Mismatch repair polymorphisms and risk of colon cancer, tumour microsatellite instability and interactions with lifestyle factors - Wikidata - awgldk - TriplyDB

Mismatch repair polymorphisms and risk of colon cancer, tumour microsatellite instability and interactions with lifestyle factors

Mismatch repair polymorphisms and risk of colon cancer, tumour microsatellite instability and interactions with lifestyle factors

http://www.wikidata.org/entity/Q33990340

about

Interdisciplinary education to integrate pathology and epidemiology: towards molecular and population-level health science Molecular pathological epidemiology of colorectal neoplasia: an emerging transdisciplinary and interdisciplinary field Lifestyle factors and microsatellite instability in colorectal cancer: the evolving field of molecular pathological epidemiology Etiologic field effect: reappraisal of the field effect concept in cancer predisposition and progression Statistical methods for studying disease subtype heterogeneity Impact of DNA repair gene polymorphisms on the risk of biochemical recurrence after radiotherapy and overall survival in prostate cancer.Specific variants in the MLH1 gene region may drive DNA methylation, loss of protein expression, and MSI-H colorectal cancer.Smoking, gender, and ethnicity predict somatic BRAF mutations in colorectal cancer.MSH6 G39E polymorphism and CpG island methylator phenotype in colon cancer Application of molecular diagnostics for the detection of Lynch syndrome.Concept of chemoprevention in colorectal cancer.Report of a Novel Mutation in MLH1 Gene in a Hispanic Family from Puerto Rico Fulfilling Classic Amsterdam Criteria for Lynch Syndrome Association between MLH1 -93G>a polymorphism and risk of colorectal cancer.Haplotype defined by the MLH1-93G/A polymorphism is associated with MLH1 promoter hypermethylation in sporadic colorectal cancers.DNA repair gene polymorphisms and tobacco smoking in the risk for colorectal adenomas Germline mutation analysis of MLH1 and MSH2 in Malaysian Lynch syndrome patients.A Meta-Regression Method for Studying Etiological Heterogeneity Across Disease Subtypes Classified by Multiple Biomarkers.Next-generation sequencing for genetic testing of familial colorectal cancer syndromes.Clinical characterization and mutation spectrum in Caribbean Hispanic families with Lynch syndrome.Genetic architecture of colorectal cancer.MLH1 methylation screening is effective in identifying epimutation carriers CYP2C9 variants increase risk of colorectal adenoma recurrence and modify associations with smoking but not aspirin treatment.Bladder cancer SNP panel predicts susceptibility and survival A promoter polymorphism in the hMLH1 gene (-93G/A) associated with sporadic colorectal cancer Early-onset colorectal cancer patients without family history are "at very low risk" for lynch syndrome.Recent advances in understanding the role of diet and obesity in the development of colorectal cancer.100 years Lynch syndrome: what have we learned about psychosocial issues?Molecular Alterations of Colorectal Cancer with Inflammatory Bowel Disease.Functional analysis of MSH2 unclassified variants found in suspected Lynch syndrome patients reveals pathogenicity due to attenuated mismatch repair.Stable expression of MutLγ in human cells reveals no specific response to mismatched DNA, but distinct recruitment to damage sites.Gene fusions by chromothripsis of chromosome 5q in the VCaP prostate cancer cell line.Association between MSH6 G39E polymorphism and cancer susceptibility: a meta-analysis of 7,046 cases and 34,554 controls.Association between MutL homolog 1 polymorphisms and the risk of colorectal cancer: a meta-analysis.Investigation of the effects of DNA repair gene polymorphisms on the risk of colorectal cancer.Association of DCC, MLH1, GSTT1, GSTM1, and TP53 gene polymorphisms with colorectal cancer in Kazakhstan.Pooling-analysis on hMLH1 polymorphisms and cancer risk: evidence based on 31,484 cancer cases and 45,494 cancer-free controls.Single nucleotide polymorphisms in MLH1 predict poor prognosis of hepatocellular carcinoma in a Chinese population.Polymorphism of MSH2 Gly322Asp and MLH1 -93G>A in non-familial colon cancer - a case-controlled study.The intricate interplay between MSI and polymorphisms of DNA repair enzymes in gastric cancer H.pylori associated.Gene Environment Interactions and Predictors of Colorectal Cancer in Family-Based, Multi-Ethnic Groups.

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Mismatch repair polymorphisms and risk of colon cancer, tumour microsatellite instability and interactions with lifestyle factors

http://www.wikidata.org/entity/Q33990340

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P2860

Multiple sequence motifs are involved in SV40 enhancer function.

A member of the C/EBP family, NF-IL6 beta, forms a heterodimer and transcriptionally synergizes with NF-IL6

Molecular models for the tissue specificity of DNA mismatch repair-deficient carcinogenesis

Germ-line msh6 mutations in colorectal cancer families

A Western-style diet induces benign and malignant neoplasms in the colon of normal C57Bl/6 mice

Mismatch repair polymorphisms and colorectal polyps: hMLH1-93G>A variant modifies risk associated with smoking

Disruption of an exon splicing enhancer in exon 3 of MLH1 is the cause of HNPCC in a Quebec family

DNA mismatch repair

Dietary modulation of carcinoma development in a mouse model for human familial adenomatous polyposis

SIFT: Predicting amino acid changes that affect protein function

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