Genetic ablation of the amplified-in-breast cancer 1 inhibits spontaneous prostate cancer progression in mice.
about
Mechanisms of resistance in castration-resistant prostate cancer (CRPC)Genetically engineered mouse models of prostate cancerIdentification of SRC3/AIB1 as a Preferred Coactivator for Hormone-activated Androgen ReceptorSRC-3 has a role in cancer other than as a nuclear receptor coactivator.SRC-3Delta4 mediates the interaction of EGFR with FAK to promote cell migrationSYT-SSX1 (synovial sarcoma translocated) regulates PIASy ligase activity to cause overexpression of NCOA3 proteinKnockout of SRC-1 and SRC-3 in Mice Decreases Cardiomyocyte Proliferation and Causes a Noncompaction Cardiomyopathy PhenotypeNCOA1 Directly Targets M-CSF1 Expression to Promote Breast Cancer Metastasis.Normal and cancer-related functions of the p160 steroid receptor co-activator (SRC) familyThe steroid receptor coactivator-3 is required for developing neuroendocrine tumor in the mouse prostateThe emerging role of speckle-type POZ protein (SPOP) in cancer development.Amplified in breast cancer 1 promotes colorectal cancer progression through enhancing notch signaling.Oncogenic activation in prostate cancer progression and metastasis: Molecular insights and future challenges.Nkx3.1 functions as para-transcription factor to regulate gene expression and cell proliferation in non-cell autonomous manner.Generation and validation of a mouse line with a floxed SRC-3/AIB1 allele for conditional knockout.Steroid receptor coactivator-3 as a potential molecular target for cancer therapy.The nuclear receptor coactivator amplified in breast cancer-1 is required for Neu (ErbB2/HER2) activation, signaling, and mammary tumorigenesis in mice.Interleukin-17 promotes formation and growth of prostate adenocarcinoma in mouse models.The AIB1 oncogene promotes breast cancer metastasis by activation of PEA3-mediated matrix metalloproteinase 2 (MMP2) and MMP9 expressionPan-cancer analyses of the nuclear receptor superfamilyThe steroid receptor coactivator-3 is required for the development of castration-resistant prostate cancerThe role of SRC-1 in murine prostate cancinogenesis is nonessential due to a possible compensation of SRC-3/AIB1 overexpressionRetinoid metabolism and ALDH1A2 (RALDH2) expression are altered in the transgenic adenocarcinoma mouse prostate modelGenistein and resveratrol, alone and in combination, suppress prostate cancer in SV-40 tag rats.Modeling prostate cancer in mice: something old, something new, something premalignant, something metastatic.Developmental and androgenic regulation of chromatin regulators EZH2 and ANCCA/ATAD2 in the prostate Via MLL histone methylase complex.Overexpression of AIB1 correlates inversely with E-cadherin expression in pancreatic adenocarcinoma and may promote lymph node metastasis.
P2860
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P2860
Genetic ablation of the amplified-in-breast cancer 1 inhibits spontaneous prostate cancer progression in mice.
description
2007 nî lūn-bûn
@nan
2007 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Genetic ablation of the amplif ...... te cancer progression in mice.
@ast
Genetic ablation of the amplif ...... te cancer progression in mice.
@en
type
label
Genetic ablation of the amplif ...... te cancer progression in mice.
@ast
Genetic ablation of the amplif ...... te cancer progression in mice.
@en
prefLabel
Genetic ablation of the amplif ...... te cancer progression in mice.
@ast
Genetic ablation of the amplif ...... te cancer progression in mice.
@en
P2093
P2860
P1433
P1476
Genetic ablation of the amplif ...... te cancer progression in mice.
@en
P2093
Arthur C-K Chung
Jean Ching-Yi Tien
Jianming Xu
Norman M Greenberg
Suoling Zhou
P2860
P304
P356
10.1158/0008-5472.CAN-06-3168
P407
P577
2007-06-01T00:00:00Z