Effects of stress and stress hormones on amyloid-beta protein and plaque deposition. - Wikidata - awgldk - TriplyDB

Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

http://www.wikidata.org/entity/Q34001966

about

Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy Cognitive and social lifestyle: links with neuropathology and cognition in late life Involvement of 5-lipoxygenase in the corticosteroid-dependent amyloid beta formation: in vitro and in vivo evidence Corticotropin-releasing factor receptor-1 antagonism mitigates beta amyloid pathology and cognitive and synaptic deficits in a mouse model of Alzheimer's disease.Cognitive functioning and late-life depression.Personality factors moderate the associations between apolipoprotein genotype and cognitive function as well as late onset Alzheimer disease.Brain neurotoxic amyloid-beta peptides: their potential role in the pathophysiology of depression and as molecular therapeutic targets.Why do we need to use animal models to study cognition and aging?Gene expression patterns in the hippocampus and amygdala of endogenous depression and chronic stress models.Is Dysregulation of the HPA-Axis a Core Pathophysiology Mediating Co-Morbid Depression in Neurodegenerative Diseases?Chronic stress exacerbates tau pathology, neurodegeneration, and cognitive performance through a corticotropin-releasing factor receptor-dependent mechanism in a transgenic mouse model of tauopathy Impact of CRFR1 Ablation on Amyloid-β Production and Accumulation in a Mouse Model of Alzheimer's Disease.Isolation Housing Exacerbates Alzheimer's Disease-Like Pathophysiology in Aged APP/PS1 Mice The association between financial hardship and amygdala and hippocampal volumes: results from the PATH through life project.Depression, hippocampal volume changes, and cognitive decline in a clinical sample of older depressed outpatients and non-depressed controls.Corticotrophin releasing factor accelerates neuropathology and cognitive decline in a mouse model of Alzheimer's disease.Lower CSF amyloid beta peptides and higher F2-isoprostanes in cognitively intact elderly individuals with major depressive disorder Reconsidering Animal Models of Major Depressive Disorder in the Elderly.Effects of aging and Alzheimer's disease along the longitudinal axis of the hippocampus.Integrative neurobiology of metabolic diseases, neuroinflammation, and neurodegeneration.Corticotropin-releasing factor receptor-1 modulates biomarkers of DNA oxidation in Alzheimer's disease mice.Stress and glucocorticoids increase transthyretin expression in rat choroid plexus via mineralocorticoid and glucocorticoid receptors.Retinoic acid receptor-α signalling antagonizes both intracellular and extracellular amyloid-β production and prevents neuronal cell death caused by amyloid-β.Neuropsychological functioning in the acute and remitted States of late-life depression.Insight into glutamate excitotoxicity from synaptic zinc homeostasis Influences of yokukansankachimpihange on aggressive behavior of zinc-deficient mice and actions of the ingredients on excessive neural exocytosis in the hippocampus of zinc-deficient rats.Assessing competence of broccoli consumption on inflammatory and antioxidant pathways in restraint-induced models: estimation in rat hippocampus and prefrontal cortex.Social Relations at Work and Incident Dementia: 29-Years' Follow-Up of the Copenhagen Male Study.Restraint stress in rats alters gene transcription and protein translation in the hippocampus.Citalopram Ameliorates Synaptic Plasticity Deficits in Different Cognition-Associated Brain Regions Induced by Social Isolation in Middle-Aged Rats.Long-term cortisol measures predict Alzheimer disease risk.Interactions between stress and physical activity on Alzheimer's disease pathology.Sex differences in chronic stress responses and Alzheimer's disease.A preclinical perspective on the enhanced vulnerability to Alzheimer's disease after early-life stress.Chronic stress as a risk factor for Alzheimer's disease: Roles of microglia-mediated synaptic remodeling, inflammation, and oxidative stress.

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P2860

Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

http://www.wikidata.org/entity/Q34001966

description

2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009 թվականի հունվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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Journal of Alzheimer's Disease

P1476

Effects of stress and stress hormones on amyloid-beta protein and plaque deposition.

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P2093

Hongxin Dong

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John G Csernansky

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P2860

A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Effects of aging and stress on hippocampal structure and function

Displacement of corticotropin releasing factor from its binding protein as a possible treatment for Alzheimer's disease

Alzheimer's disease: genes, proteins, and therapy

Physiology and neurobiology of stress and adaptation: central role of the brain

Regulation of NMDA receptor trafficking by amyloid-beta

Corticotropin-releasing factor and Urocortin I modulate excitatory glutamatergic synaptic transmission

Maternal stress produces learning deficits associated with impairment of NMDA receptor-mediated synaptic plasticity

APP processing and synaptic function

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459-469

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scholarly article

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10.3233/JAD-2009-1152

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2

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18

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2905685

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