Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease. - Wikidata - awgldk - TriplyDB

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

http://www.wikidata.org/entity/Q33997514

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Genetic and Transcriptomic Profiles of Inflammation in Neurodegenerative Diseases: Alzheimer, Parkinson, Creutzfeldt-Jakob and Tauopathies Altered Ca2+ homeostasis induces Calpain-Cathepsin axis activation in sporadic Creutzfeldt-Jakob disease Complex Inflammation mRNA-Related Response in ALS Is Region Dependent Cystatin F is a biomarker of prion pathogenesis in mice.Proteomics applications in prion biology and structure.Roles of microglia in brain development, tissue maintenance and repair.The Role of Microglia in Prion Diseases: A Paradigm of Functional Diversity.Inflammation in Lafora Disease: Evolution with Disease Progression in Laforin and Malin Knock-out Mouse Models.A roadmap for investigating the role of the prion protein in depression associated with neurodegenerative disease YKL-40 in the brain and cerebrospinal fluid of neurodegenerative dementias.Neuroinflammatory Gene Regulation, Mitochondrial Function, Oxidative Stress, and Brain Lipid Modifications With Disease Progression in Tau P301S Transgenic Mice as a Model of Frontotemporal Lobar Degeneration-Tau.Regional and subtype-dependent miRNA signatures in sporadic Creutzfeldt-Jakob disease are accompanied by alterations in miRNA silencing machinery and biogenesis.Sisyphus in Neverland.Fluorodeoxyglucose Positron Emission Tomography (FDG-PET) Correlation of Histopathology and MRI in Prion Disease.Identification of new molecular alterations in fatal familial insomnia.Diversity of astroglial responses across human neurodegenerative disorders and brain aging.Molecular Alterations in the Cerebellum of Sporadic Creutzfeldt-Jakob Disease Subtypes with DJ-1 as a Key Regulator of Oxidative Stress.Limited Unfolded Protein Response and Inflammation in Neuroserpinopathy.Cytoskeleton-Associated Risk Modifiers Involved in Early and Rapid Progression of Sporadic Creutzfeldt-Jakob Disease.Altered Mitochondria, Protein Synthesis Machinery, and Purine Metabolism Are Molecular Contributors to the Pathogenesis of Creutzfeldt-Jakob Disease.

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P2860

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

http://www.wikidata.org/entity/Q33997514

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

@ast

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

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type

label

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

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Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

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prefLabel

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

@ast

Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

@en

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FNAGI.2014.00198

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Frontiers in Aging Neuroscience

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Subtype and regional-specific neuroinflammation in sporadic creutzfeldt-jakob disease.

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P2093

Franc Llorens

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Inga Zerr

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Irene López-González

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Isidre Ferrer

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Katrin Thüne

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Margarita Carmona

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Olivier Andréoletti

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Saima Zafar

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P2860

Signaling through the JAK/STAT pathway, recent advances and future challenges

Cyclooxygenase-2, prostaglandin E2, and microglial activation in prion diseases

The toll-like receptor agonist imiquimod is active against prions

Microglia-mediated neurotoxicity: uncovering the molecular mechanisms

Inducible cytokine gene expression in the brain in the ME7/CV mouse model of scrapie is highly restricted, is at a strikingly low level relative to the degree of gliosis and occurs only late in disease.

Intracerebral administration of interleukin-12 (IL-12) and IL-18 modifies the course of mouse scrapie

Genes contributing to prion pathogenesis

NF-kappaB in neuronal plasticity and neurodegenerative disorders

Prion pathogenesis in the absence of Toll-like receptor signalling

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198

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10.3389/FNAGI.2014.00198

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6

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2014-08-04T00:00:00Z

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