Nrf2, a PPARγ alternative pathway to promote CD36 expression on inflammatory macrophages: implication for malaria. - Wikidata - awgldk - TriplyDB

Nrf2, a PPARγ alternative pathway to promote CD36 expression on inflammatory macrophages: implication for malaria.

Nrf2, a PPARγ alternative pathway to promote CD36 expression on inflammatory macrophages: implication for malaria.

http://www.wikidata.org/entity/Q34031187

about

Dichotomous roles for externalized cardiolipin in extracellular signaling: promotion of phagocytosis and attenuation of innate immunity Nrf2 as a master regulator of tissue damage control and disease tolerance to infection Comparative Studies of Vertebrate Platelet Glycoprotein 4 (CD36)Lipid storage by adipose tissue macrophages regulates systemic glucose tolerance Induction of CD36 and thrombospondin-1 in macrophages by hypoxia-inducible factor 1 and its relevance in the inflammatory process CD36 upregulation mediated by intranasal LV-NRF2 treatment mitigates hypoxia-induced progression of Alzheimer's-like pathogenesis.Cleaning up after ICH: the role of Nrf2 in modulating microglia function and hematoma clearance Nrf2-driven CD36 and HO-1 gene expression in circulating monocytes correlates with favourable clinical outcome in pregnancy-associated malaria Anti-TNF certolizumab pegol induces antioxidant response in human monocytes via reverse signaling.LPA/PKD-1-FoxO1 Signaling Axis Mediates Endothelial Cell CD36 Transcriptional Repression and Proangiogenic and Proarteriogenic Reprogramming.Redox control of inflammation in macrophages.Are reactive oxygen species always detrimental to pathogens?Tim-3 inhibits macrophage control of Listeria monocytogenes by inhibiting Nrf2.Molecular mechanisms of hypolipidemic effects of curcumin.The mononuclear phagocyte system in homeostasis and disease: a role for heme oxygenase-1.The immunological balance between host and parasite in malaria.Reduced circulating levels of sTWEAK are associated with NAFLD and may affect hepatocyte triglyceride accumulation.Macrophage heterogeneity and cholesterol homeostasis: classically-activated macrophages are associated with reduced cholesterol accumulation following treatment with oxidized LDL.LRH-1 mediates anti-inflammatory and antifungal phenotype of IL-13-activated macrophages through the PPARγ ligand synthesis Antioxidant defense of Nrf2 vs pro-inflammatory system of NF-κB during the amoebic liver infection in hamster.Collaborative Power of Nrf2 and PPARγ Activators against Metabolic and Drug-Induced Oxidative Injury Haematoma scavenging in intracerebral haemorrhage: from mechanisms to the clinic.High-density lipoprotein from subjects with coronary artery disease promotes macrophage foam cell formation: role of scavenger receptor CD36 and ERK/MAPK signaling.

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P2860

Nrf2, a PPARγ alternative pathway to promote CD36 expression on inflammatory macrophages: implication for malaria.

http://www.wikidata.org/entity/Q34031187

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Nrf2, a PPARγ alternative path ...... ages: implication for malaria.

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JOURNAL.PPAT.1002254

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Nrf2, a PPARγ alternative path ...... ages: implication for malaria.

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Agnès Aubouy

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Agnès Coste

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Antoine Berry

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Bernard Pipy

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Christophe Dardenne

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Etienne Meunier

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Françoise Benoit-Vical

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Lise Lefèvre

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Rose-Anne Lavergne

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P2860

Oxidized LDL regulates macrophage gene expression through ligand activation of PPARgamma

Human malaria parasites in continuous culture

Tumor necrosis factor alpha and adalimumab differentially regulate CD36 expression in human monocytes

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

Interleukin-4-dependent production of PPAR-gamma ligands in macrophages by 12/15-lipoxygenase

Disruption of Nrf2, a key inducer of antioxidant defenses, attenuates ApoE-mediated atherosclerosis in mice.

PPARγ controls Dectin-1 expression required for host antifungal defense against Candida albicans.

PPARγ ligands switched high fat diet-induced macrophage M2b polarization toward M2a thereby improving intestinal Candida elimination.

Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.

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e1002254

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9

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7

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Bernhard Ryffel

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2011-09-15T00:00:00Z

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51665580

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