Enhanced atherosclerosis and kidney dysfunction in eNOS(-/-)Apoe(-/-) mice are ameliorated by enalapril treatment.
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AtherosclerosisPPARalpha deficiency reduces insulin resistance and atherosclerosis in apoE-null miceBidirectional regulation of monocyte chemoattractant protein-1 gene at distinct sites of its promoter by nitric oxide in vascular smooth muscle cellsVascular respiratory uncoupling increases blood pressure and atherosclerosisEzetimibe potently reduces vascular inflammation and arteriosclerosis in eNOS-deficient ApoE ko mice.Reduced antioxidant capacity and diet-induced atherosclerosis in uncoupling protein-2-deficient mice.Dimethylarginine dimethylaminohydrolase overexpression ameliorates atherosclerosis in apolipoprotein E-deficient mice by lowering asymmetric dimethylarginine.Angiotensin II synergizes with BAFF to promote atheroprotective regulatory B cells.Mass spectrometric quantification of amino acid oxidation products identifies oxidative mechanisms of diabetic end-organ damage.Abscisic acid ameliorates atherosclerosis by suppressing macrophage and CD4+ T cell recruitment into the aortic wallEmphysema is associated with increased inflammation in lungs of atherosclerosis-prone mice by cigarette smoke: implications in comorbidities of COPD.Genetic modifiers of atherosclerosis in miceAngiopoietin-2 confers Atheroprotection in apoE-/- mice by inhibiting LDL oxidation via nitric oxide.SIRT1 decreases Lox-1-mediated foam cell formation in atherogenesiseNOS protects from atherosclerosis despite relevant superoxide production by the enzyme in apoE mice.α-Lipoic acid protects diabetic apolipoprotein E-deficient mice from nephropathyCyclophilin A is an inflammatory mediator that promotes atherosclerosis in apolipoprotein E-deficient mice.Interactions between endothelial nitric oxide synthase and sex hormones in vascular protection in mice.Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.Low circulating insulin-like growth factor I increases atherosclerosis in ApoE-deficient mice.Protein engineering to develop a redox insensitive endothelial nitric oxide synthaseIncreased atherosclerosis and smooth muscle cell hypertrophy in natriuretic peptide receptor A-/-apolipoprotein E-/- mice.Inducible nitric oxide synthase mediates prostaglandin h2 synthase nitration and suppresses eicosanoid productionEndothelial dysfunction exacerbates renal interstitial fibrosis through enhancing fibroblast Smad3 linker phosphorylation in the mouse obstructed kidney.PKCzeta decreases eNOS protein stability via inhibitory phosphorylation of ERK5.Protective effects of let-7a and let-7b on oxidized low-density lipoprotein induced endothelial cell injuriesAcute exercise activates AMPK and eNOS in the mouse aortaFunctional expression of endothelial nitric oxide synthase fused to green fluorescent protein in transgenic mice.The two faces of endothelial nitric oxide synthase in the pathophysiology of atherosclerosis.Role of interferon alpha in endothelial dysfunction: insights into endothelial nitric oxide synthase-related mechanismsAssociation between Body Mass Index, Asymmetric Dimethylarginine and Risk of Cardiovascular Events and Mortality in Norwegian Patients with Suspected Stable Angina Pectoris.Genetic basis of atherosclerosis: insights from mice and humansNOS3 polymorphisms, cigarette smoking, and cardiovascular disease risk: the Atherosclerosis Risk in Communities study.Pro- and antiatherogenic effects of a dominant-negative P465L mutation of peroxisome proliferator-activated receptor-γ in apolipoprotein E-Null mice.Relative reduction of endothelial nitric-oxide synthase expression and transcription in atherosclerosis-prone regions of the mouse aorta and in an in vitro model of disturbed flow.Requirement of apelin-apelin receptor system for oxidative stress-linked atherosclerosis.KLF2 Is a novel transcriptional regulator of endothelial proinflammatory activation.Recent advances in understanding endothelial dysfunction in atherosclerosis.The estrogen effects on endothelial repair and mitogen-activated protein kinase activation are abolished in endothelial nitric-oxide (NO) synthase knockout mice, but not by NO synthase inhibition by N-nitro-L-arginine methyl ester.Regulation of smooth muscle by inducible nitric oxide synthase and NADPH oxidase in vascular proliferative diseases
P2860
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P2860
Enhanced atherosclerosis and kidney dysfunction in eNOS(-/-)Apoe(-/-) mice are ameliorated by enalapril treatment.
description
2000 nî lūn-bûn
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2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@ast
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@en
type
label
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@ast
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@en
prefLabel
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@ast
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@en
P2093
P2860
P356
P1476
Enhanced atherosclerosis and k ...... orated by enalapril treatment.
@en
P2093
E G Shesely
J C Jennette
J W Knowles
R L Reddick
P2860
P304
P356
10.1172/JCI8376
P407
P577
2000-02-01T00:00:00Z