Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome. - Wikidata - awgldk - TriplyDB

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

http://www.wikidata.org/entity/Q34010790

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Prospects for Improving Brain Function in Individuals with Down Syndrome Monoacylglycerol lipase inhibitor JZL184 improves behavior and neural properties in Ts65Dn mice, a model of down syndrome Pharmacological approaches to improving cognitive function in Down syndrome: current status and considerations.Chronic P7C3 treatment restores hippocampal neurogenesis in the Ts65Dn mouse model of Down Syndrome [Corrected].Increased excitability and altered action potential waveform in cerebellar granule neurons of the Ts65Dn mouse model of Down syndrome Involvement of Potassium and Cation Channels in Hippocampal Abnormalities of Embryonic Ts65Dn and Tc1 Trisomic Mice Molecular and cellular alterations in Down syndrome: toward the identification of targets for therapeutics From abnormal hippocampal synaptic plasticity in down syndrome mouse models to cognitive disability in down syndrome.Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists.GIRK channel modulation by assembly with allosterically regulated RGS proteins Weaker control of the electrical properties of cerebellar granule cells by tonically active GABAA receptors in the Ts65Dn mouse model of Down's syndrome.Increased Sparsity of Hippocampal CA1 Neuronal Ensembles in a Mouse Model of Down Syndrome Assayed by Arc Expression.The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome Glutamatergic transmission aberration: a major cause of behavioral deficits in a murine model of Down's syndrome.GABAergic dysfunction in pediatric neuro-developmental disorders.Altered intrinsic and network properties of neocortical neurons in the Ts65Dn mouse model of Down syndrome GABAergic hyperinnervation of dentate granule cells in the Ts65Dn mouse model of down syndrome: Exploring the role of App.Cognitive and pharmacological insights from the Ts65Dn mouse model of Down syndrome Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome.Aerobic exercise and a BDNF-mimetic therapy rescue learning and memory in a mouse model of Down syndrome.Learning by observation and learning by doing in Down and Williams syndromes.Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.The medial temporal memory system in Down syndrome: Translating animal models of hippocampal compromise.Infantile spasms in down syndrome: Rescue by knockdown of the GIRK2 channel.Altered distribution of hippocampal interneurons in the murine Down Syndrome model Ts65Dn.

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Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

http://www.wikidata.org/entity/Q34010790

description

2011 nî lūn-bûn

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2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@ast

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@en

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@nl

type

label

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@ast

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@en

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@nl

prefLabel

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@ast

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@en

Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@nl

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J.EXPNEUROL.2011.11.033

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Experimental Neurology

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Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome.

@en

P2093

Lina Chakrabarti

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Nathan P Cramer

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Tarik F Haydar

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Tyler K Best

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Zygmunt Galdzicki

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P2860

G protein-coupled receptors form stable complexes with inwardly rectifying potassium channels and adenylyl cyclase

Anchoring proteins confer G protein sensitivity to an inward-rectifier K(+) channel through the GK domain

Evidence for association of GABA(B) receptors with Kir3 channels and regulators of G protein signalling (RGS4) proteins.

Evolution of GABA(A) receptor diversity in the human genome

Functional and biochemical evidence for G-protein-gated inwardly rectifying K+ (GIRK) channels composed of GIRK2 and GIRK3

Synapse-associated protein 97 selectively associates with a subset of AMPA receptors early in their biosynthetic pathway

Olig1 and Olig2 triplication causes developmental brain defects in Down syndrome

Chromosome 21 and down syndrome: from genomics to pathophysiology

Heteromultimerization of G-protein-gated inwardly rectifying K+ channel proteins GIRK1 and GIRK2 and their altered expression in weaver brain

[Mongolism; a chromosomal disease (trisomy).]

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10.1016/J.EXPNEUROL.2011.11.033

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