Elevated expression levels of miR-143/5 in saphenous vein smooth muscle cells from patients with Type 2 diabetes drive persistent changes in phenotype and function.
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Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle DisordersAberrant phenotype in human endothelial cells of diabetic origin: implications for saphenous vein graft failure?microRNAs Distinctively Regulate Vascular Smooth Muscle and Endothelial Cells: Functional Implications in Angiogenesis, Atherosclerosis, and In-Stent Restenosis.Emerging roles of the myocardin family of proteins in lipid and glucose metabolism.Prognostic role of miR-200c in various malignancies: a systematic review and meta-analysis.Messenger RNA and MicroRNA transcriptomic signatures of cardiometabolic risk factors.MicroRNA-145 regulates platelet-derived growth factor-induced human aortic vascular smooth muscle cell proliferation and migration by targeting CD40.Phenotypic switching of vascular smooth muscle cells in the 'normal region' of aorta from atherosclerosis patients is regulated by miR-145An rs4705342 T>C polymorphism in the promoter of miR-143/145 is associated with a decreased risk of ischemic stroke.Mapping the methylation status of the miR-145 promoter in saphenous vein smooth muscle cells from individuals with type 2 diabetes.Vascular Smooth Muscle as a Target for Novel Therapeutics.Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy.Circulating miR-92a expression level in patients with essential hypertension: a potential marker of atherosclerosis.Circulating early- and mid-pregnancy microRNAs and risk of gestational diabetes.Advanced glycation end products impair the functions of saphenous vein but not thoracic artery smooth muscle cells through RAGE/MAPK signalling pathway in diabetes.MicroRNAs in the pathogenesis of type 2 diabetes: new research progress and future direction.Progressive Development of Aberrant Smooth Muscle Cell Phenotype in Abdominal Aortic Aneurysm Disease.miR-29a regulates vascular neointimal hyperplasia by targeting YY1.Phenotypic switching of vascular smooth muscle cells in animal model of rat thoracic aortic aneurysm.
P2860
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P2860
Elevated expression levels of miR-143/5 in saphenous vein smooth muscle cells from patients with Type 2 diabetes drive persistent changes in phenotype and function.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Elevated expression levels of ...... ges in phenotype and function.
@ast
Elevated expression levels of ...... ges in phenotype and function.
@en
Elevated expression levels of ...... ges in phenotype and function.
@en-gb
Elevated expression levels of ...... ges in phenotype and function.
@nl
type
label
Elevated expression levels of ...... ges in phenotype and function.
@ast
Elevated expression levels of ...... ges in phenotype and function.
@en
Elevated expression levels of ...... ges in phenotype and function.
@en-gb
Elevated expression levels of ...... ges in phenotype and function.
@nl
prefLabel
Elevated expression levels of ...... ges in phenotype and function.
@ast
Elevated expression levels of ...... ges in phenotype and function.
@en
Elevated expression levels of ...... ges in phenotype and function.
@en-gb
Elevated expression levels of ...... ges in phenotype and function.
@nl
P2093
P2860
P50
P1154
2-s2.0-84904761497
P1476
Elevated expression levels of ...... ges in phenotype and function.
@en
P2093
Aliah R Alshanwani
David J O'Regan
Philip Warburton
Stephen G Ball
P2860
P304
P356
10.1016/J.YJMCC.2014.05.018
P577
2014-06-10T00:00:00Z