Plasmodium falciparum-infected erythrocytes induce tissue factor expression in endothelial cells and support the assembly of multimolecular coagulation complexes - Wikidata - awgldk - TriplyDB

Plasmodium falciparum-infected erythrocytes induce tissue factor expression in endothelial cells and support the assembly of multimolecular coagulation complexes

Plasmodium falciparum-infected erythrocytes induce tissue factor expression in endothelial cells and support the assembly of multimolecular coagulation complexes

http://www.wikidata.org/entity/Q33941766

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Inhibition of Plasmodium falciparum field isolates-mediated endothelial cell apoptosis by Fasudil: therapeutic implications for severe malaria Endothelium-based biomarkers are associated with cerebral malaria in Malawian children: a retrospective case-control study Tempol, an intracellular antioxidant, inhibits tissue factor expression, attenuates dendritic cell function, and is partially protective in a murine model of cerebral malaria.Blood coagulation, inflammation, and malaria.Malaria: a haematological disease.Platelets and infections in the resource-limited countries with a focus on malaria and viral haemorrhagic fevers.Association of Plasmodium falciparum with Human Endothelial Cells in vitro.Adhesion of Plasmodium falciparum-infected erythrocytes to human cells: molecular mechanisms and therapeutic implications.Does activation of the blood coagulation cascade have a role in malaria pathogenesis?Aegyptin, a novel mosquito salivary gland protein, specifically binds to collagen and prevents its interaction with platelet glycoprotein VI, integrin alpha2beta1, and von Willebrand factor Atypical neurological manifestations of malaria.Maternal malaria induces a procoagulant and antifibrinolytic state that is embryotoxic but responsive to anticoagulant therapy.Augmented plasma microparticles during acute Plasmodium vivax infection EPCR and Malaria Severity: The Center of a Perfect Storm.A potential role for plasma uric acid in the endothelial pathology of Plasmodium falciparum malaria.Malaria biology and disease pathogenesis: insights for new treatments.Malaria-induced splenic infarction.Inhibition of antithrombin by Plasmodium falciparum histidine-rich protein II Tissue factor, blood coagulation, and beyond: an overview.Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria The Impact of HIV Coinfection on Cerebral Malaria Pathogenesis.Laboratory evidence of disseminated intravascular coagulation is associated with a fatal outcome in children with cerebral malaria despite an absence of clinically evident thrombosis or bleeding.Cytoadherence and severe malaria.Ixolaris binding to factor X reveals a precursor state of factor Xa heparin-binding exosite.Diverse functional outcomes of Plasmodium falciparum ligation of EPCR: potential implications for malarial pathogenesis.Protection against cerebral malaria by the low-molecular-weight thiol pantethine.Depletion of Phagocytic Cells during Nonlethal Plasmodium yoelii Infection Causes Severe Malaria Characterized by Acute Renal Failure in Mice.Loss of endothelial protein C receptors links coagulation and inflammation to parasite sequestration in cerebral malaria in African children Arginine, nitric oxide, carbon monoxide, and endothelial function in severe malaria.Thrombin Cleavage of Plasmodium falciparum Erythrocyte Membrane Protein 1 Inhibits Cytoadherence.The Impact of HIV and Malaria Coinfection: What Is Known and Suggested Venues for Further Study.Dysregulation of coagulation in cerebral malaria.Leishmania amazonensis exhibits phosphatidylserine-dependent procoagulant activity, a process that is counteracted by sandfly saliva.Malaria: modification of the red blood cell and consequences in the human host.Cerebral malaria pathogenesis: revisiting parasite and host contributions.The immunological balance between host and parasite in malaria.Dynamic interactions of Plasmodium spp. with vascular endothelium.Evaluation of some haemostatic parameters in falciparum malaria and HIV co-infection.Sepsis-associated disseminated intravascular coagulation and thromboembolic disease.Defibrotide: a Swiss Army knife intervention in the battle against cerebral malaria.

P2860

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P2860

Plasmodium falciparum-infected erythrocytes induce tissue factor expression in endothelial cells and support the assembly of multimolecular coagulation complexes

http://www.wikidata.org/entity/Q33941766

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2006 nî lūn-bûn

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2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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J.1538-7836.2006.02232.X

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Journal of Thrombosis and Haemostasis

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Plasmodium falciparum-infected ...... olecular coagulation complexes

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A L L Noronha

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C R Erexson

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G R Ostera

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I M B Francischetti

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J M Ward

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K B Seydel

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R Q Monteiro

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S B Kamiza

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P2860

The transcriptome of the intraerythrocytic developmental cycle of Plasmodium falciparum

Bench-to-bedside review: functional relationships between coagulation and the innate immune response and their respective roles in the pathogenesis of sepsis

Estimation of the total parasite biomass in acute falciparum malaria from plasma PfHRP2

Role of platelet P-selectin and microparticle PSGL-1 in thrombus formation

Role of tissue factor in hemostasis, thrombosis, and vascular development

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

The dynamics of thrombin formation

The large diverse gene family var encodes proteins involved in cytoadherence and antigenic variation of plasmodium falciparum-infected erythrocytes

The pathogenic basis of malaria

Tissue factor-dependent coagulation is preferentially up-regulated within arterial branching areas in a baboon model of Escherichia coli sepsis.

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155-165

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10.1111/J.1538-7836.2006.02232.X

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1

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Plasmodium falciparum

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