IL-7 promotes T cell viability, trafficking, and functionality and improves survival in sepsis. - Wikidata - awgldk - TriplyDB

IL-7 promotes T cell viability, trafficking, and functionality and improves survival in sepsis.

IL-7 promotes T cell viability, trafficking, and functionality and improves survival in sepsis.

http://www.wikidata.org/entity/Q34039180

about

Detrimental cross-talk between sepsis and acute kidney injury: new pathogenic mechanisms, early biomarkers and targeted therapies CD1d- and MR1-Restricted T Cells in Sepsis T-cell-mediated immunity and the role of TRAIL in sepsis-induced immunosuppression The coagulopathy of acute sepsis The inflammatory response in sepsis Clinical review: sepsis and septic shock--the potential of gene arrays Advances in the management of sepsis and the understanding of key immunologic defects The significance and regulatory mechanisms of innate immune cells in the development of sepsis Immunosuppression after sepsis: systemic inflammation and sepsis induce a loss of naïve T-cells but no enduring cell-autonomous defects in T-cell function Emerging therapeutic targets of sepsis-associated acute kidney injury.Increased CD4(+) T cell co-inhibitory immune receptor CEACAM1 in neonatal sepsis and soluble-CEACAM1 in meningococcal sepsis: a role in sepsis-associated immune suppression?Interleukin-7 is decreased and maybe plays a pro-inflammatory function in primary immune thrombocytopenia.Targeting the programmed cell death 1: programmed cell death ligand 1 pathway reverses T cell exhaustion in patients with sepsis Reduced plasma levels of soluble interleukin-7 receptor during graft-versus-host disease (GVHD) in children and adults.Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy.JunD/AP-1-mediated gene expression promotes lymphocyte growth dependent on interleukin-7 signal transduction.Interleukin-7 (IL-7) treatment accelerates neutrophil recruitment through gamma delta T-cell IL-17 production in a murine model of sepsis.Impact of sepsis on CD4 T cell immunity Cdc25A-driven proliferation regulates CD62L levels and lymphocyte movement in response to interleukin-7.Interleukin-7, but not thymic stromal lymphopoietin, plays a key role in the T cell response to influenza A virus.Revisiting caspases in sepsis.Chronic alcohol ingestion increases mortality and organ injury in a murine model of septic peritonitis.Helminths and their implication in sepsis - a new branch of their immunomodulatory behaviour?Neonatal sepsis and inflammatory mediators.Cutting edge: critical role for C5aRs in the development of septic lymphopenia in mice.Prevention of lymphocyte apoptosis in septic mice with cancer increases mortality.Immune unresponsiveness to secondary heterologous bacterial infection after sepsis induction is TRAIL dependent Interleukin-7 and anti-programmed cell death 1 antibody have differing effects to reverse sepsis-induced immunosuppression.Persistent lymphopenia after diagnosis of sepsis predicts mortality.Role of cytokines as a double-edged sword in sepsis Validation of a gene expression-based subclassification strategy for pediatric septic shock.Alterations in antigen-specific naive CD4 T cell precursors after sepsis impairs their responsiveness to pathogen challenge.Post-operative infection and sepsis in humans is associated with deficient gene expression of γc cytokines and their apoptosis mediators PD-L1 blockade improves survival in experimental sepsis by inhibiting lymphocyte apoptosis and reversing monocyte dysfunction.Upregulation of programmed death-1 on T cells and programmed death ligand-1 on monocytes in septic shock patients.Decreased interleukin 7 responsiveness of T lymphocytes in patients with idiopathic CD4 lymphopenia Clinical review: flow cytometry perspectives in the ICU - from diagnosis of infection to monitoring of injury-induced immune dysfunctions.IL-15 Superagonist Expands mCD8+ T, NK and NKT Cells after Burn Injury but Fails to Improve Outcome during Burn Wound Infection Immunosuppression in patients who die of sepsis and multiple organ failure.Acute-Phase Deaths from Murine Polymicrobial Sepsis Are Characterized by Innate Immune Suppression Rather Than Exhaustion

