Endocrine disruptors and polycystic ovary syndrome (PCOS): elevated serum levels of bisphenol A in women with PCOS.
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An overview of the effects of organic compounds on women's reproductive health and birth outcomesBisphenol-A and Female Infertility: A Possible Role of Gene-Environment InteractionsFetal programming of polycystic ovary syndromePolycystic ovary syndrome: do endocrine-disrupting chemicals play a role?The epigenetic lorax: gene-environment interactions in human healthEffects of Endocrine-Disrupting Chemicals on the OvaryBisphenol A and the female reproductive tract: an overview of recent laboratory evidence and epidemiological studiesEDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting ChemicalsDNA methylation changes in whole blood is associated with exposure to the environmental contaminants, mercury, lead, cadmium and bisphenol A, in women undergoing ovarian stimulation for IVFBisphenol A down-regulates rate-limiting Cyp11a1 to acutely inhibit steroidogenesis in cultured mouse antral folliclesAndrogen and Progesterone Receptors Are Targets for Bisphenol A (BPA), 4-Methyl-2,4-bis-(P-Hydroxyphenyl)Pent-1-Ene--A Potent Metabolite of BPA, and 4-Tert-Octylphenol: A Computational InsightThe paradox of progress: environmental disruption of metabolism and the diabetes epidemicSoy but not bisphenol A (BPA) induces hallmarks of polycystic ovary syndrome (PCOS) and related metabolic co-morbidities in ratsScientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.Bisphenol-A: epigenetic reprogramming and effects on reproduction and behaviorBisphenol a and reproductive health: update of experimental and human evidence, 2007-2013Effect of urinary bisphenolA on androgenic hormones and insulin resistance in preadolescent girls: a pilot study from the Ewha Birth & Growth CohortExposure to endocrine disrupters and nuclear receptor gene expression in infertile and fertile women from different Italian areasSubcellular redistribution of NHERF1 in response to dehydroepiandrosterone (DHEA) administration in endometrial glands of Wistar ratsChronic high dose intraperitoneal bisphenol A (BPA) induces substantial histological and gene expression alterations in rat penile tissue without impairing erectile functionEnvironmental pollution: a tangible risk for NAFLD pathogenesisBisphenol A and Ovarian Reserve among Infertile Women with Polycystic Ovarian SyndromeAssessment of a dielectric barrier discharge plasma reactor at atmospheric pressure for the removal of bisphenol A and tributyltin.Gender differences in the associations between urinary bisphenol A and body composition among American children: The National Health and Nutrition Examination Survey, 2003-2006.A round robin approach to the analysis of bisphenol A (BPA) in human blood samples.Oral Bisphenol A (BPA) given to rats at moderate doses is associated with erectile dysfunction, cavernosal lipofibrosis and alterations of global gene transcriptionBisphenol A in polycystic ovary syndrome and its association with liver-spleen axis.Human excretion of bisphenol A: blood, urine, and sweat (BUS) study.Bisphenol A (BPA) and its potential role in the pathogenesis of the polycystic ovary syndrome (PCOS).Industrial endocrine disruptors and polycystic ovary syndrome.Usefulness of bovine and porcine IVM/IVF models for reproductive toxicology.Exploring the potential association between brominated diphenyl ethers, polychlorinated biphenyls, organochlorine pesticides, perfluorinated compounds, phthalates, and bisphenol A in polycystic ovary syndrome: a case-control studyBisphenol A, benzophenone-type ultraviolet filters, and phthalates in relation to uterine leiomyoma.Effects of early prepubertal exposure to bisphenol A on the onset of puberty, ovarian weights, and estrous cycle in female mice.Serum free estradiol and estrogen receptor-α mediated activity are related to decreased incident hip fractures in older women.Using bisphenol-a to study the onset of polycystic ovarian syndrome.Prenatal Exposure to Endocrine Disruptors: A Developmental Etiology for Polycystic Ovary Syndrome.Sheep models of polycystic ovary syndrome phenotype.A review of the carcinogenic potential of bisphenol A.Bisphenol A exposure during adulthood causes augmentation of follicular atresia and luteal regression by decreasing 17β-estradiol synthesis via downregulation of aromatase in rat ovary.
P2860
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P2860
Endocrine disruptors and polycystic ovary syndrome (PCOS): elevated serum levels of bisphenol A in women with PCOS.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@ast
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@en
Endocrine disruptors and polycystic ovary syndrome
@nl
type
label
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@ast
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@en
Endocrine disruptors and polycystic ovary syndrome
@nl
prefLabel
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@ast
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@en
Endocrine disruptors and polycystic ovary syndrome
@nl
P2093
P356
P1476
Endocrine disruptors and polyc ...... isphenol A in women with PCOS.
@en
P2093
Antonis Chatzigeorgiou
Dimitrios Panidis
Eleni Kandaraki
Eleni Palioura
Frangiscos Economou
Michael Koutsilieris
Sarantis Livadas
Sotiria Palimeri
P304
P356
10.1210/JC.2010-1658
P407
P577
2010-12-30T00:00:00Z