Atm is dispensable for p53 apoptosis and tumor suppression triggered by cell cycle dysfunction.
about
Immunoglobulin class switch recombination is impaired in Atm-deficient miceLimited role of murine ATM in oncogene-induced senescence and p53-dependent tumor suppression.Defining the ATM-mediated barrier to tumorigenesis in somatic mammary cells following ErbB2 activationCiprofloxacin as a potential radio-sensitizer to tumor cells and a radio-protectant for normal cells: differential effects on γ-H2AX formation, p53 phosphorylation, Bcl-2 production, and cell deathChk2 is dispensable for p53-mediated G1 arrest but is required for a latent p53-mediated apoptotic response.DNA damage-induced apoptosis requires the DNA-dependent protein kinase, and is mediated by the latent population of p53.RAG-mediated V(D)J recombination is not essential for tumorigenesis in Atm-deficient miceATM promotes apoptosis and suppresses tumorigenesis in response to Myc.The pathological response to DNA damage does not contribute to p53-mediated tumour suppression.Recombinase-activating gene (RAG) 2-mediated V(D)J recombination is not essential for tumorigenesis in Atm-deficient mice.A coordinated DNA damage response promotes adult quiescent neural stem cell activation.Immature thymocytes undergoing receptor rearrangements are resistant to an Atm-dependent death pathway activated in mature T cells by double-stranded DNA breaksEndoplasmic reticulum stress and unfolded protein response in Atm-deficient thymocytes and thymic lymphoma cells are attributable to oxidative stressApoptosis associated with deregulated E2F activity is dependent on E2F1 and Atm/Nbs1/Chk2.The catalytic subunit of DNA-dependent protein kinase selectively regulates p53-dependent apoptosis but not cell-cycle arrest.T antigen transgenic mouse modelsActivation of AMP-activated protein kinase in cerebella of Atm-/- mice is attributable to accumulation of reactive oxygen species.Requirement for DNA ligase IV during embryonic neuronal development.Induction and utilization of an ATM signaling pathway by polyomavirus.Tumor suppression by p53 in the absence of Atm.Identifying and analyzing different cancer subtypes using RNA-seq data of blood platelets.
P2860
Q24647660-A4670436-DDE1-47C2-817D-F68652E5677AQ33440643-F0F7D5A3-8A3B-4180-86C9-A295BEFA1895Q33734684-705939A4-E233-4188-B900-DCDC1F2F5052Q34004301-CC5614E8-3A3F-40F6-B2FF-D322EE93BDF0Q34075834-967E09ED-019C-4346-83B9-B4874B2630E5Q34089341-DD65EBB1-9154-4A08-AF52-E0A5F9964C13Q34278357-6D72DF10-B75C-4FA5-B2E5-00E8860B94C9Q34298505-A3905FB9-9FD8-45FD-8131-41FA97A62AF5Q34564198-8E184C8E-FF78-4916-8C89-87C3F9A2D022Q35795447-B2284E86-155B-4315-828D-097F4A0F74BFQ36367916-BF4C7D2F-1352-4F00-9E5B-E9103A0646FDQ36368896-34A21A6D-521E-410B-995F-8878E12A630EQ36462824-75238E70-08F3-4D4B-B541-A7F544329782Q37011615-163D810E-CC72-4043-AE81-1507D96BBC73Q37109768-09A88844-C9AC-401E-9E64-274F9BA3554CQ37511197-DA80B3D8-3358-4BE2-BA92-E00AD2DCE1E4Q41811796-6CADA492-1002-4AA2-B8A2-8D2D00A0A3B5Q41935216-69CA90B5-7C0B-46A3-BE5A-A313592AF7E9Q42109796-566F5114-D341-4C6B-8C51-F3AE2CAC2579Q43130332-AC879D46-177A-4D1A-B50E-CBC6DD9314E6Q45943794-E58C350F-ECDA-4FAE-919C-47F3BD247E7E
P2860
Atm is dispensable for p53 apoptosis and tumor suppression triggered by cell cycle dysfunction.
description
1999 nî lūn-bûn
@nan
1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@ast
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@en
type
label
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@ast
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@en
prefLabel
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@ast
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@en
P2093
P2860
P356
P1476
Atm is dispensable for p53 apo ...... red by cell cycle dysfunction.
@en
P2093
P2860
P304
P356
10.1128/MCB.19.4.3095
P407
P577
1999-04-01T00:00:00Z