Differential polarization of alveolar macrophages and bone marrow-derived monocytes following chemically and pathogen-induced chronic lung inflammation.
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Spatial, Temporal, and Functional Aspects of Macrophages during "The Good, the Bad, and the Ugly" Phases of InflammationRupatadine protects against pulmonary fibrosis by attenuating PAF-mediated senescence in rodentsChitinase dependent control of protozoan cyst burden in the brainPhosphodiesterase-4 inhibition combined with isoniazid treatment of rabbits with pulmonary tuberculosis reduces macrophage activation and lung pathologyBronchoalveolar Lavage Fluid Utilized Ex Vivo to Validate In Vivo Findings: Inhibition of Gap Junction Activity in Lung Tumor Promotion is Toll-Like Receptor 4-DependentEnhanced M1 macrophage polarization in human helicobacter pylori-associated atrophic gastritis and in vaccinated mice.Tumor necrosis factor-α accelerates the resolution of established pulmonary fibrosis in mice by targeting profibrotic lung macrophages.Macrophage migration inhibitory factor downregulation: a novel mechanism of resistance to anti-angiogenic therapy.Pathogenic Mycobacterium bovis strains differ in their ability to modulate the proinflammatory activation phenotype of macrophages.Assessing anti-fungal activity of isolated alveolar macrophages by confocal microscopyExtracellular mycobacterial DnaK polarizes macrophages to the M2-like phenotypeDepletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomasGranulomatous response to Coxiella burnetii, the agent of Q fever: the lessons from gene expression analysisPreferential macrophage recruitment and polarization in LPS-induced animal model for COPD: noninvasive tracking using MRI.Inflammatory cytokine and chemokine expression in sympathetic ophthalmia: a pilot studyAge and sex dimorphisms contribute to the severity of bleomycin-induced lung injury and fibrosis.M1 and M2 macrophages: the chicken and the egg of immunityIntrapulmonary administration of bone-marrow derived M1/M2 macrophages to enhance the resolution of LPS-induced lung inflammation: noninvasive monitoring using free-breathing MR and CT imaging protocols.Mycobacterium tuberculosis-Induced Polarization of Human Macrophage Orchestrates the Formation and Development of Tuberculous Granulomas In Vitro.Macrophage polarization: convergence point targeted by mycobacterium tuberculosis and HIV.Janus-faced liposomes enhance antimicrobial innate immune response in Mycobacterium tuberculosis infectionMyeloid-specific Fos-related antigen-1 regulates cigarette smoke-induced lung inflammation, not emphysema, in miceNeutrophils are required for 3-methylcholanthrene-initiated, butylated hydroxytoluene-promoted lung carcinogenesis.Biliverdin Reductase A (BVRA) Mediates Macrophage Expression of Interleukin-10 in Injured KidneyResection of non-small cell lung cancers reverses tumor-induced gene expression changes in the peripheral immune system.Arginase 1: an unexpected mediator of pulmonary capillary barrier dysfunction in models of acute lung injury.Macrophage polarization in interstitial lung diseasesLocal arginase 1 activity is required for cutaneous wound healing.Pulmonary macrophage subpopulations in the induction and resolution of acute lung injury.M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection.Modulation of cell death by M. tuberculosis as a strategy for pathogen survivalDiversity and dialogue in immunity to helminths.Alveolar macrophages in diabetes: friends or foes?Local immune responses in human tuberculosis: learning from the site of infection.Characteristics of suppressor macrophages induced by mycobacterial and protozoal infections in relation to alternatively activated M2 macrophages.Within the Enemy's Camp: contribution of the granuloma to the dissemination, persistence and transmission of Mycobacterium tuberculosis.Immune Homeostatic Macrophages Programmed by the Bacterial Surface Protein NhhA Potentiate Nasopharyngeal Carriage of Neisseria meningitidis.Macrophage takeover and the host-bacilli interplay during tuberculosis.A mechanistic review of silica-induced inhalation toxicity.Cell Origin Dictates Programming of Resident versus Recruited Macrophages during Acute Lung Injury.
P2860
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P2860
Differential polarization of alveolar macrophages and bone marrow-derived monocytes following chemically and pathogen-induced chronic lung inflammation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Differential polarization of a ...... ced chronic lung inflammation.
@ast
Differential polarization of a ...... ced chronic lung inflammation.
@en
type
label
Differential polarization of a ...... ced chronic lung inflammation.
@ast
Differential polarization of a ...... ced chronic lung inflammation.
@en
prefLabel
Differential polarization of a ...... ced chronic lung inflammation.
@ast
Differential polarization of a ...... ced chronic lung inflammation.
@en
P2093
P2860
P356
P1476
Differential polarization of a ...... uced chronic lung inflammation
@en
P2093
Alvin M Malkinson
David M Higgins
Elizabeth F Redente
Ian M Orme
Lori D Dwyer-Nield
P2860
P304
P356
10.1189/JLB.0609378
P577
2010-04-01T00:00:00Z