Lytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional response
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Cell Culture, Technology: Enhancing the Culture of Diagnosing Human DiseasesRestarting Lytic Gene Transcription at the Onset of Herpes Simplex Virus ReactivationNeuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.Immune Escape via a Transient Gene Expression Program Enables Productive Replication of a Latent Pathogen.Herpesviruses dUTPases: A New Family of Pathogen-Associated Molecular Pattern (PAMP) Proteins with Implications for Human DiseaseHuman Cytomegalovirus Latency: Approaching the Gordian Knot.Establishment of HSV1 latency in immunodeficient mice facilitates efficient in vivo reactivation.The Current State of Vaccine Development for Ocular HSV-1 InfectionLytic Promoters Express Protein during Herpes Simplex Virus Latency.Autophagy interaction with herpes simplex virus type-1 infectionA Herpesviral Lytic Protein Regulates the Structure of Latent Viral Chromatin.Analyses of herpes simplex virus type 1 latency and reactivation at the single cell level using fluorescent reporter mice.Neurons versus herpes simplex virus: the innate immune interactions that contribute to a host-pathogen standoffEpigenetics and Genetics of Viral Latency.Impact of Type I Interferon on the Safety and Immunogenicity of an Experimental Live-Attenuated Herpes Simplex Virus 1 Vaccine in Mice.Defining nervous system susceptibility during acute and latent herpes simplex virus-1 infection.Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency.Influence of an immunodominant herpes simplex virus type 1 CD8+ T cell epitope on the target hierarchy and function of subdominant CD8+ T cells.The loss of binary: Pushing the herpesvirus latency paradigm.Transcriptome-wide characterization of human cytomegalovirus in natural infection and experimental latency.Effective Priming of Herpes Simplex Virus-Specific CD8+ T Cells In Vivo Does Not Require Infected Dendritic Cells.Laser Microdissection.Increasing antigen presentation on HSV-1-infected cells increases lesion size but does not alter neural infection or latency.A Single-Cell Approach to the Elusive Latent Human Cytomegalovirus Transcriptome.Strength in diversity: Understanding the pathways to herpes simplex virus reactivation
P2860
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P2860
Lytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional response
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Lytic gene expression is frequ ...... insic transcriptional response
@ast
Lytic gene expression is frequ ...... insic transcriptional response
@en
type
label
Lytic gene expression is frequ ...... insic transcriptional response
@ast
Lytic gene expression is frequ ...... insic transcriptional response
@en
prefLabel
Lytic gene expression is frequ ...... insic transcriptional response
@ast
Lytic gene expression is frequ ...... insic transcriptional response
@en
P2860
P50
P1433
P1476
Lytic gene expression is frequ ...... insic transcriptional response
@en
P2093
Francis R Carbone
Tim Spelman
P2860
P304
P356
10.1371/JOURNAL.PPAT.1004237
P577
2014-07-24T00:00:00Z