MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
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MicroRNA Expression in β-Thalassemia and Sickle Cell Disease: A Role in The Induction of Fetal HemoglobinmiRNAs as Biomarkers in Chronic Myelogenous LeukemiaReciprocal regulation of γ-globin expression by exo-miRNAs: Relevance to γ-globin silencing in β-thalassemia major.3'LIFE: a functional assay to detect miRNA targets in high-throughputMicroRNA-486-3p regulates γ-globin expression in human erythroid cells by directly modulating BCL11A.Detection of miRNA Targets in High-throughput Using the 3'LIFE AssayInfluence of microRNA on the maintenance of human iron metabolismLIN28B-mediated expression of fetal hemoglobin and production of fetal-like erythrocytes from adult human erythroblasts ex vivoOriginal Research: Stable expression of miR-34a mediates fetal hemoglobin induction in K562 cells.A feedback loop consisting of microRNA 23a/27a and the β-like globin suppressors KLF3 and SP1 regulates globin gene expressionMicroRNAs as Haematopoiesis Regulators.The up-regulation of miR-199b-5p in erythroid differentiation is associated with GATA-1 and NF-E2.MicroRNAs as components of regulatory networks controlling erythropoiesis.Recent trends for novel options in experimental biological therapy of β-thalassemia.Recent trends in the gene therapy of β-thalassemia.Modulation of microRNAs expression in hematopoietic stem cells treated with sodium butyrate in inducing fetal hemoglobin expression.N-methyl-D-aspartate receptors in human erythroid precursor cells and in circulating red blood cells contribute to the intracellular calcium regulation.Genomic conservation of erythropoietic microRNAs (erythromiRs) in white-blooded Antarctic icefish.Fetal hemoglobin in sickle cell anemia: The Arab-Indian haplotype and new therapeutic agents.A candidate transacting modulator of fetal hemoglobin gene expression in the Arab-Indian haplotype of sickle cell anemia.Elevated levels of miR-210 correlate with anemia in β-thalassemia/HbE patients.Comparison of MicroRNAs Mediated in Reactivation of the γ-Globin in β-Thalassemia Patients, Responders and Non-Responders to Hydroxyurea.Genetic disruption of the KLF1 gene to overexpress the γ-globin gene using the CRISPR/Cas9 system.Orphan Drugs and Potential Novel Approaches for Therapies of β-Thalassemia: Current Status and Future Expectations
P2860
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P2860
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@ast
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@en
type
label
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@ast
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@en
prefLabel
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@ast
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@en
P2093
P2860
P1433
P1476
MicroRNA-96 directly inhibits γ-globin expression in human erythropoiesis.
@en
P2093
Bernd Wollscheid
Hansjoerg Moest
Imane Azzouzi
Jean-Claude Fauchère
Jeannine Winkler
Markus Schmugge
Markus Stoffel
Oliver Speer
P2860
P304
P356
10.1371/JOURNAL.PONE.0022838
P407
P577
2011-07-28T00:00:00Z