Increased adipocyte O2 consumption triggers HIF-1α, causing inflammation and insulin resistance in obesity. - Wikidata - awgldk - TriplyDB

Increased adipocyte O2 consumption triggers HIF-1α, causing inflammation and insulin resistance in obesity.

Increased adipocyte O2 consumption triggers HIF-1α, causing inflammation and insulin resistance in obesity.

http://www.wikidata.org/entity/Q33965673

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Adipose Tissue Remodeling: Its Role in Energy Metabolism and Metabolic Disorders Adipose tissue macrophages: going off track during obesity The Crosstalk between Hypoxia and Innate Immunity in the Development of Obesity-Related Nonalcoholic Fatty Liver Disease Insulin resistance, glucose intolerance and diabetes mellitus in obstructive sleep apnoea Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?Regulation of energy balance by inflammation: common theme in physiology and pathology Obesity and cancer: the role of adipose tissue and adipo-cytokines-induced chronic inflammation The cell biology of fat expansion Hypoxia-regulated mechanisms in the pathogenesis of obesity and non-alcoholic fatty liver disease.Data on biochemical indexes of HFD-fed mice treatment with metformin or resveratrol A Systems Genetics Approach Identified GPD1L and its Molecular Mechanism for Obesity in Human Adipose Tissue.Effects of siRNA-mediated HIF-1α gene silencing on angiogenesis in osteosarcoma.Inflammation in sleep apnea: an update.P65 inactivation in adipocytes and macrophages attenuates adipose inflammatory response in lean but not in obese mice.Immune regulation of metabolic homeostasis in health and disease.Adipocyte SIRT1 knockout promotes PPARγ activity, adipogenesis and insulin sensitivity in chronic-HFD and obesity.LTB4 promotes insulin resistance in obese mice by acting on macrophages, hepatocytes and myocytes.Oxidized LDL and Fructosamine Associated with Severity of Coronary Artery Atherosclerosis in Insulin Resistant Pigs Fed a High Fat/High NaCl Diet.Human Adenine Nucleotide Translocase (ANT) Modulators Identified by High-Throughput Screening of Transgenic Yeast.Stearic acid induces proinflammatory cytokine production partly through activation of lactate-HIF1α pathway in chondrocytes.Diffuse optical spectroscopic imaging of subcutaneous adipose tissue metabolic changes during weight loss.Beneficial metabolic activities of inflammatory cytokine interleukin 15 in obesity and type 2 diabetes.Limited OXPHOS capacity in white adipocytes is a hallmark of obesity in laboratory mice irrespective of the glucose tolerance status.SENP1-mediated NEMO deSUMOylation in adipocytes limits inflammatory responses and type-1 diabetes progression Hypoxia Potentiates Palmitate-induced Pro-inflammatory Activation of Primary Human Macrophages.Higher BNP levels within physiological range correlate with beneficial nonfasting lipid profiles in the elderly: a cross-sectional study Transient hypoxia reprograms differentiating adipocytes for enhanced insulin sensitivity and triglyceride accumulation Adipocyte-Specific Hypoxia-Inducible Factor 2α Deficiency Exacerbates Obesity-Induced Brown Adipose Tissue Dysfunction and Metabolic Dysregulation p75 Neurotrophin Receptor Regulates Energy Balance in Obesity.The effect of palmitate supplementation on gene expression profile in proliferating myoblasts Hypoxia-inducible factor 3A gene expression and methylation in adipose tissue is related to adipose tissue dysfunction.Intermedin Restores Hyperhomocysteinemia-induced Macrophage Polarization and Improves Insulin Resistance in Mice.Ginsenoside Rg5 Inhibits Succinate-Associated Lipolysis in Adipose Tissue and Prevents Muscle Insulin Resistance.Maternal obesity epigenetically alters visceral fat progenitor cell properties in male offspring mice.The Hypoxia-Inducible Factor Pathway in Adipocytes: The Role of HIF-2 in Adipose Inflammation and Hypertrophic Cardiomyopathy.Molecular and physiological roles of Pellino E3 ubiquitin ligases in immunity.Hypoxia in Obesity and Diabetes: Potential Therapeutic Effects of Hyperoxia and Nitrate.Phytochemicals in quinoa and amaranth grains and their antioxidant, anti-inflammatory, and potential health beneficial effects: a review.Biological plausibility linking sleep apnoea and metabolic dysfunction.Geographical variation in the progression of type 2 diabetes in Peru: The CRONICAS Cohort Study.

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P2860

Increased adipocyte O2 consumption triggers HIF-1α, causing inflammation and insulin resistance in obesity.

http://www.wikidata.org/entity/Q33965673

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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J.CELL.2014.05.012

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Increased adipocyte O2 consump ...... insulin resistance in obesity.

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Ai Chen

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Da Young Oh

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Heekyung Chung

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Olivia Osborne

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Oswald Quehenberger

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Roman Sasik

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Steven M Watkins

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P2860

Adipocyte NCoR knockout decreases PPARγ phosphorylation and enhances PPARγ activity and insulin sensitivity

The immune system's involvement in obesity-driven type 2 diabetes

HIF1α and HIF2α: sibling rivalry in hypoxic tumour growth and progression

Effects of peroxisome proliferator-activated receptor delta on placentation, adiposity, and colorectal cancer.

Transcriptional cofactor TBLR1 controls lipid mobilization in white adipose tissue

Dysregulation of adipose glutathione peroxidase 3 in obesity contributes to local and systemic oxidative stress.

Differential activation and antagonistic function of HIF-{alpha} isoforms in macrophages are essential for NO homeostasis.

Adipocytokine orosomucoid integrates inflammatory and metabolic signals to preserve energy homeostasis by resolving immoderate inflammation.

Splanchnic lipolysis in human obesity.

Kdo2-lipid A, a TLR4-specific agonist, induces de novo sphingolipid biosynthesis in RAW264.7 macrophages, which is essential for induction of autophagy

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Randall S. Johnson

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