Mucosal adjuvant activity of cholera toxin requires Th17 cells and protects against inhalation anthrax. - Wikidata - awgldk - TriplyDB

Mucosal adjuvant activity of cholera toxin requires Th17 cells and protects against inhalation anthrax.

Mucosal adjuvant activity of cholera toxin requires Th17 cells and protects against inhalation anthrax.

http://www.wikidata.org/entity/Q33935005

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Antigen-specific memory T cell responses after vaccination with an oral killed cholera vaccine in Bangladeshi children and comparison to responses in patients with naturally acquired cholera The cAMP Pathway as Therapeutic Target in Autoimmune and Inflammatory Diseases Porcine reproductive and respiratory syndrome virus vaccines: Immunogenicity, efficacy and safety aspects Vaccine approaches for multidrug resistant Gram negative infections Toxicity and immunogenicity of Enterotoxigenic Escherichia coli heat-labile and heat-stable toxoid fusion 3xSTa(A14Q)-LT(S63K/R192G/L211A) in a murine model.A study of Chitosan and c-di-GMP as mucosal adjuvants for intranasal influenza H5N1 vaccine.Colonic immune suppression, barrier dysfunction, and dysbiosis by gastrointestinal bacillus anthracis Infection.T(H)17-based vaccine design for prevention of Streptococcus pneumoniae colonization Implications of non-canonical G-protein signaling for the immune system Loss of sialic acid binding domain redirects protein σ1 to enhance M cell-directed vaccination Educating CD4 T cells with vaccine adjuvants: lessons from lipopolysaccharide.The A subunit of Escherichia coli heat-labile enterotoxin functions as a mucosal adjuvant and promotes IgG2a, IgA, and Th17 responses to vaccine antigens The adjuvant double mutant Escherichia coli heat labile toxin enhances IL-17A production in human T cells specific for bacterial vaccine antigens Colonic immune stimulation by targeted oral vaccine.Cholera toxin enhances vaccine-induced protection against Mycobacterium tuberculosis challenge in mice.Cyclic AMP concentrations in dendritic cells induce and regulate Th2 immunity and allergic asthma.Vibrio cholerae O1 infection induces proinflammatory CD4+ T-cell responses in blood and intestinal mucosa of infected humans.Natural cutaneous anthrax infection, but not vaccination, induces a CD4(+) T cell response involving diverse cytokines Divergent requirement for Gαs and cAMP in the differentiation and inflammatory profile of distinct mouse Th subsets.Neutrophils negatively regulate induction of mucosal IgA responses after sublingual immunization.Prostaglandin E2 suppresses antifungal immunity by inhibiting interferon regulatory factor 4 function and interleukin-17 expression in T cells.Single-cell sequencing provides clues about the host interactions of segmented filamentous bacteria (SFB).Cholera Toxin Promotes Th17 Cell Differentiation by Modulating Expression of Polarizing Cytokines and the Antigen-Presenting Potential of Dendritic Cells.Immune responses to cholera in children.The catalytic A1 domains of cholera toxin and heat-labile enterotoxin are potent DNA adjuvants that evoke mixed Th1/Th17 cellular immune responses Vaccine-induced th17 cells are maintained long-term postvaccination as a distinct and phenotypically stable memory subset.Mucosal vaccine adjuvants update.Inhibition of Gαs/cAMP Signaling Decreases TCR-Stimulated IL-2 transcription in CD4(+) T Helper Cells.Nod2-mediated recognition of the microbiota is critical for mucosal adjuvant activity of cholera toxin Intranasal immunization with influenza antigens conjugated with cholera toxin subunit B stimulates broad spectrum immunity against influenza viruses.Synthetic mast-cell granules as adjuvants to promote and polarize immunity in lymph nodes.B cell-intrinsic and -extrinsic regulation of antibody responses by PARP14, an intracellular (ADP-ribosyl)transferase.Cholera toxin directly enhances IL-17A production from human CD4+ T cells.Mucosal pre-exposure to Th17-inducing adjuvants exacerbates pathology after influenza infection.Basophil-derived IL-6 regulates TH17 cell differentiation and CD4 T cell immunity.Mechanisms of Cholera Toxin in the Modulation of TH17 Responses.Innate immune protection against infectious diseases by pulmonary administration of a phospholipid-conjugated TLR7 ligand.Toll-like receptor 2-dependent protection against pneumococcal carriage by immunization with lipidated pneumococcal proteins.Molecular basis of the mucosal immune system: from fundamental concepts to advances in liposome-based vaccines.Induction of gut IgA production through T cell-dependent and T cell-independent pathways.

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Mucosal adjuvant activity of cholera toxin requires Th17 cells and protects against inhalation anthrax.

http://www.wikidata.org/entity/Q33935005

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Mucosal adjuvant activity of c ...... ts against inhalation anthrax.

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Mucosal adjuvant activity of c ...... ts against inhalation anthrax.

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Mucosal adjuvant activity of c ...... ts against inhalation anthrax.

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Mucosal adjuvant activity of c ...... ts against inhalation anthrax.

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PNAS.1002348107

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Proceedings of the National Academy of Sciences of the United States of America

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Mucosal adjuvant activity of c ...... ts against inhalation anthrax.

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Donald G Guiney

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Eyal Raz

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Ivan Mihajlov

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Jose M Gonzalez-Navajas

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Joshua Fierer

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Kim Phung L Nguyen

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Mojgan Sabet

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Nicholas J Webster

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Patricia A Valdez

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Paul A Insel

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P2860

Epac is a Rap1 guanine-nucleotide-exchange factor directly activated by cyclic AMP

A family of cAMP-binding proteins that directly activate Rap1

Stimulation of the intracellular bacterial sensor NOD2 programs dendritic cells to promote interleukin-17 production in human memory T cells

The biological functions of T helper 17 cell effector cytokines in inflammation

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

IL-17 and Th17 Cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Transforming growth factor-beta induces development of the T(H)17 lineage

Control of T(reg) and T(H)17 cell differentiation by the aryl hydrocarbon receptor

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

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10638-10643

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107

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