EBV latency types adopt alternative chromatin conformations. - Wikidata - awgldk - TriplyDB

EBV latency types adopt alternative chromatin conformations.

EBV latency types adopt alternative chromatin conformations.

http://www.wikidata.org/entity/Q33987454

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Keeping it quiet: chromatin control of gammaherpesvirus latency Targeting CTCF to Control Virus Gene Expression: A Common Theme amongst Diverse DNA Viruses EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1 Epigenetic regulation of EBV persistence and oncogenesis.PARP1 restricts Epstein Barr Virus lytic reactivation by binding the BZLF1 promoter Snapshots: chromatin control of viral infection.Interpreting the Epstein-Barr Virus (EBV) epigenome using high-throughput data Cis and trans acting factors involved in human cytomegalovirus experimental and natural latent infection of CD14 (+) monocytes and CD34 (+) cells.Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming.CCCTC-binding factor recruitment to the early region of the human papillomavirus 18 genome regulates viral oncogene expression.Host genetics of Epstein-Barr virus infection, latency and disease.Genome wide nucleosome mapping for HSV-1 shows nucleosomes are deposited at preferred positions during lytic infection.Human papillomaviruses activate and recruit SMC1 cohesin proteins for the differentiation-dependent life cycle through association with CTCF insulators Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.Insulators, long-range interactions, and genome function Global Transcriptome Analysis Reveals That Poly(ADP-Ribose) Polymerase 1 Regulates Gene Expression through EZH2.CTCF occupation of the herpes simplex virus 1 genome is disrupted at early times postreactivation in a transcription-dependent manner.Telomeres and viruses: common themes of genome maintenance.The open chromatin landscape of Kaposi's sarcoma-associated herpesvirus Epstein-Barr Virus Oncoprotein LMP1 Mediates Epigenetic Changes in Host Gene Expression through PARP1.Epigenetic deregulation of the LMP1/LMP2 locus of Epstein-Barr virus by mutation of a single CTCF-cohesin binding site.CTCF interacts with the lytic HSV-1 genome to promote viral transcription.Adenovirus-based vaccines against rhesus lymphocryptovirus EBNA-1 induce expansion of specific CD8+ and CD4+ T cells in persistently infected rhesus macaques.Epigenetic regulation of EBV and KSHV latency Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation.Host-virus interactions: from the perspectives of epigenetics.The insulator protein CTCF binding sites in the orf73/LANA promoter region of herpesvirus saimiri are involved in conferring episomal stability in latently infected human T cells.Genipin as a novel chemical activator of EBV lytic cycle.Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases.3D genomics imposes evolution of the domain model of eukaryotic genome organization.DNA replication-dependent binding of CTCF plays a critical role in adenovirus genome functions.Contributions of CTCF and DNA methyltransferases DNMT1 and DNMT3B to Epstein-Barr virus restricted latency.Epstein-Barr virus: a master epigenetic manipulator.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.HCF1 and OCT2 Cooperate with EBNA1 To Enhance OriP-Dependent Transcription and Episome Maintenance of Latent Epstein-Barr Virus.Epstein-Barr virus-host cell interactions: an epigenetic dialog?Novel STIM1-dependent control of Ca2+ clearance regulates NFAT activity during T-cell activation.The Epstein-Barr virus episome maneuvers between nuclear chromatin compartments during reactivation.Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naïve B cells, and facilitates recruitment of transcription factors to the viral genome.EBNA1: Oncogenic Activity, Immune Evasion and Biochemical Functions Provide Targets for Novel Therapeutic Strategies against Epstein-Barr Virus- Associated Cancers.

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EBV latency types adopt alternative chromatin conformations.

http://www.wikidata.org/entity/Q33987454

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

EBV latency types adopt alternative chromatin conformations.

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EBV latency types adopt alternative chromatin conformations.

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type

label

EBV latency types adopt alternative chromatin conformations.

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EBV latency types adopt alternative chromatin conformations.

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prefLabel

EBV latency types adopt alternative chromatin conformations.

@ast

EBV latency types adopt alternative chromatin conformations.

@en

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EBV latency types adopt alternative chromatin conformations.

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Italo Tempera

http://www.w3.org/2001/XMLSchema#string

Michael Klichinsky

http://www.w3.org/2001/XMLSchema#string

Paul M Lieberman

http://www.w3.org/2001/XMLSchema#string

P2860

ORC binding to TRF2 stimulates OriP replication

An exceptionally conserved transcriptional repressor, CTCF, employs different combinations of zinc fingers to bind diverged promoter sequences of avian and mammalian c-myc oncogenes

CTCF: master weaver of the genome

Mediator and cohesin connect gene expression and chromatin architecture

Capturing chromosome conformation

CTCF is a uniquely versatile transcription regulator linked to epigenetics and disease

Epigenotypes of latent herpesvirus genomes

A novel sequence-specific DNA binding protein which interacts with three regularly spaced direct repeats of the CCCTC-motif in the 5'-flanking sequence of the chicken c-myc gene

Nuclear organization of the genome and the potential for gene regulation

CTCF mediates long-range chromatin looping and local histone modification in the beta-globin locus

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e1002180

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scholarly article

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10.1371/JOURNAL.PPAT.1002180

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7

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21829357

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3145795

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