles of nuclear receptor response elements in replication of hepatitis B virus.Autostimulation of the Epstein-Barr virus BRLF1 promoter is mediated through consensus Sp1 and Sp3 binding sites.The bovine papillomavirus constitutive enhancer is essential for viral transformation, DNA replication, and the maintenance of latencyIs oral contraceptive associated with genital warts?Transcriptional enhancer factor (TEF)-1 and its cell-specific co-activator activate human papillomavirus-16 E6 and E7 oncogene transcription in keratinocytes and cervical carcinoma cells.Retinoic acid-mediated repression of human papillomavirus 18 transcription and different ligand regulation of the retinoic acid receptor beta gene in non-tumorigenic and tumorigenic HeLa hybrid cellsEstrogen and ERalpha: culprits in cervical cancer?Constitutive expression of c-myc oncogene confers hormone independence and enhanced growth-factor responsiveness on cells transformed by human papilloma virus type 16.The upstream regulatory region of human papillomavirus type 31 is insensitive to glucocorticoid induction.Pathogenesis of human papillomaviruses in differentiating epithelia.The octamer binding site in the HPV16 regulatory region produces opposite effects on gene expression in cervical and non-cervical cellsCpG methylation of human papillomavirus type 16 DNA in cervical cancer cell lines and in clinical specimens: genomic hypomethylation correlates with carcinogenic progressionDuring negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element.Transactivation of the human papilloma virus 16 octamer motif by the octamer binding protein Oct-2 requires both the N and C terminal activation domains.The influence of bio-behavioural factors on tumour biology: pathways and mechanisms.Clusters of nuclear factor I binding sites identify enhancers of several papillomaviruses but alone are not sufficient for enhancer function.Influence of ovarian hormones on urogenital infection.Molecular events in uterine cervical cancer.The constitutively expressed octamer binding protein OTF-1 and a novel octamer binding protein expressed specifically in cervical cells bind to an octamer-related sequence in the human papillomavirus 16 enhancer.Transcription of the transforming genes of the oncogenic human papillomavirus-16 is stimulated by tumor promotors through AP1 binding sites.YY1 represses human papillomavirus type 16 transcription by quenching AP-1 activity.CCAAT displacement protein, a regulator of differentiation-specific gene expression, binds a negative regulatory element within the 5' end of the human papillomavirus type 6 long control region.Differential effects of nerve growth factor and dexamethasone on herpes simplex virus type 1 oriL- and oriS-dependent DNA replication in PC12 cells.Transcriptional activation of human papillomavirus 16 by nuclear factor I, AP1, steroid receptors and a possibly novel transcription factor, PVF: a model for the composition of genital papillomavirus enhancers.Quantitative keratinocyte assay detects two biological activities of human papillomavirus DNA and identifies viral types associated with cervical carcinoma.Characterization of keratin and cell cycle protein expression in cell lines from squamous intraepithelial lesions progressing towards a malignant phenotype.Comprehensive comparison of the interaction of the E2 master regulator with its cognate target DNA sites in 73 human papillomavirus types by sequence statistics.Human genomic diversity, viral genomics and proteomics, as exemplified by human papillomaviruses and H5N1 influenza viruses.A steroid hormone response unit in the late leader of the noncoding control region of the human polyomavirus BK confers enhanced host cell permissivity.
P2860
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P2860
The upstream regulatory region of the human papilloma virus-16 contains an E2 protein-independent enhancer which is specific for cervical carcinoma cells and regulated by glucocorticoid hormones.
description
1987 nî lūn-bûn
@nan
1987 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
The upstream regulatory region ...... ed by glucocorticoid hormones.
@ast
The upstream regulatory region ...... ed by glucocorticoid hormones.
@en
type
label
The upstream regulatory region ...... ed by glucocorticoid hormones.
@ast
The upstream regulatory region ...... ed by glucocorticoid hormones.
@en
prefLabel
The upstream regulatory region ...... ed by glucocorticoid hormones.
@ast
The upstream regulatory region ...... ed by glucocorticoid hormones.
@en
P2093
P2860
P1433
P1476
The upstream regulatory region ...... ed by glucocorticoid hormones.
@en
P2093
P2860
P304
P407
P577
1987-12-01T00:00:00Z