cAMP facilitates EDHF-type relaxations in conduit arteries by enhancing electrotonic conduction via gap junctions.
about
Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?TRPA1 channels in the vasculatureEffects of connexin-mimetic peptides on gap junction functionality and connexin expression in cultured vascular cellsConnexins: gaps in our knowledge of vascular function.Enhanced spontaneous Ca2+ events in endothelial cells reflect signalling through myoendothelial gap junctions in pressurized mesenteric arteriesRegulator of G protein signaling 2 deficiency causes endothelial dysfunction and impaired endothelium-derived hyperpolarizing factor-mediated relaxation by dysregulating Gi/o signalingPotassium softens vascular endothelium and increases nitric oxide release.Vascular smooth muscle modulates endothelial control of vasoreactivity via reactive oxygen species production through myoendothelial communications.Traumatic brain injury in vivo and in vitro contributes to cerebral vascular dysfunction through impaired gap junction communication between vascular smooth muscle cells5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation.Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries.Membrane hyperpolarization is not required for sustained muscarinic agonist-induced increases in intracellular Ca2+ in arteriolar endothelial cellsAcetylcholine-induced vasodilation may depend entirely upon NO in the femoral artery of young pigletsOuabain exerts biphasic effects on connexin functionality and expression in vascular smooth muscle cellsControl of muscle blood flow during exercise: local factors and integrative mechanismsP2Y₂ receptor activation decreases blood pressure via intermediate conductance potassium channels and connexin 37Impaired cAMP signaling does not account for the attenuated EDHF-mediated dilations in female rat middle cerebral artery.Factors, fiction and endothelium-derived hyperpolarizing factor.Role of endothelium-derived hyperpolarizing factor in endothelial dysfunction during diabetes.Endothelium-Derived Hyperpolarization and Coronary Vasodilation: Diverse and Integrated Roles of Epoxyeicosatrienoic Acids, Hydrogen Peroxide, and Gap Junctions.Endothelium-dependent hyperpolarizations: past beliefs and present facts.Activation of KCa3.1 by SKA-31 induces arteriolar dilatation and lowers blood pressure in normo- and hypertensive connexin40-deficient mice.Myoendothelial contacts, gap junctions, and microdomains: anatomical links to function?Endothelial control of vasodilation: integration of myoendothelial microdomain signalling and modulation by epoxyeicosatrienoic acids.Interactions between thromboxane A₂, thromboxane/prostaglandin (TP) receptors, and endothelium-derived hyperpolarization.Role of connexins and pannexins in cardiovascular physiology.Endothelium-derived hyperpolarizing factor and diabetes.Connexin-mimetic peptides dissociate electrotonic EDHF-type signalling via myoendothelial and smooth muscle gap junctions in the rabbit iliac artery.Enhanced inhibition of the EDHF phenomenon by a phenyl methoxyalaninyl phosphoramidate derivative of dideoxyadenosineAnalysis of effects of connexin-mimetic peptides in rat mesenteric small arteries.Reduced hyperpolarization in endothelial cells of rabbit aortic valve following chronic nitroglycerine administration.Attenuated store-operated Ca2+ entry underpins the dual inhibition of nitric oxide and EDHF-type relaxations by iodinated contrast media.Exchange protein activated by cAMP (Epac) induces vascular relaxation by activating Ca2+-sensitive K+ channels in rat mesenteric artery.Alterations in EDHF-type relaxation and phosphodiesterase activity in mesenteric arteries from diabetic rats.Developmental changes in myoendothelial gap junction mediated vasodilator activity in the rat saphenous artery.Attenuation of conducted vasodilatation in rat mesenteric arteries during hypertension: role of inwardly rectifying potassium channels.Hyperpolarization of murine small caliber mesenteric arteries by activation of endothelial proteinase-activated receptor 2.Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.Functional changes in adenylyl cyclases and associated decreases in relaxation responses in mesenteric arteries from diabetic rats.Connexin mimetic peptides fail to inhibit vascular conducted calcium responses in renal arterioles.
