Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo. - Wikidata - awgldk - TriplyDB

Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.

Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.

http://www.wikidata.org/entity/Q33950241

about

T lymphocytes amplify the anabolic activity of parathyroid hormone through Wnt10b signaling Identification of the homeobox protein Prx1 (MHox, Prrx-1) as a regulator of osterix expression and mediator of tumor necrosis factor α action in osteoblast differentiation Osteoimmunology: interactions of the bone and immune system The molecular mechanism of osteoclastogenesis in rheumatoid arthritis Osteoporosis in experimental postmenopausal polyarthritis: the relative contributions of estrogen deficiency and inflammation Osteoimmunology and the influence of pro-inflammatory cytokines on osteoclasts Nuclear receptors in bone physiology and diseases Immune regulation of osteoclast function in postmenopausal osteoporosis: a critical interdisciplinary perspective HIV: inflammation and bone Pathophysiological roles for IL-18 in inflammatory arthritis Estrogen regulation of immune cell bone interactions Disorders of bone remodeling Pharmacologic stem cell based intervention as a new approach to osteoporosis treatment in rodents IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor kappa B ligand and tumor necrosis factor alpha from T cells Identification of B cells participated in the mechanism of postmenopausal women osteoporosis using microarray analysis DLK1 is a novel regulator of bone mass that mediates estrogen deficiency-induced bone loss in mice.IL-7 up-regulates TNF-alpha-dependent osteoclastogenesis in patients affected by solid tumor.Lymphocytes and the Dap12 adaptor are key regulators of osteoclast activation associated with gonadal failure.Differential cytokine regulation by NF-kappaB and AP-1 in Jurkat T-cells.A bone-protective role for IL-17 receptor signaling in ovariectomy-induced bone loss.Minireview: osteoprotective action of estrogens is mediated by osteoclastic estrogen receptor-alpha Role of direct estrogen receptor signaling in wear particle-induced osteolysis.Disruption of PTH receptor 1 in T cells protects against PTH-induced bone loss Tob deficiency superenhances osteoblastic activity after ovariectomy to block estrogen deficiency-induced osteoporosis Bone phenotypes in response to gonadotropin misexpression: the role for gonadotropins in postmenopausal osteoporosis Gender dimorphisms in progenitor and stem cell function in cardiovascular disease.Is aspirin treatment an appropriate intervention to osteoporosis?The inflammatory phase of fracture healing is influenced by oestrogen status in mice Daidzein prevents the increase in CD4+CD28null T cells and B lymphopoesis in ovariectomized mice: a key mechanism for anti-osteoclastogenic effect.T cells: critical bone regulators in health and disease.An IL-7-dependent rebound in thymic T cell output contributes to the bone loss induced by estrogen deficiency.The immune system and bone.Calcineurin regulates bone formation by the osteoblast Bone mineral density in rheumatoid arthritis patients 1 year after adalimumab therapy: arrest of bone loss.Oxidative stress stimulates apoptosis and activates NF-kappaB in osteoblastic cells via a PKCbeta/p66shc signaling cascade: counter regulation by estrogens or androgens.Receptor activator of NF-{kappa}B (RANK) cytoplasmic IVVY535-538 motif plays an essential role in tumor necrosis factor-{alpha} (TNF)-mediated osteoclastogenesis.The effect of Astragalus extractive on alveolar bone rebuilding progress of tooth extracted socket of ovariectomied rats Stem cell transplantation improves aging-related diseases Estrogen deficiency induces the differentiation of IL-17 secreting Th17 cells: a new candidate in the pathogenesis of osteoporosis.Transplantation of SHED prevents bone loss in the early phase of ovariectomy-induced osteoporosis.

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P2860

Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.

http://www.wikidata.org/entity/Q33950241

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Proceedings of the National Academy of Sciences of the United States of America

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P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction

Functional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.

M-CSF neutralization and egr-1 deficiency prevent ovariectomy-induced bone loss.

Two TNF receptors.

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