Intracellular infection by the human granulocytic ehrlichiosis agent inhibits human neutrophil apoptosis.
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Effects of Anaplasma phagocytophila on NADPH oxidase components in human neutrophils and HL-60 cells.Type IV secretion in the obligatory intracellular bacterium Anaplasma phagocytophilumNeutrophils in innate host defense against Staphylococcus aureus infectionsAnaplasma phagocytophilum Ats-1 is imported into host cell mitochondria and interferes with apoptosis inductionEhrlichia chaffeensis uses its surface protein EtpE to bind GPI-anchored protein DNase X and trigger entry into mammalian cellsInfection with Anaplasma phagocytophilum induces multilineage alterations in hematopoietic progenitor cells and peripheral blood cells.Anaplasma phagocytophilum modulates gp91phox gene expression through altered interferon regulatory factor 1 and PU.1 levels and binding of CCAAT displacement protein.Influence of Microbes on Neutrophil Life and Death.Molecular events involved in cellular invasion by Ehrlichia chaffeensis and Anaplasma phagocytophilumComparison and characterization of granulocyte cell models for Anaplasma phagocytophilum infection.Defective phagocytosis in Anaplasma phagocytophilum-infected neutrophils.Human ehrlichiosis and anaplasmosis.Neutrophils exposed to A. phagocytophilum under shear stress fail to fully activate, polarize, and transmigrate across inflamed endothelium.Human granulocytic ehrlichiosis agent inhibits superoxide anion generation by human neutrophils.Exploitation of interleukin-8-induced neutrophil chemotaxis by the agent of human granulocytic ehrlichiosisRapid activation of protein tyrosine kinase and phospholipase C-gamma2 and increase in cytosolic free calcium are required by Ehrlichia chaffeensis for internalization and growth in THP-1 cells.Characterization and transcriptional analysis of gene clusters for a type IV secretion machinery in human granulocytic and monocytic ehrlichiosis agents.Rickettsia-macrophage interactions: host cell responses to Rickettsia akari and Rickettsia typhi.Roles of p38 mitogen-activated protein kinase, NF-kappaB, and protein kinase C in proinflammatory cytokine mRNA expression by human peripheral blood leukocytes, monocytes, and neutrophils in response to Anaplasma phagocytophila.Anaplasma phagocytophilum infection induces protracted neutrophil degranulation.Anaplasma phagocytophilum delay of neutrophil apoptosis through the p38 mitogen-activated protein kinase signal pathway.Early transcriptional response of human neutrophils to Anaplasma phagocytophilum infectionTwo monoclonal antibodies with defined epitopes of P44 major surface proteins neutralize Anaplasma phagocytophilum by distinct mechanisms.Anaplasma phagocytophilum reduces neutrophil apoptosis in vivoMechanisms of obligatory intracellular infection with Anaplasma phagocytophilum.Nuclear factor kappa B protects against host cell apoptosis during Rickettsia rickettsii infection by inhibiting activation of apical and effector caspases and maintaining mitochondrial integrity.Diminished adhesion of Anaplasma phagocytophilum-infected neutrophils to endothelial cells is associated with reduced expression of leukocyte surface selectin.A role for apoptosis induced by acute herpes simplex virus infection in mice.Animal model of fatal human monocytotropic ehrlichiosis.Innate immunity against Granulibacter bethesdensis, an emerging gram-negative bacterial pathogenImmunopathology and ehrlichial propagation are regulated by interferon-gamma and interleukin-10 in a murine model of human granulocytic ehrlichiosis.Virulence potential of Ehrlichia chaffeensis strains of distinct genome sequencesSurface-exposed proteins of Ehrlichia chaffeensis.Ehrlichia chaffeensis TRP32 interacts with host cell targets that influence intracellular survivalIdentification of novel surface proteins of Anaplasma phagocytophilum by affinity purification and proteomicsAnaplasma and Ehrlichia infection.Role of neutrophils in innate immunity: a systems biology-level approachSurvival strategy of obligately intracellular Ehrlichia chaffeensis: novel modulation of immune response and host cell cycles.Anaplasma phagocytophilum utilizes multiple host evasion mechanisms to thwart NADPH oxidase-mediated killing during neutrophil infectionLessons from Anaplasma phagocytophilum: chromatin remodeling by bacterial effectors.
P2860
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P2860
Intracellular infection by the human granulocytic ehrlichiosis agent inhibits human neutrophil apoptosis.
description
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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name
Intracellular infection by the ...... ts human neutrophil apoptosis.
@ast
Intracellular infection by the ...... ts human neutrophil apoptosis.
@en
Intracellular infection by the ...... ts human neutrophil apoptosis.
@nl
type
label
Intracellular infection by the ...... ts human neutrophil apoptosis.
@ast
Intracellular infection by the ...... ts human neutrophil apoptosis.
@en
Intracellular infection by the ...... ts human neutrophil apoptosis.
@nl
prefLabel
Intracellular infection by the ...... ts human neutrophil apoptosis.
@ast
Intracellular infection by the ...... ts human neutrophil apoptosis.
@en
Intracellular infection by the ...... ts human neutrophil apoptosis.
@nl
P2093
P2860
P1476
Intracellular infection by the ...... ts human neutrophil apoptosis.
@en
P2093
P2860
P304
P356
10.1128/IAI.68.3.1125-1133.2000
P407
P577
2000-03-01T00:00:00Z