Effects of chitosan on intestinal inflammation in weaned pigs challenged by enterotoxigenic Escherichia coli. - Wikidata - awgldk - TriplyDB

Effects of chitosan on intestinal inflammation in weaned pigs challenged by enterotoxigenic Escherichia coli.

Effects of chitosan on intestinal inflammation in weaned pigs challenged by enterotoxigenic Escherichia coli.

http://www.wikidata.org/entity/Q34000229

about

Oral administration of synthetic porcine beta-defensin-2 improves growth performance and cecal microbial flora and down-regulates the expression of intestinal toll-like receptor-4 and inflammatory cytokines in weaned piglets challenged with enteroto Fatty acids, inflammation and intestinal health in pigs.Dietary Chitosan Supplementation Increases Microbial Diversity and Attenuates the Severity of Citrobacter rodentium Infection in Mice.Low Dosage of Chitosan Supplementation Improves Intestinal Permeability and Impairs Barrier Function in Mice.Chitosan Modulates Inflammatory Responses in Rats Infected with Enterotoxigenic Escherichia coli.L-Glutamine and L-arginine protect against enterotoxigenic Escherichia coli infection via intestinal innate immunity in mice.Asparagine improves intestinal integrity, inhibits TLR4 and NOD signaling, and differently regulates p38 and ERK1/2 signaling in weanling piglets after LPS challenge.Enterotoxigenic Escherichia coli is phagocytosed by macrophages underlying villus-like intestinal epithelial cells: modeling ex vivo innate immune defenses of the human gut.Low-Molecular-Weight Chitosan Supplementation Increases the Population of Prevotella in the Cecal Contents of Weanling Pigs.Combined effects of chitosan and microencapsulated Enterococcus faecalis CG1.0007 probiotic supplementation on performance and diarrhea incidences in enterotoxigenic Escherichia coli K88+ challenged piglets.Effects of low-molecular-weight chitosan on the growth performance, intestinal morphology, barrier function, cytokine expression and antioxidant system of weaned piglets.

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Effects of chitosan on intestinal inflammation in weaned pigs challenged by enterotoxigenic Escherichia coli.

http://www.wikidata.org/entity/Q34000229

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年學術文章

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name

Effects of chitosan on intesti ...... erotoxigenic Escherichia coli.

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Effects of chitosan on intesti ...... erotoxigenic Escherichia coli.

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Effects of chitosan on intesti ...... erotoxigenic Escherichia coli.

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Effects of chitosan on intesti ...... erotoxigenic Escherichia coli.

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JOURNAL.PONE.0104192

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Effects of chitosan on intesti ...... erotoxigenic Escherichia coli.

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Charles M Nyachoti

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Dingfu Xiao

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Maoliang Ran

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Sung Woo Kim

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Wei Qiu

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Xionggui Hu

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Yongfei Wang

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Yulong Yin

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Zemeng Feng

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Zhiru Tang

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P2860

The effect of water-soluble chitosan on macrophage activation and the attenuation of mite allergen-induced airway inflammation.

Daily exposure to dust alters innate immunity

Role of faecal calprotectin as non-invasive marker of intestinal inflammation.

Role of fecal calprotectin as a biomarker of intestinal inflammation in inflammatory bowel disease.

Effects of dietary administering chitosan on growth performance, jejunal morphology, jejunal mucosal sIgA, occludin, claudin-1 and TLR4 expression in weaned piglets challenged by enterotoxigenic Escherichia coli.

Exposure to inflammatory cytokines IL-1β and TNFα induces compromise and death of astrocytes; implications for chronic neuroinflammation.

Chitosan as antimicrobial agent: applications and mode of action.

Immunobiotic Lactobacillus jensenii elicits anti-inflammatory activity in porcine intestinal epithelial cells by modulating negative regulators of the Toll-like receptor signaling pathway

Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil priming

Escherichia coli in postweaning diarrhea in pigs: an update on bacterial types, pathogenesis, and prevention strategies.

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