P2860
Q24672135-D4AFE4AA-4E8B-450C-A7B7-F9C7F92E392CQ27024164-F1D2BC3C-B8BD-47DC-A5F5-FEC14FDE6457Q28573218-1CAF2E16-222D-4430-8172-518F53C0BE51Q30434523-51787E2B-B19F-44BB-8F37-33FBE851D525Q30485814-F23EF7F4-B6C1-4BE0-8E08-4210A16B18D2Q30512752-DB4782B9-1AFC-4A9D-B61C-88C793C09B60Q33406847-7139F3F8-8783-4CD9-9FF0-6140AE6FC335Q33489567-70407163-432B-49E3-A68B-0EA729BCA50BQ33720087-36BC990E-EDFE-46FE-A1DA-FD731068603AQ33784391-A023F9CE-9CF3-45D0-9F36-D6FAE6267340Q34393364-C4EDBFC0-2126-4A4D-9156-33DEA06B7B39Q34461415-E54EEF91-2971-49C1-B909-27F05390708FQ35044844-4178B975-7407-488D-BB97-18977DFFA2ECQ35045938-147E92AA-5D44-40F5-88AD-C9FECBD0D8C5Q35170190-E90511EE-286F-4C94-BB93-17B18B6F5992Q35642178-CA913462-58A5-451C-BFAB-6911FA4A4DC4Q35746012-CD0AE0C7-94C3-4DD4-9F12-91E1F670B665Q35915560-14432F91-E1C1-4A2E-9185-359A2A7F3078Q36107894-7108F4F4-AEB4-46D6-8DBE-CF9564A95D2BQ36450226-D0E1B5BB-2AD8-4E58-BF74-6125CF1F9DBBQ36939441-5D338BCC-3C95-414D-8A3C-F5B5624D81ABQ37327372-508D3E85-1BF0-4249-845F-F7288ABE797BQ37954713-22C50B20-1FD7-4330-9C8E-8CBCF5509EB7Q38113090-01A6722D-EEA7-47FF-81CD-909C7E39FAAEQ38182425-B542DB0D-5805-4E66-9C9B-D1B723CD5A13Q38532371-EA8F4727-EEEE-4B37-8FD5-52F54D054850Q41813606-B228799A-CF03-4DFC-B54B-81BD6ACB3DE2Q42148610-95D86AB2-74B3-4949-90AC-9E78B37DBDA5Q42258763-44064D13-D71C-422E-A209-E8072F78C7EBQ42489777-C40EF9E9-2DD0-402B-A45C-4FC2B3329607Q43054888-5A3F693E-E085-4D0D-B4AA-43E5693A97A8Q43645319-9EEE088B-7D82-4D14-90E0-B72C98205593Q43862835-ABCB5295-6579-411C-AAA1-A5735F38F31FQ44470070-A4B3EACC-D433-4802-8E69-E648AEE24E18Q44758487-56DC4A75-1D71-4590-B7D0-D315A135A28DQ45154666-6133D709-28A5-41CF-B50F-FAE72828F5EBQ45220380-E8B0999E-D987-4571-8E4A-F8E53157BE91Q46121556-33F12BB0-3598-4593-9E27-C589DDAD8AFCQ46491173-993EFBC6-918F-4301-B38E-660692C8509AQ46492713-861838D7-367C-4D9C-92EB-38C6DD4A3804
P2860
cAMP facilitates EDHF-type relaxations in conduit arteries by enhancing electrotonic conduction via gap junctions.
description
2002 nî lūn-bûn
@nan
2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@ast
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@en
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@nl
type
label
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@ast
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@en
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@nl
prefLabel
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@ast
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@en
cAMP facilitates EDHF-type rel ...... conduction via gap junctions.
@nl
P2093
P2860
P356
P1476
cAMP facilitates EDHF-type rel ...... c conduction via gap junctions
@en
P2093
Andrew T Chaytor
Beverley D Giddings
Hannah J Taylor
Tudor M Griffith
P2860
P304
P356
10.1073/PNAS.092089799
P407
P577
2002-04-23T00:00:00Z