The mitochondrial permeability transition: its molecular mechanism and role in reperfusion injury.
about
Reye syndrome--insights on causation and prognosisBrain injury following cardiac arrest: pathophysiology for neurocritical careMitochondrial permeability transition pore is a potential drug target for neurodegenerationAuranofin increases apoptosis and ischaemia-reperfusion injury in the rat isolated heartCellular functions of cardiolipin in yeastHeme oxygenase-1 induction improves cardiac function following myocardial ischemia by reducing oxidative stressIncretin-modulated beta cell energetics in intact islets of Langerhans.Targeting mitochondria for resuscitation from cardiac arrest.Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Uncoupling proteins 2 and 3 are highly active H(+) transporters and highly nucleotide sensitive when activated by coenzyme Q (ubiquinone)Mitochondrial mechanisms in cerebral vascular control: shared signaling pathways with preconditioning.Mitochondrial permeability transition in the crustacean Artemia franciscana: absence of a calcium-regulated pore in the face of profound calcium storage.Mitochondrial Ca2+-activated K+ channels more efficiently reduce mitochondrial Ca2+ overload in rat ventricular myocytes.Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.Neuroprotection by rasagiline: a new therapeutic approach to Parkinson's disease?Intersection between mitochondrial permeability pores and mitochondrial fusion/fission.The permeability transition pore in cell death.Minocycline and N-methyl-4-isoleucine cyclosporin (NIM811) mitigate storage/reperfusion injury after rat liver transplantation through suppression of the mitochondrial permeability transition.C-Jun N-terminal kinase 2 promotes graft injury via the mitochondrial permeability transition after mouse liver transplantation.Mitochondrial permeability transition in liver ischemia and reperfusion: role of c-Jun N-terminal kinase 2.Mitochondrial-mediated suppression of ROS production upon exposure of neurons to lethal stress: mitochondrial targeted preconditioning.Probing the molecular mechanisms of neuronal degeneration: importance of mitochondrial dysfunction and calcineurin activation.Investigation of reperfusion injury and ischemic preconditioning in microsurgery.Mitochondrial permeability transition pore in Alzheimer's disease: cyclophilin D and amyloid betaA novel neuroprotective strategy for ischemic stroke: transient mild acidosis treatment by CO2 inhalation at reperfusion.Translocation of iron from lysosomes to mitochondria during ischemia predisposes to injury after reperfusion in rat hepatocytes.Intracellular calcium signaling pathways during liver ischemia and reperfusion.Mitochondria, calcium-dependent neuronal death and neurodegenerative disease.Optimising cardioprotection during myocardial ischaemia: targeting potential intracellular pathways with glucagon-like peptide-1Cyclophilin D over-expression increases mitochondrial complex III activity and accelerates supercomplex formation.Induction of apoptosis by antimycin A in differentiated PC12 cell line.A novel method for the detection of viable human pancreatic beta cells by flow cytometry using fluorophores that selectively detect labile zinc, mitochondrial membrane potential and protein thiols.Targeted expression of Kir6.2 in mitochondria confers protection against hypoxic stress.Gene expression analysis in mitochondria from chagasic mice: alterations in specific metabolic pathways.In vivo opening of the mitochondrial permeability transition pore in a rat model of ventricular fibrillation and closed-chest resuscitation.Cardioprotective activity of a novel and potent competitive inhibitor of lactate dehydrogenase.Cytochrome c release from isolated rat liver mitochondria can occur independently of outer-membrane rupture: possible role of contact sitesCyclophilin and viruses: cyclophilin as a cofactor for viral infection and possible anti-viral target.Selenite sensitizes mitochondrial permeability transition pore opening in vitro and in vivo: a possible mechanism for chemo-protection.Dissimilar mechanisms of cytochrome c release induced by octyl glucoside-activated BAX and by BAX activated with truncated BID.
P2860
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P2860
The mitochondrial permeability transition: its molecular mechanism and role in reperfusion injury.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
The mitochondrial permeability ...... nd role in reperfusion injury.
@ast
The mitochondrial permeability ...... nd role in reperfusion injury.
@en
The mitochondrial permeability ...... nd role in reperfusion injury.
@nl
type
label
The mitochondrial permeability ...... nd role in reperfusion injury.
@ast
The mitochondrial permeability ...... nd role in reperfusion injury.
@en
The mitochondrial permeability ...... nd role in reperfusion injury.
@nl
prefLabel
The mitochondrial permeability ...... nd role in reperfusion injury.
@ast
The mitochondrial permeability ...... nd role in reperfusion injury.
@en
The mitochondrial permeability ...... nd role in reperfusion injury.
@nl
P356
P1476
The mitochondrial permeability ...... nd role in reperfusion injury.
@en
P2093
Halestrap AP
P304
P356
10.1042/BSS0660181
P577
1999-01-01T00:00:00Z