c-mip impairs podocyte proximal signaling and induces heavy proteinuria - Wikidata - awgldk - TriplyDB

c-mip impairs podocyte proximal signaling and induces heavy proteinuria

c-mip impairs podocyte proximal signaling and induces heavy proteinuria

http://www.wikidata.org/entity/Q33965018

about

Disruption of cytokeratin-8 interaction with F508del-CFTR corrects its functional defect Podocytes CD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survival Direct regulation of nephrin tyrosine phosphorylation by Nck adaptor proteins.Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis.Transcriptional factors, Mafs and their biological roles Innovative delivery of siRNA to solid tumors by super carbonate apatite.Minimal change nephrotic syndrome associated with non-Hodgkin lymphoid disorders: a retrospective study of 18 cases.TLR-mediated albuminuria needs TNFα-mediated cooperativity between TLRs present in hematopoietic tissues and CD80 present on non-hematopoietic tissues in mice.Upregulation of c-mip is closely related to podocyte dysfunction in membranous nephropathy Immunopathogenesis of idiopathic nephrotic syndrome with relapse.Physiopathology of idiopathic nephrotic syndrome: lessons from glucocorticoids and epigenetic perspectives.Tyrosine kinase signaling in kidney glomerular podocytes.Signaling from the podocyte intercellular junction to the actin cytoskeleton.The podocyte slit diaphragm--from a thin grey line to a complex signalling hub.Inhibition of the VEGF signalling pathway and glomerular disorders.Onco-nephrology: current concepts and future perspectives.Nephrin Tyrosine Phosphorylation Is Required to Stabilize and Restore Podocyte Foot Process Architecture.Rituximab for minimal-change nephrotic syndrome in adulthood: predictive factors for response, long-term outcomes and tolerance.Interaction of CD80 with Neph1: a potential mechanism of podocyte injury.Molecular and Cellular Mechanisms for Proteinuria in Minimal Change Disease.Proteinuric Kidney Diseases: A Podocyte's Slit Diaphragm and Cytoskeleton Approach Focal segmental glomerulosclerosis lesion associated with inhibition of tyrosine kinases by lenvatinib: a case report

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c-mip impairs podocyte proximal signaling and induces heavy proteinuria

http://www.wikidata.org/entity/Q33965018

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

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name

c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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type

label

c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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prefLabel

c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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c-mip impairs podocyte proximal signaling and induces heavy proteinuria

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SCISIGNAL.2000678

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Science Signaling

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c-mip impairs podocyte proximal signaling and induces heavy proteinuria

@en

P2093

André Pawlak

http://www.w3.org/2001/XMLSchema#string

Christo Christov

http://www.w3.org/2001/XMLSchema#string

Djillali Sahali

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Dominique Desvaux

http://www.w3.org/2001/XMLSchema#string

Fatima BenMohamed

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Georges Guellaën

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Gilles Mangiapan

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Karine Dahan

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Marie Matignon

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Marina Candelier

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P2860

Regulation of Fyn through translocation of activated Lck into lipid rafts

Nck adaptor proteins link nephrin to the actin cytoskeleton of kidney podocytes

Positional cloning uncovers mutations in PLCE1 responsible for a nephrotic syndrome variant that may be reversible

Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome

NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome

Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis

CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility

Wiskott-Aldrich syndrome protein (WASp) is a binding partner for c-Src family protein-tyrosine kinases

Nephrin redistribution on podocytes is a potential mechanism for proteinuria in patients with primary acquired nephrotic syndrome

TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

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10.1126/SCISIGNAL.2000678

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122

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