Mice with altered serotonin 2C receptor RNA editing display characteristics of Prader-Willi syndrome. - Wikidata - awgldk - TriplyDB

Mice with altered serotonin 2C receptor RNA editing display characteristics of Prader-Willi syndrome.

Mice with altered serotonin 2C receptor RNA editing display characteristics of Prader-Willi syndrome.

http://www.wikidata.org/entity/Q34006076

about

A-to-I editing of coding and non-coding RNAs by ADARs To edit or not to edit: regulation of ADAR editing specificity and efficiency Adenosine-to-inosine RNA editing and human disease The emerging landscape of small nucleolar RNAs in cell biology The molecular basis of cognitive deficits in pervasive developmental disorders Quantitative analysis of 5HT(2C) receptor RNA editing patterns in psychiatric disorders Rapid and dynamic transcriptome regulation by RNA editing and RNA modifications Disruption of copper homeostasis due to a mutation of Atp7a delays the onset of prion disease The dynamic epitranscriptome: A to I editing modulates genetic information Transcript diversification in the nervous system: a to I RNA editing in CNS function and disease development Understanding RNA modifications: the promises and technological bottlenecks of the 'epitranscriptome'.Editing of neurotransmitter receptor and ion channel RNAs in the nervous system Mood, food, and obesity.Haploinsufficiency of the E3 ubiquitin ligase C-terminus of heat shock cognate 70 interacting protein (CHIP) produces specific behavioral impairments.Potent and selective inhibition of A-to-I RNA editing with 2'-O-methyl/locked nucleic acid-containing antisense oligoribonucleotides.Comparative RNA editing in autistic and neurotypical cerebella.Quantitative analysis of focused a-to-I RNA editing sites by ultra-high-throughput sequencing in psychiatric disorders.Prevention of 5-hydroxytryptamine2C receptor RNA editing and alternate splicing in C57BL/6 mice activates the hypothalamic-pituitary-adrenal axis and alters mood Molecular recognition of 6'-N-5-hexynoate kanamycin A and RNA 1x1 internal loops containing CA mismatches.Recognition of duplex RNA by the deaminase domain of the RNA editing enzyme ADAR2 The serotonergic anatomy of the developing human medulla oblongata: implications for pediatric disorders of homeostasis Oligophrenin-1 (OPHN1), a gene involved in X-linked intellectual disability, undergoes RNA editing and alternative splicing during human brain development.Altered ADAR 2 equilibrium and 5HT(2C) R editing in the prefrontal cortex of ADAR 2 transgenic mice.Prader-Willi syndrome and autism spectrum disorders: an evolving story.Dynamic 5-HT2C receptor editing in a mouse model of obesity.RNA Structures as Mediators of Neurological Diseases and as Drug Targets.TPH2 G/T polymorphism is associated with hyperphagia, IQ, and internalizing problems in Prader-Willi syndrome.Hyperactive hypothalamus, motivated and non-distractible chronic overeating in ADAR2 transgenic mice.Serotonin-2C and -2a receptor co-expression on cells in the rat medial prefrontal cortex.Nucleoside analog studies indicate mechanistic differences between RNA-editing adenosine deaminases.Dysregulated A to I RNA editing and non-coding RNAs in neurodegeneration.Serotonin and the regulation of mammalian energy balance.5-HT2 Receptor Regulation of Mitochondrial Genes: Unexpected Pharmacological Effects of Agonists and Antagonists Reciprocal regulation of A-to-I RNA editing and the vertebrate nervous system Structures of human ADAR2 bound to dsRNA reveal base-flipping mechanism and basis for site selectivity Oligonucleotide-induced alternative splicing of serotonin 2C receptor reduces food intake Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite.CAPS1 RNA Editing Promotes Dense Core Vesicle Exocytosis Regulation of alternative splicing by short non-coding nuclear RNAs.ADARs: allies or enemies? The importance of A-to-I RNA editing in human disease: from cancer to HIV-1.

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Mice with altered serotonin 2C receptor RNA editing display characteristics of Prader-Willi syndrome.

http://www.wikidata.org/entity/Q34006076

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@ast

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

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Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

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type

label

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

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Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

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Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@nl

prefLabel

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@ast

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@en

Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@nl

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P2860

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P356

J.NBD.2010.04.004

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Neurobiology of Disease

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Mice with altered serotonin 2C ...... tics of Prader-Willi syndrome.

@en

P2093

Atheir I Abbas

http://www.w3.org/2001/XMLSchema#string

Bryan L Roth

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David L Hachey

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David S Kump

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Jennifer L Hood

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Michael V Morabito

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Michelle M Jacobs

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Robert A Kesterson

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Ronald B Emeson

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P2860

Deletion of the MBII-85 snoRNA gene cluster in mice results in postnatal growth retardation

SnoRNA Snord116 (Pwcr1/MBII-85) deletion causes growth deficiency and hyperphagia in mice

Evidence that RNA editing modulates splice site selection in the 5-HT2C receptor gene

Prader-Willi phenotype caused by paternal deficiency for the HBII-85 C/D box small nucleolar RNA cluster

A deletion of the HBII-85 class of small nucleolar RNAs (snoRNAs) is associated with hyperphagia, obesity and hypogonadism

Identification of brain-specific and imprinted small nucleolar RNA genes exhibiting an unusual genomic organization

Site-directed mutagenesis by overlap extension using the polymerase chain reaction

Eating disorder and epilepsy in mice lacking 5-HT2c serotonin receptors

RNA editing of the human serotonin 5-hydroxytryptamine 2C receptor silences constitutive activity

Messenger RNA editing of the human serotonin 5-HT2C receptor

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169-180

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scholarly article

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10.1016/J.NBD.2010.04.004

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P433

2

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P478

39

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2010-04-13T00:00:00Z

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43159360

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20394819

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2906772

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