Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response. - Wikidata - awgldk - TriplyDB

Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response.

Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response.

http://www.wikidata.org/entity/Q33979882

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Multiple immune factors are involved in controlling acute and chronic chikungunya virus infection The Roles of RNase-L in Antimicrobial Immunity and the Cytoskeleton-Associated Innate Response Role of type I interferons in the activation of autoreactive B cells Immunogenicity and protective capacity of a virosomal respiratory syncytial virus vaccine adjuvanted with monophosphoryl lipid A in mice Programming the magnitude and persistence of antibody responses with innate immunity.Infergen Stimulated Macrophages Restrict Mycobacterium tuberculosis Growth by Autophagy and Release of Nitric Oxide.Type I interferon signaling is required for the APOBEC3/Rfv3-dependent neutralizing antibody response but not innate retrovirus restriction.Lysophosphatidic acid receptor 5 inhibits B cell antigen receptor signaling and antibody response An essential role of caspase 1 in the induction of murine lupus and its associated vascular damage Murine B cell development and antibody responses to model antigens are not impaired in the absence of the TNF receptor GITR.IFN-{gamma} produced by CD8 T cells induces T-bet-dependent and -independent class switching in B cells in responses to alum-precipitated protein vaccine.Sialylation of Campylobacter jejuni lipo-oligosaccharides: impact on phagocytosis and cytokine production in mice.Marginal zone B cells: virtues of innate-like antibody-producing lymphocytes.Laboratory animal models to study foot-and-mouth disease: a review with emphasis on natural and vaccine-induced immunity.Infection of equine monocyte-derived macrophages with an attenuated equine infectious anemia virus (EIAV) strain induces a strong resistance to the infection by a virulent EIAV strain.The loss of Gnai2 and Gnai3 in B cells eliminates B lymphocyte compartments and leads to a hyper-IgM like syndrome.A palindromic CpG-containing phosphodiester oligodeoxynucleotide as a mucosal adjuvant stimulates plasmacytoid dendritic cell-mediated T(H)1 immunity.B cells enhance early innate immune responses during bacterial sepsis.Systemic responses during local viral infections: type I IFNs sound the alarm.Antigen-specific transforming growth factor β-induced Treg cells, but not natural Treg cells, ameliorate autoimmune arthritis in mice by shifting the Th17/Treg cell balance from Th17 predominance to Treg cell predominance.STING Negatively Regulates Double-Stranded DNA-Activated JAK1-STAT1 Signaling via SHP-1/2 in B Cells.Effects of adjuvants on IgG subclasses elicited by virus-like particles.B cell maintenance of subcapsular sinus macrophages protects against a fatal viral infection independent of adaptive immunity B Cell-Intrinsic IDO1 Regulates Humoral Immunity to T Cell-Independent Antigens.Macrophages induce differentiation of plasma cells through CXCL10/IP-10 TLR9 and STING agonists synergistically induce innate and adaptive type-II IFN.Presentation of type B peptide-MHC complexes from hen egg white lysozyme by TLR ligands and type I IFNs independent of H2-DM regulation.B-cell selection and the development of autoantibodies.HIV-1 gp140 epitope recognition is influenced by immunoglobulin DH gene segment sequence.The Vaccine Adjuvant Chitosan Promotes Cellular Immunity via DNA Sensor cGAS-STING-Dependent Induction of Type I Interferons TLR3- and MyD88-dependent signaling differentially influences the development of West Nile virus-specific B cell responses in mice following immunization with RepliVAX WN, a single-cycle flavivirus vaccine candidate Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection.STAT1 regulates marginal zone B cell differentiation in response to inflammation and infection with blood-borne bacteria.Division of labor during primary humoral immunity.Beyond passive immunization: toward a nanoparticle-based IL-17 vaccine as first in class of future immune treatments.Breaching peripheral tolerance promotes the production of HIV-1-neutralizing antibodies.Programmed cell death 1 suppresses B-1b cell expansion and long-lived IgG production in response to T cell-independent type 2 antigens.Pathogenesis of Human Systemic Lupus Erythematosus: A Cellular Perspective.FcγRIIB prevents inflammatory type I IFN production from plasmacytoid dendritic cells during a viral memory response.Spontaneous germinal centers and autoimmunity.

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Type I IFN enhances follicular B cell contribution to the T cell-independent antibody response.

http://www.wikidata.org/entity/Q33979882

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

@wuu

name

Type I IFN enhances follicular ...... independent antibody response.

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Type I IFN enhances follicular ...... independent antibody response.

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type

label

Type I IFN enhances follicular ...... independent antibody response.

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Type I IFN enhances follicular ...... independent antibody response.

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prefLabel

Type I IFN enhances follicular ...... independent antibody response.

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Type I IFN enhances follicular ...... independent antibody response.

@en

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P1433

Journal of Experimental Medicine

P1476

Type I IFN enhances follicular ...... independent antibody response.

@en

P2093

Cristina L Swanson

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Pamela Strauch

http://www.w3.org/2001/XMLSchema#string

Raul M Torres

http://www.w3.org/2001/XMLSchema#string

Roberta Pelanda

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P2860

Functional role of type I and type II interferons in antiviral defense

Length-dependent recognition of double-stranded ribonucleic acids by retinoic acid-inducible gene-I and melanoma differentiation-associated gene 5

Functional heterogeneity of marginal zone B cells revealed by their ability to generate both early antibody-forming cells and germinal centers with hypermutation and memory in response to a T-dependent antigen

Adjuvant-enhanced antibody responses in the absence of toll-like receptor signaling

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

Marginal-zone B cells

Autoreactivity by design: innate B and T lymphocytes

B-1a and B-1b cells exhibit distinct developmental requirements and have unique functional roles in innate and adaptive immunity to S. pneumoniae

Approaching the asymptote? Evolution and revolution in immunology

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1485-1500

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scholarly article

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10.1084/JEM.20092695

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7

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207

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Timothy J. Wilson

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2010-06-21T00:00:00Z

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44691183

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20566717

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2901065

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