SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells. - Wikidata - awgldk - TriplyDB

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

http://www.wikidata.org/entity/Q34025110

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Modulation of cardiac contractility by the phospholamban/SERCA2a regulatome Gene therapy for cardiovascular disease: advances in vector development, targeting, and delivery for clinical translation Structural Dynamics and Topology of Phosphorylated Phospholamban Homopentamer Reveal Its Role in the Regulation of Calcium Transport Adeno-associated virus-mediated gene therapy in cardiovascular disease Expression of sarco (endo) plasmic reticulum calcium ATPase (SERCA) system in normal mouse cardiovascular tissues, heart failure and atherosclerosis Post-translational Modifications in Heart Failure: Small Changes, Big Impact Small-molecule activation of SERCA2a SUMOylation for the treatment of heart failure.Attenuation of endoplasmic reticulum stress-related myocardial apoptosis by SERCA2a gene delivery in ischemic heart disease.Rescuing the failing heart by targeted gene transfer SERCA2a gene transfer improves electrocardiographic performance in aged mdx mice Neutralizing antibodies against AAV serotypes 1, 2, 6, and 9 in sera of commonly used animal models The evolution of heart gene delivery vectors Preexisting Neutralizing Antibodies to Adeno-Associated Virus Capsids in Large Animals Other Than Monkeys May Confound In Vivo Gene Therapy Studies.The AAV vector toolkit: poised at the clinical crossroads A Decoy Peptide Targeted to Protein Phosphatase 1 Attenuates Degradation of SERCA2a in Vascular Smooth Muscle Cells.Potential of gene therapy as a treatment for heart failure Small Molecular Allosteric Activator of the Sarco/Endoplasmic Reticulum Ca2+-ATPase (SERCA) Attenuates Diabetes and Metabolic Disorders.Gene therapy for the treatment of heart failure: promise postponed SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope.The synthetic antimicrobial peptide 19-2.5 attenuates septic cardiomyopathy and prevents down-regulation of SERCA2 in polymicrobial sepsis Therapeutic efficacy of AAV1.SERCA2a in monocrotaline-induced pulmonary arterial hypertension.Targeted gene therapy for the treatment of heart failure.Calcium cycling proteins and their association with heart failure.Cardiac gene therapy in large animals: bridge from bench to bedside.Gene therapy for heart failure: where do we stand?Promise of adeno-associated virus as a gene therapy vector for cardiovascular diseases.Use of Adeno-Associated Virus Vector for Cardiac Gene Delivery in Large-Animal Surgical Models of Heart Failure.Intratracheal Gene Delivery of SERCA2a Ameliorates Chronic Post-Capillary Pulmonary Hypertension: A Large Animal Model.Efficient transduction of vascular smooth muscle cells with a translational AAV2.5 vector: a new perspective for in-stent restenosis gene therapy.SERCA2a controls the mode of agonist-induced intracellular Ca2+ signal, transcription factor NFAT and proliferation in human vascular smooth muscle cells.SERCA2a gene transfer prevents intimal proliferation in an organ culture of human internal mammary artery.Benefit of SERCA2a gene transfer to vascular endothelial and smooth muscle cells: a new aspect in therapy of cardiovascular diseases.Correcting Calcium Dysregulation in Chronic Heart Failure Using SERCA2a Gene Therapy.

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SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

http://www.wikidata.org/entity/Q34025110

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

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name

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

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SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@en

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

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type

label

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@ast

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@en

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@nl

prefLabel

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@ast

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@en

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@nl

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Molecular Therapy

P1476

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

@en

P2093

Anne-Marie Lompré

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Dennis Ladage

http://www.w3.org/2001/XMLSchema#string

Djamel Lebeche

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Evangelia G Kranias

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Fabrice Atassi

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Jane A Leopold

http://www.w3.org/2001/XMLSchema#string

Kiyotake Ishikawa

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Lahouaria Hadri

http://www.w3.org/2001/XMLSchema#string

Roger J Hajjar

http://www.w3.org/2001/XMLSchema#string

Yoshiaki Kawase

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P2860

Improvement in survival and cardiac metabolism after gene transfer of sarcoplasmic reticulum Ca(2+)-ATPase in a rat model of heart failure

Basal transcription of the mouse sarco(endo)plasmic reticulum Ca2+-ATPase type 3 gene in endothelial cells is controlled by Ets-1 and Sp1

Adenoviral gene transfer of SERCA2a improves left-ventricular function in aortic-banded rats in transition to heart failure.

Molecular mechanisms involved in the regulation of the endothelial nitric oxide synthase.

Restoration of mechanical and energetic function in failing aortic-banded rat hearts by gene transfer of calcium cycling proteins.

How many Ca(2)+ATPase isoforms are expressed in a cell type? A growing family of membrane proteins illustrated by studies in platelets.

The cardiac sarcoplasmic/endoplasmic reticulum calcium ATPase: a potent target for cardiovascular diseases.

Sarcoplasmic reticulum Ca(2+) ATPase as a therapeutic target for heart failure.

Abrogation of ventricular arrhythmias in a model of ischemia and reperfusion by targeting myocardial calcium cycling.

Histamine upregulates gene expression of endothelial nitric oxide synthase in human vascular endothelial cells.

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1284-1292

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scholarly article

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10.1038/MT.2010.77

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7

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18

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Larissa Lipskaia

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2010-05-11T00:00:00Z

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232797732

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20461063

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2911258

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