GABAergic neuron deficit as an idiopathic generalized epilepsy mechanism: the role of BRD2 haploinsufficiency in juvenile myoclonic epilepsy.
about
The Bromodomain and Extra-Terminal Domain (BET) Family: Functional Anatomy of BET Paralogous ProteinsRac1 and rac3 GTPases control synergistically the development of cortical and hippocampal GABAergic interneuronsMaternal separation with early weaning: a rodent model providing novel insights into neglect associated developmental deficitsThe C-terminal domain of Brd2 is important for chromatin interaction and regulation of transcription and alternative splicing.Pleiotrophin antagonizes Brd2 during neuronal differentiation.Genetic susceptibility in Juvenile Myoclonic Epilepsy: Systematic review of genetic association studies.Sex-specific behavioral traits in the Brd2 mouse model of juvenile myoclonic epilepsyDouble-bromo and extraterminal (BET) domain proteins regulate dendrite morphology and mechanosensory functionSex, epilepsy, and epigenetics.The bromodomain-containing gene BRD2 is regulated at transcription, splicing, and translation levels.A Review of the Differences in Developmental, Psychiatric, and Medical Endophenotypes Between Males and Females with Autism Spectrum Disorder.Estradiol does not affect spasms in the betamethasone-NMDA rat model of infantile spasms.Bromodomains and their pharmacological inhibitors.Roles of Rac1 and Rac3 GTPases during the development of cortical and hippocampal GABAergic interneuronsBromodomain and extra-terminal (BET) family proteins: New therapeutic targets in major diseases.Genetic and epigenetic mechanisms of epilepsy: a review.Thalamic volume reduction in drug-naive patients with new-onset genetic generalized epilepsy.Frontal and thalamic changes of GABA concentration indicate dysfunction of thalamofrontal networks in juvenile myoclonic epilepsy.Identifying gene expression profile of spinal cord injury in rat by bioinformatics strategy.
P2860
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P2860
GABAergic neuron deficit as an idiopathic generalized epilepsy mechanism: the role of BRD2 haploinsufficiency in juvenile myoclonic epilepsy.
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@ast
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@en
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@nl
type
label
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@ast
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@en
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@nl
prefLabel
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@ast
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@en
GABAergic neuron deficit as an ...... n juvenile myoclonic epilepsy.
@nl
P2093
P2860
P1433
P1476
GABAergic neuron deficit as an ...... in juvenile myoclonic epilepsy
@en
P2093
David A Greenberg
Debra J Wolgemuth
Enyuan Shang
Giorgi Maglakelidze
Jana Velíšková
Stephania Macchiarulo
Tamar Chachua
P2860
P304
P356
10.1371/JOURNAL.PONE.0023656
P407
P577
2011-08-24T00:00:00Z