Selective inactivation of p53 facilitates mouse epithelial tumor progression without chromosomal instability.
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pRb inactivation in mammary cells reveals common mechanisms for tumor initiation and progression in divergent epitheliaExcessive centrosome abnormalities without ongoing numerical chromosome instability in a Burkitt's lymphomaGrowth hormone-secreting tumors: genetic aspects and data from animal modelsA mouse model of human oral-esophageal cancer.Apoptosis is the essential target of selective pressure against p53, whereas loss of additional p53 functions facilitates carcinoma progression.Telomeres, stem cells, senescence, and cancer.Tumor escape in a Wnt1-dependent mouse breast cancer model is enabled by p19Arf/p53 pathway lesions but not p16 Ink4a loss.Inactivation of SNF5 cooperates with p53 loss to accelerate tumor formation in Snf5(+/-);p53(+/-) mice.Establishment and characterization of MRT cell lines from genetically engineered mouse models and the influence of genetic background on their development.Inactivation of gadd45a sensitizes epithelial cancer cells to ionizing radiation in vivo resulting in prolonged survival.Morphologic manifestations of gene-specific molecular alterations ("genetic addictions") in mouse models of disease.Unravelling mechanisms of p53-mediated tumour suppression.Magnetic resonance angiography visualization of abnormal tumor vasculature in genetically engineered mice.p19(ARF) is dispensable for oncogenic stress-induced p53-mediated apoptosis and tumor suppression in vivo.Attenuated p53 activation in tumour-associated stromal cells accompanies decreased sensitivity to etoposide and vincristine.Kaposi's sarcoma-associated herpesvirus-encoded latency-associated nuclear antigen induces chromosomal instability through inhibition of p53 function.Computer-assisted measurement of vessel shape from 3T magnetic resonance angiography of mouse brainMalignancy-associated vessel tortuosity: a computer-assisted, MR angiographic study of choroid plexus carcinoma in genetically engineered mice.Analysis of genetic damage and gene polymorphism in hepatocellular carcinoma (HCC) patients in a South Indian population.Deciphering cancer complexities in genetically engineered mice.
P2860
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P2860
Selective inactivation of p53 facilitates mouse epithelial tumor progression without chromosomal instability.
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Selective inactivation of p53 ...... thout chromosomal instability.
@ast
Selective inactivation of p53 ...... thout chromosomal instability.
@en
type
label
Selective inactivation of p53 ...... thout chromosomal instability.
@ast
Selective inactivation of p53 ...... thout chromosomal instability.
@en
prefLabel
Selective inactivation of p53 ...... thout chromosomal instability.
@ast
Selective inactivation of p53 ...... thout chromosomal instability.
@en
P2093
P2860
P1476
Selective inactivation of p53 ...... thout chromosomal instability.
@en
P2093
P2860
P304
P356
10.1128/MCB.21.17.6017-6030.2001
P407
P577
2001-09-01T00:00:00Z