Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function. - Wikidata - awgldk - TriplyDB

Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.

Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.

http://www.wikidata.org/entity/Q33979918

about

Myeloid-Derived Suppressor Cells in Bacterial Infections Liver immunology and its role in inflammation and homeostasis Hepatocytes: a key cell type for innate immunity The Tumor Macroenvironment: Cancer-Promoting Networks Beyond Tumor Beds The role of the liver in sepsis Regulation of tumor metastasis by myeloid-derived suppressor cells Differential role of gp130-dependent STAT and Ras signalling for haematopoiesis following bone-marrow transplantation The digestive tract as the origin of systemic inflammation Invariant NKT cells modulate the suppressive activity of IL-10-secreting neutrophils differentiated with serum amyloid A Coordinated regulation of myeloid cells by tumours Hepatocyte growth factor modulates interleukin-6 production in bone marrow derived macrophages: implications for inflammatory mediated diseases Myeloid-derived suppressor cells: the dark knight or the joker in viral infections?Myeloid-derived suppressor cells in sepsis Interleukin-6 induces Gr-1+CD11b+ myeloid cells to suppress CD8+ T cell-mediated liver injury in mice Role of myeloid-derived suppressor cells in amelioration of experimental autoimmune hepatitis following activation of TRPV1 receptors by cannabidiol Gp130-dependent release of acute phase proteins is linked to the activation of innate immune signaling pathways.Aryl hydrocarbon receptor control of a disease tolerance defence pathway MicroRNA 21 (miR-21) and miR-181b couple with NFI-A to generate myeloid-derived suppressor cells and promote immunosuppression in late sepsis Neutrophils with myeloid derived suppressor function deplete arginine and constrain T cell function in septic shock patients.A paradoxical role for myeloid-derived suppressor cells in sepsis and trauma The calcineurin-NFAT axis controls allograft immunity in myeloid-derived suppressor cells through reprogramming T cell differentiation Protein tyrosine phosphatase 1B deficiency ameliorates murine experimental colitis via the expansion of myeloid-derived suppressor cells Chronic psychological stress induces the accumulation of myeloid-derived suppressor cells in mice.Anti-HMGB1 monoclonal antibody ameliorates immunosuppression after peripheral tissue trauma: attenuated T-lymphocyte response and increased splenic CD11b (+) Gr-1 (+) myeloid-derived suppressor cells require HMGB1.Long-term effects of subacute ruminal acidosis (SARA) on milk quality and hepatic gene expression in lactating goats fed a high-concentrate diet Myeloid-derived suppressor cells: paradoxical roles in infection and immunity.Zinc regulates the acute phase response and serum amyloid A production in response to sepsis through JAK-STAT3 signaling Dynamics of lung defense in pneumonia: resistance, resilience, and remodeling.The Role and Potential Therapeutic Application of Myeloid-Derived Suppressor Cells in Allo- and Autoimmunity Metabolic regulation of hepatitis B immunopathology by myeloid-derived suppressor cells.Innate immunity in acute HIV-1 infection Hepatocyte-specific mutation of both NF-κB RelA and STAT3 abrogates the acute phase response in mice IL-10 immunomodulation of myeloid cells regulates a murine model of ovarian cancer.Serum Amyloid A Induces Inflammation, Proliferation and Cell Death in Activated Hepatic Stellate Cells.Adaptive immunity suppresses formation and progression of diethylnitrosamine-induced liver cancer Epigenetic modifiers reduce inflammation and modulate macrophage phenotype during endotoxemia-induced acute lung injury.An Immature Myeloid/Myeloid-Suppressor Cell Response Associated with Necrotizing Inflammation Mediates Lethal Pulmonary Tularemia.Myeloid-derived suppressor cells evolve during sepsis and can enhance or attenuate the systemic inflammatory response.The Lung-Liver Axis: A Requirement for Maximal Innate Immunity and Hepatoprotection during Pneumonia.Activation of Hepatic STAT3 Maintains Pulmonary Defense during Endotoxemia

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Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function.

http://www.wikidata.org/entity/Q33979918

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

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name

Hepatic acute-phase proteins c ...... ived suppressor cell function.

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Hepatic acute-phase proteins c ...... ived suppressor cell function.

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type

label

Hepatic acute-phase proteins c ...... ived suppressor cell function.

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Hepatic acute-phase proteins c ...... ived suppressor cell function.

@en

prefLabel

Hepatic acute-phase proteins c ...... ived suppressor cell function.

@ast

Hepatic acute-phase proteins c ...... ived suppressor cell function.

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Journal of Experimental Medicine

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Hepatic acute-phase proteins c ...... rived suppressor cell function

@en

P2093

Christian Trautwein

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Frank Tacke

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J Magarian Blander

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Reinhold P Linke

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Sara Dutton Sackett

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Uta Dierssen

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P2860

Serum amyloid A is a chemoattractant: induction of migration, adhesion, and tissue infiltration of monocytes and polymorphonuclear leukocytes

Inhibition of dendritic cell differentiation and accumulation of myeloid-derived suppressor cells in cancer is regulated by S100A9 protein

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Myeloid-derived suppressor cells as regulators of the immune system

Serum amyloid A is an innate immune opsonin for Gram-negative bacteria

IL-6-induced homodimerization of gp130 and associated activation of a tyrosine kinase

Cutting edge: TLR2 is a functional receptor for acute-phase serum amyloid A

Acute-phase proteins and other systemic responses to inflammation

Recognition of microorganisms and activation of the immune response

The pathophysiology and treatment of sepsis

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10.1084/JEM.20091474

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7

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207

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Michael Müller

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2010-06-07T00:00:00Z

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44655952

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20530204

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2901069

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