Pharmacological studies of the mechanism and function of interleukin-1beta-induced miRNA-146a expression in primary human airway smooth muscle. - Wikidata - awgldk - TriplyDB

Pharmacological studies of the mechanism and function of interleukin-1beta-induced miRNA-146a expression in primary human airway smooth muscle.

Pharmacological studies of the mechanism and function of interleukin-1beta-induced miRNA-146a expression in primary human airway smooth muscle.

http://www.wikidata.org/entity/Q33951715

about

MicroRNA-146a: A Dominant, Negative Regulator of the Innate Immune Response Epigenetics and miRNA emerge as key regulators of smooth muscle cell phenotype and function The calcium-sensing receptor mediates hypoxia-induced proliferation of rat pulmonary artery smooth muscle cells through MEK1/ERK1,2 and PI3K pathways Role of non-coding RNAs in maintaining primary airway smooth muscle cells MicroRNA-146a and microRNA-146b expression and anti-inflammatory function in human airway smooth muscle.MiR-181a regulates inflammation responses in monocytes and macrophages miR-146a inhibits cell growth, cell migration and induces apoptosis in non-small cell lung cancer cells MiR-29a reduces TIMP-1 production by dermal fibroblasts via targeting TGF-β activated kinase 1 binding protein 1, implications for systemic sclerosis BET bromodomains regulate transforming growth factor-β-induced proliferation and cytokine release in asthmatic airway smooth muscle.miRNA-146a expression positively regulates tumor necrosis factor-α-induced interleukin-8 production in mesenchymal stem cells and differentiated lung epithelial-like cells.Airway smooth muscle in airway reactivity and remodeling: what have we learned?miR-146a-mediated suppression of the inflammatory response in human adipocytes.Airway smooth muscle hyperproliferation is regulated by microRNA-221 in severe asthma.Transcriptional profiling identifies the long noncoding RNA plasmacytoma variant translocation (PVT1) as a novel regulator of the asthmatic phenotype in human airway smooth muscle.The emerging role of microRNAs in asthma.The emerging role of microRNAs in regulating immune and inflammatory responses in the lung.Targeting translational control as a novel way to treat inflammatory disease: the emerging role of microRNAs.Epigenetic targets for novel therapies of lung diseases.MicroRNAs as therapeutics for future drug delivery systems in treatment of lung diseases.Emerging concepts in smooth muscle contributions to airway structure and function: implications for health and disease.MicroRnas Profiling in Asthma: Potential Biomarkers and Therapeutic Targets.MicroRNAs-control of essential genes: Implications for pulmonary vascular disease.MiR-146a negatively regulates neutrophil elastase-induced MUC5AC secretion from 16HBE human bronchial epithelial cells.Role of Altered Expression of miR-146a, miR-155, and miR-122 in Pediatric Patients with Inflammatory Bowel Disease.α-Asarone suppresses the proliferation and migration of ASMCs through targeting the lncRNA-PVT1/miR-203a/E2F3 signal pathway in RSV-infected rats.Mycobacterium tuberculosis controls microRNA-99b (miR-99b) expression in infected murine dendritic cells to modulate host immunity Airway smooth muscle inflammation is regulated by microRNA-145 in COPD.Lipopolysaccharide-induced microRNA-146a targets CARD10 and regulates angiogenesis in human umbilical vein endothelial cells.MicroRNA Regulation of Smooth Muscle Phenotype.Interleukin-1β and microRNA-146a in an immature rat model and children with mesial temporal lobe epilepsy.Identification of differentially expressed miRNAs associated with chronic kidney disease-mineral bone disorder.Association of the miR-196a2, miR-146a, and miR-499 Polymorphisms with Asthma Phenotypes in a Korean Population.Lung injury caused by paraquat poisoning results in increased interleukin-6 and decreased microRNA-146a levels.The anti-proliferative and anti-inflammatory response of COPD airway smooth muscle cells to hydrogen sulfide.Anti-Inflammatory MicroRNAs and Their Potential for Inflammatory Diseases Treatment.

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Pharmacological studies of the mechanism and function of interleukin-1beta-induced miRNA-146a expression in primary human airway smooth muscle.

http://www.wikidata.org/entity/Q33951715

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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The role of microRNA genes in papillary thyroid carcinoma

A microRNA expression signature of human solid tumors defines cancer gene targets

Interleukin-1 (IL-1) receptor-associated kinase leads to activation of TAK1 by inducing TAB2 translocation in the IL-1 signaling pathway

MicroRNAs: target recognition and regulatory functions

Foxp3-dependent microRNA155 confers competitive fitness to regulatory T cells by targeting SOCS1 protein

MicroRNA-155 modulates the interleukin-1 signaling pathway in activated human monocyte-derived dendritic cells

MicroRNA-155 suppresses activation-induced cytidine deaminase-mediated Myc-Igh translocation

Inositol phosphatase SHIP1 is a primary target of miR-155

Induction and regulatory function of miR-9 in human monocytes and neutrophils exposed to proinflammatory signals

Sustained expression of microRNA-155 in hematopoietic stem cells causes a myeloproliferative disorder

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