Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection - Wikidata - awgldk - TriplyDB

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

http://www.wikidata.org/entity/Q34003719

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Blastocystis ratti induces contact-independent apoptosis, F-actin rearrangement, and barrier function disruption in IEC-6 cells Infection Strategies of Intestinal Parasite Pathogens and Host Cell Responses Coral pathogens identified for White Syndrome (WS) epizootics in the Indo-Pacific Identification of Trichomonas vaginalis cysteine proteases that induce apoptosis in human vaginal epithelial cells.Strain-dependent induction of enterocyte apoptosis by Giardia lamblia disrupts epithelial barrier function in a caspase-3-dependent manner.Tritrichomonas foetus induces apoptotic cell death in bovine vaginal epithelial cells.Strain-dependent induction of human enterocyte apoptosis by blastocystis disrupts epithelial barrier and ZO-1 organization in a caspase 3- and 9-dependent manner Cell death and inflammatory bowel diseases: apoptosis, necrosis, and autophagy in the intestinal epithelium.Ups and downs of mucosal cellular immunity against protozoan parasites.MyD88-dependent pathways mediate resistance to Cryptosporidium parvum infection in mice The cell biology of cryptosporidium infection Host-flora interactions in inflammatory bowel disease.Structural analysis of Cryptosporidium parvum.Survival of protozoan intracellular parasites in host cells.Lawsonia intracellularis exploits β-catenin/Wnt and Notch signalling pathways during infection of intestinal crypt to alter cell homeostasis and promote cell proliferation.Mucosal defences against orally acquired protozoan parasites, emphasis on Toxoplasma gondii infections.Microbial-epithelial cell crosstalk during inflammation: the host response.Inhibition of apoptosis in Cryptosporidium parvum-infected intestinal epithelial cells is dependent on survivin.Intestinal immune response to human Cryptosporidium sp. infection.Biphasic modulation of apoptotic pathways in Cryptosporidium parvum-infected human intestinal epithelial cells Targeting caspases in intracellular protozoan infections.Over-expression and localization of a host protein on the membrane of Cryptosporidium parvum infected epithelial cells.Host cell tropism underlies species restriction of human and bovine Cryptosporidium parvum genotypes.Cryptosporidium parvum at different developmental stages modulates host cell apoptosis in vitro.Challenges in understanding the immunopathogenesis of Cryptosporidium infections in humans.Modulatory mechanisms of enterocyte apoptosis by viral, bacterial and parasitic pathogens.Diagnostic phrasing is independently correlated with the decision to treat for graft-versus-host disease: retrospective review of colon biopsies with rare apoptosis.CCL20 Displays Antimicrobial Activity Against Cryptosporidium parvum, but Its Expression Is Reduced During Infection in the Intestine of Neonatal Mice.Gregarine infection accelerates larval development of the cat flea Ctenocephalides felis (Bouché).Cryptosporidium parvum-specific mucosal immune response in C57BL/6 neonatal and gamma interferon-deficient mice: role of tumor necrosis factor alpha in protection.Role of gamma interferon in chemokine expression in the ileum of mice and in a murine intestinal epithelial cell line after Cryptosporidium parvum infection.Up-regulation of Fas (CD95) and induction of apoptosis in intestinal epithelial cells by nematode-derived molecules.Host cell fate on Cryptosporidium parvum egress from MDCK cells.Differential regulation of beta-defensin gene expression during Cryptosporidium parvum infection Apoptosis induced by parasitic diseases Proteomics analysis and protein expression during sporozoite excystation of Cryptosporidium parvum (Coccidia, Apicomplexa).Preferential infection of dividing cells by Cryptosporidium parvum.NF-kappaB-mediated expression of iNOS promotes epithelial defense against infection by Cryptosporidium parvum in neonatal piglets.Pulmonary cryptosporidiosis: role of COX2 and NF-kB.Cryptosporidium parvum disrupts intestinal epithelial barrier function via altering expression of key tight junction and adherens junction proteins.

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Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

http://www.wikidata.org/entity/Q34003719

description

2000 nî lūn-bûn

@nan

2000 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի մարտին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

@zh-hk

2000年論文

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2000年論文

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2000年论文

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name

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@ast

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@en

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@nl

type

label

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@ast

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@en

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@nl

prefLabel

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@ast

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@en

Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@nl

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IAI.68.3.1710-1713.2000

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Infection and Immunity

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Intestinal epithelial cell apoptosis following Cryptosporidium parvum infection

@en

P2093

D F McCole

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F Laurent

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L Eckmann

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M F Kagnoff

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P2860

Caspase cleavage of keratin 18 and reorganization of intermediate filaments during epithelial cell apoptosis

TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB

An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

Suppression of TNF-alpha-induced apoptosis by NF-kappaB

Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-kappaB activation prevents cell death

Human intestinal epithelial cells respond to Cryptosporidium parvum infection with increased prostaglandin H synthase 2 expression and prostaglandin E2 and F2alpha production

Cryptosporidium parvum infection of Caco-2 cell monolayers induces an apical monolayer defect, selectively increases transmonolayer permeability, and causes epithelial cell death.

Cryptosporidium parvum infection of human intestinal epithelial cells induces the polarized secretion of C-X-C chemokines

Attachment of Cryptosporidium parvum sporozoites to MDCK cells in vitro.

Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death.

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10.1128/IAI.68.3.1710-1713.2000

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97335

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