Loss of TBL1XR1 disrupts glucocorticoid receptor recruitment to chromatin and results in glucocorticoid resistance in a B-lymphoblastic leukemia model. - Wikidata - awgldk - TriplyDB

Loss of TBL1XR1 disrupts glucocorticoid receptor recruitment to chromatin and results in glucocorticoid resistance in a B-lymphoblastic leukemia model.

Loss of TBL1XR1 disrupts glucocorticoid receptor recruitment to chromatin and results in glucocorticoid resistance in a B-lymphoblastic leukemia model.

http://www.wikidata.org/entity/Q33946996

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Pharmacogenetics predictive of response and toxicity in acute lymphoblastic leukemia therapy Diffuse large B-cell lymphoma: can genomics improve treatment options for a curable cancer?NALP3 inflammasome upregulation and CASP1 cleavage of the glucocorticoid receptor cause glucocorticoid resistance in leukemia cells Genome-Wide DNA Copy Number Analysis of Acute Lymphoblastic Leukemia Identifies New Genetic Markers Associated with Clinical Outcome.MAPK signaling cascades mediate distinct glucocorticoid resistance mechanisms in pediatric leukemia.MLL-Rearranged Leukemias-An Update on Science and Clinical Approaches.The mutational landscape of ocular marginal zone lymphoma identifies frequent alterations in TNFAIP3 followed by mutations in TBL1XR1 and CREBBP.TBL1XR1 in physiological and pathological states Towards an understanding of the biology and targeted treatment of paediatric relapsed acute lymphoblastic leukaemia.Personalization of dexamethasone therapy in childhood acute lymphoblastic leukaemia.Suppression of B-cell development genes is key to glucocorticoid efficacy in treatment of acute lymphoblastic leukemia.Glucocorticoids and Reproduction: Traffic Control on the Road to Reproduction.Molecular mechanisms of OLIG2 transcription factor in brain cancer.Transducin (β)-like 1 X-linked receptor 1 correlates with clinical prognosis and clinicopathological characteristics in human solid carcinomas.Cytoplasmic, full length and novel cleaved variant, TBLR1 reduces apoptosis in prostate cancer under androgen deprivation.MSH6 haploinsufficiency at relapse contributes to the development of thiopurine resistance in pediatric B-lymphoblastic leukemia.New targeted therapies for relapsed pediatric acute lymphoblastic leukemia.IKZF1 deletion is enriched in pediatric B-cell precursor acute lymphoblastic leukemia patients showing prednisolone resistance.Tumor suppressor IKZF1 mediates glucocorticoid resistance in B-cell precursor acute lymphoblastic leukemia.

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P2860

Loss of TBL1XR1 disrupts glucocorticoid receptor recruitment to chromatin and results in glucocorticoid resistance in a B-lymphoblastic leukemia model.

http://www.wikidata.org/entity/Q33946996

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Loss of TBL1XR1 disrupts gluco ...... -lymphoblastic leukemia model.

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Loss of TBL1XR1 disrupts gluco ...... -lymphoblastic leukemia model.

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Loss of TBL1XR1 disrupts gluco ...... -lymphoblastic leukemia model.

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Loss of TBL1XR1 disrupts gluco ...... -lymphoblastic leukemia model.

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Loss of TBL1XR1 disrupts gluco ...... -lymphoblastic leukemia model.

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Journal of Biological Chemistry

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Loss of TBL1XR1 disrupts gluco ...... B-lymphoblastic leukemia model

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Courtney L Jones

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Danielle S Bitterman

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Debra J Morrison

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Elizabeth A Raetz

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Jinhua Wang

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Michael J Garabedian

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Steven W Paugh

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Teena Bhatla

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Wallace Bourgeois

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William E Evans

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P2860

TBLR1 regulates the expression of nuclear hormone receptor co-repressors

A novel cofactor for p300 that regulates the p53 response

TBL1 and TBLR1 phosphorylation on regulated gene promoters overcomes dual CtBP and NCoR/SMRT transcriptional repression checkpoints

Mechanism of SMRT corepressor recruitment by the BCL6 BTB domain

Mechanisms of glucocorticoid-mediated apoptosis in hematological malignancies

A corepressor/coactivator exchange complex required for transcriptional activation by nuclear receptors and other regulated transcription factors

Cooperative NCoR/SMRT interactions establish a corepressor-based strategy for integration of inflammatory and anti-inflammatory signaling pathways

Genome-wide analysis of genetic alterations in acute lymphoblastic leukaemia

The origin and nature of tightly clustered BTG1 deletions in precursor B-cell acute lymphoblastic leukemia support a model of multiclonal evolution.

Analysis of the coding genome of diffuse large B-cell lymphoma

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20502-20515

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10.1074/JBC.M114.569889

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David T Teachey

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