Mutations in the unc-52 gene responsible for body wall muscle defects in adult Caenorhabditis elegans are located in alternatively spliced exons
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Analysis of smu-1, a gene that regulates the alternative splicing of unc-52 pre-mRNA in Caenorhabditis elegansA network of HSPG core proteins and HS modifying enzymes regulates netrin-dependent guidance of D-type motor neurons in Caenorhabditis elegansZinc Levels Modulate Lifespan through Multiple Longevity Pathways in Caenorhabditis elegansA novel mutation in β integrin reveals an integrin-mediated interaction between the extracellular matrix and cki-1/p27KIP1Colored polydimethylsiloxane micropillar arrays for high throughput measurements of forces applied by genetic model organisms.Calpains mediate integrin attachment complex maintenance of adult muscle in Caenorhabditis elegansIntronic alternative splicing regulators identified by comparative genomics in nematodesMutations in the Caenorhabditis elegans U2AF large subunit UAF-1 alter the choice of a 3' splice site in vivoA current view of perlecan in physiology and pathology: A mosaic of functions.Dietary restriction suppresses proteotoxicity and enhances longevity by an hsf-1-dependent mechanism in Caenorhabditis elegans.Inhibition of touch cell fate by egl-44 and egl-46 in C. elegansTILLING is an effective reverse genetics technique for Caenorhabditis elegans.C. elegans agrin is expressed in pharynx, IL1 neurons and distal tip cells and does not genetically interact with genes involved in synaptogenesis or muscle function.Developmental genetics of the C. elegans pharyngeal neurons NSML and NSMR.C. elegans ten-1 is synthetic lethal with mutations in cytoskeleton regulators, and enhances many axon guidance defective mutants.C. elegans dysferlin homolog fer-1 is expressed in muscle, and fer-1 mutations initiate altered gene expression of muscle enriched genes.Functional overlap between the mec-8 gene and five sym genes in Caenorhabditis elegans.Perlecan and tumor angiogenesis.SMU-2 and SMU-1, Caenorhabditis elegans homologs of mammalian spliceosome-associated proteins RED and fSAP57, work together to affect splice site choiceA decline in transcript abundance for Heterodera glycines homologs of Caenorhabditis elegans uncoordinated genes accompanies its sedentary parasitic phaseRegulation of sex-specific differentiation and mating behavior in C. elegans by a new member of the DM domain transcription factor family.CCDC-55 is required for larval development and distal tip cell migration in Caenorhabditis elegans.Sarcomeres Pattern Proprioceptive Sensory Dendritic Endings through UNC-52/Perlecan in C. elegans.Structural components of the nonstriated contractile apparatuses in the Caenorhabditis elegans gonadal myoepithelial sheath and their essential roles for ovulation.The C. elegans UNC-23 protein, a member of the BCL-2-associated athanogene (BAG) family of chaperone regulators, interacts with HSP-1 to regulate cell attachment and maintain hypodermal integrity.Ca2+-dependent muscle dysfunction caused by mutation of the Caenorhabditis elegans troponin T-1 geneThe UNC-112 gene in Caenorhabditis elegans encodes a novel component of cell-matrix adhesion structures required for integrin localization in the muscle cell membraneBasement Membranes in the Worm: A Dynamic Scaffolding that Instructs Cellular Behaviors and Shapes Tissues.Complex patterns of alternative splicing mediate the spatial and temporal distribution of perlecan/UNC-52 in Caenorhabditis elegansA genetic screen for suppressors of a mutated 5' splice site identifies factors associated with later steps of spliceosome assembly.HRP-2, the Caenorhabditis elegans homolog of mammalian heterogeneous nuclear ribonucleoproteins Q and R, is an alternative splicing factor that binds to UCUAUC splicing regulatory elements.Border patrol: insights into the unique role of perlecan/heparan sulfate proteoglycan 2 at cell and tissue bordersRole of the extracellular matrix in epithelial morphogenesis: a view from C. elegans.MIP-MAP: High Throughput Mapping of Caenorhabditis elegans Temperature-Sensitive Mutants via Molecular Inversion Probes.Extracellular matrix regulates UNC-6 (netrin) axon guidance by controlling the direction of intracellular UNC-40 (DCC) outgrowth activity.Switch-like regulation of tissue-specific alternative pre-mRNA processing patterns revealed by customized fluorescence reporters.Genetic Suppression of Basement Membrane Defects in Caenorhabditis elegans by Gain of Function in Extracellular Matrix and Cell-Matrix Attachment Genes.
P2860
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P2860
Mutations in the unc-52 gene responsible for body wall muscle defects in adult Caenorhabditis elegans are located in alternatively spliced exons
description
1995 nî lūn-bûn
@nan
1995 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@ast
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@en
type
label
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@ast
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@en
prefLabel
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@ast
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@en
P2093
P2860
P1433
P1476
Mutations in the unc-52 gene r ...... in alternatively spliced exons
@en
P2093
Gilchrist EJ
Moerman DG
Rogalski TM
P2860
P304
P407
P577
1995-01-01T00:00:00Z