Decreased infectivity in Borrelia burgdorferi strain B31 is associated with loss of linear plasmid 25 or 28-1.
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The emergence of Lyme diseaseBiology of infection with Borrelia burgdorferiBorrelia burgdorferi changes its surface antigenic expression in response to host immune responsesRole of outer surface protein D in the Borrelia burgdorferi life cycleLyme Borreliosis: Is there a preexisting (natural) variation in antimicrobial susceptibility among Borrelia burgdorferi strains?vls Antigenic Variation Systems of Lyme Disease Borrelia: Eluding Host Immunity through both Random, Segmental Gene Conversion and Framework HeterogeneityCrystal structure of Lyme disease variable surface antigen VlsE of Borrelia burgdorferiRole of acetyl-phosphate in activation of the Rrp2-RpoN-RpoS pathway in Borrelia burgdorferiPotentially conflicting selective forces that shape the vls antigenic variation system in Borrelia burgdorferiInteraction of variable bacterial outer membrane lipoproteins with brain endotheliumThe diguanylate cyclase, Rrp1, regulates critical steps in the enzootic cycle of the Lyme disease spirochetes.Vascular binding of a pathogen under shear force through mechanistically distinct sequential interactions with host macromolecules.Lyme borreliosis: a review of data on transmission time after tick attachment.Central role of the Holliday junction helicase RuvAB in vlsE recombination and infectivity of Borrelia burgdorferi.Examination of the Borrelia burgdorferi transcriptome in Ixodes scapularis during feeding.Identification of potential virulence determinants by Himar1 transposition of infectious Borrelia burgdorferi B31.Borrelia burgdorferi complement regulator-acquiring surface protein 2 does not contribute to complement resistance or host infectivity.Detailed analysis of sequence changes occurring during vlsE antigenic variation in the mouse model of Borrelia burgdorferi infection.Characterization of a conditional bosR mutant in Borrelia burgdorferi.Characterization of the highly regulated antigen BBA05 in the enzootic cycle of Borrelia burgdorferi.The BosR regulatory protein of Borrelia burgdorferi interfaces with the RpoS regulatory pathway and modulates both the oxidative stress response and pathogenic properties of the Lyme disease spirochete.High-throughput plasmid content analysis of Borrelia burgdorferi B31 by using Luminex multiplex technology.Induction of type I and type III interferons by Borrelia burgdorferi correlates with pathogenesis and requires linear plasmid 36.bptA (bbe16) is essential for the persistence of the Lyme disease spirochete, Borrelia burgdorferi, in its natural tick vector.BosR (BB0647) controls the RpoN-RpoS regulatory pathway and virulence expression in Borrelia burgdorferi by a novel DNA-binding mechanism.The bba64 gene of Borrelia burgdorferi, the Lyme disease agent, is critical for mammalian infection via tick bite transmission.Use of the Cre-lox recombination system to investigate the lp54 gene requirement in the infectious cycle of Borrelia burgdorferiAnalysis of the ospC regulatory element controlled by the RpoN-RpoS regulatory pathway in Borrelia burgdorferi.Role of Borrelia burgdorferi linear plasmid 25 in infection of Ixodes scapularis ticks.Role of the surface lipoprotein BBA07 in the enzootic cycle of Borrelia burgdorferi.Plasmid stability during in vitro propagation of Borrelia burgdorferi assessed at a clonal levelStrain-specific variation of the decorin-binding adhesin DbpA influences the tissue tropism of the lyme disease spirocheteVraA (BBI16) protein of Borrelia burgdorferi is a surface-exposed antigen with a repetitive motif that confers partial protection against experimental Lyme borreliosis.Dissociation of infectivity and pathogenicity in Borrelia burgdorferiDNA microarray analysis of differential gene expression in Borrelia burgdorferi, the Lyme disease spirochete.The Borrelia hermsii factor H binding protein FhbA is not required for infectivity in mice or for resistance to human complement in vitro.Clonal polymorphism of Borrelia burgdorferi strain B31 MI: implications for mutagenesis in an infectious strain backgroundDecreased electroporation efficiency in Borrelia burgdorferi containing linear plasmids lp25 and lp56: impact on transformation of infectious B. burgdorferi.The essential nature of the ubiquitous 26-kilobase circular replicon of Borrelia burgdorferi.Genome stability of Lyme disease spirochetes: comparative genomics of Borrelia burgdorferi plasmids.
P2860
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P2860
Decreased infectivity in Borrelia burgdorferi strain B31 is associated with loss of linear plasmid 25 or 28-1.
description
2001 nî lūn-bûn
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2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
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2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@ast
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@en
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@nl
type
label
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@ast
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@en
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@nl
prefLabel
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@ast
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@en
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@nl
P2860
P1476
Decreased infectivity in Borre ...... of linear plasmid 25 or 28-1.
@en
P2093
M Labandeira-Rey
P2860
P304
P356
10.1128/IAI.69.1.446-455.2001
P407
P577
2001-01-01T00:00:00Z