P2860

Q26765788-C6A9685A-B9AF-4479-AFB8-9367DC01F26B Q26796132-01591378-DB4B-4FDE-B82F-864F0E199AFC Q26823892-E36DF6DB-695C-4F92-BA9B-68920E2CC948 Q26826950-9A61A9C7-BDBE-4E5B-B154-EE79F6EF607C Q26849300-1E86DDEC-44CB-47C5-8372-8F7CAA4826C3 Q26864138-63F8D79E-A947-407B-982F-FA27C7C51C7C Q26865386-DBF7D9DA-D1C1-432C-BD40-0D229ABAD307 Q26866126-F490F413-FC2C-48CF-9CDD-1C5854189BC3 Q27320494-65103F15-DAE4-41D2-B756-998D2BB72B1C Q30300333-D246A8B0-5CAB-462B-A555-2FB24B5D0B8D Q31124179-3C5E6671-8273-4FF0-817D-343A1300386F Q33414739-81559674-7F4B-4E4E-AF82-A180D2FAAE5A Q33749945-815A4BCF-0DF6-4E61-8E1E-D61CFAE734E9 Q33817588-FD4933A1-DC08-49B5-A0EF-33093C40181F Q33828888-7A1D9E7C-DE8D-47D3-8EEC-5D88F5890CB9 Q34182432-E9444A18-36FD-4D0D-8FDE-03DE6F238061 Q34290886-601EFE26-9BF7-4B5F-B733-1DDB0D78FFAB Q34341378-2042D7F6-20F0-4EFD-92DD-FF3F3DE5E8DE Q34443792-2DC21C1C-91A0-4798-BECF-1EC15C6CF19A Q34494025-9C77D0AE-CCE1-4A47-B75F-6FE4B2E24EDE Q34662282-0FD455A0-742D-4726-BCEC-ED4DDDC8553F Q34743871-C0ABD9ED-6447-4AFA-BE76-A5EB7D7BC136 Q34844755-F6786411-44FC-419E-B478-0F5F925C084D Q34970523-919B4C58-8614-41C1-8D74-94788EEDBDCD Q34980303-9BB2E77F-2C13-4C0E-A009-6413E535B68F Q35147518-B6366F35-67D0-42F5-856E-29498EDC808E Q35175936-8863F046-DF73-4FA5-A9AE-F1F2D1F70109 Q35177535-0165D99F-5BE9-4BF7-9222-FC394566B9C9 Q35184876-A93B099C-CE6C-453F-91AD-A9AD02A6101B Q35230688-099A37A6-F30D-4BE7-A1CC-1FA980C4611D Q35387550-8FDC663A-BEC8-435C-8026-DD9A5EB638E3 Q35546243-45191758-7A98-49C6-A476-E5DD732D2FCD Q35558396-6D90E254-45EF-4D81-931B-84DF15C388D1 Q35561405-668CC22D-BA26-4D68-A737-31A00713B844 Q35567048-DDB79E0B-C5C7-4D69-8AD7-E867B8AF3973 Q35883466-9F222A04-A088-4A7C-88DE-6EC9FDD6C6EF Q35908002-7BECC346-5A46-4CCD-BF5B-6C9148903579 Q35918321-576E33F6-0679-4DF3-A6A2-B5B67D463CF2 Q35991015-D5C758F0-4460-4817-8A73-5976BE7AB8A9 Q36119404-A915E474-92A6-4B37-AFA1-AB02934EF2E6

P2860

IL-7 promotes T cell viability, trafficking, and functionality and improves survival in sepsis.

http://www.wikidata.org/entity/Q34039180

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մարտին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@ast

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@en

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@nl

type

label

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@ast

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@en

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@nl

prefLabel

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@ast

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@en

IL-7 promotes T cell viability ...... d improves survival in sepsis.

@nl

P1433

Q34039180-8288537F-157C-425F-93D5-892746C91FAD

P1476

Q34039180-4C3A24A9-047C-49EA-A861-BBB7EE6CD890

P2093

Q34039180-260CFE41-D25B-4C7E-BFC7-148AE2F63CE3

Q34039180-2A4A13A8-0D5A-4124-85AC-042565D17373

Q34039180-38489F6B-7BE7-4C88-BB03-B642639A7A6B

Q34039180-530B0DF7-D833-4D33-A421-9D8EB7839BA8

Q34039180-54C8A50E-FF90-4884-8A5A-E6D5FE029380

Q34039180-6081F3EF-1732-4B5E-9F20-9ECBC996FE60

Q34039180-7D608982-4A17-404F-9A3D-A44BA4BFDB93

Q34039180-CA562283-F1E4-4447-BB87-16C0E2F2D220

Q34039180-DA52F307-2286-4047-97AA-6541026EF98B

P2860

Q34039180-014A2080-E4B5-46C6-A6D3-A8061958A996

Q34039180-03C6EC45-E448-4DE4-B91E-4C43D77F2E0E

Q34039180-03E41572-E32D-4BDF-981B-BA99C92611D3

Q34039180-05F98709-5ACD-408F-A27A-DCC2A513E473

Q34039180-0AD5A25F-A71F-46D5-81C3-7194A2E8322E

Q34039180-10BAE174-98D9-4455-A695-AC5E977B19F5

Q34039180-1665CB1A-9A15-4DB3-8775-D18DB3EE5369

Q34039180-169F3A72-A427-4832-8801-25F689608EFF

Q34039180-197F799D-2C30-4301-B3E5-746B56D823B7

Q34039180-1C93037D-0089-4156-A18A-0D7919272C96

P304

Q34039180-7541D1BA-8084-4FE7-B137-D00DB4E9B2A4

P31

Q34039180-9ADB70F6-AD28-4785-9970-124E56890C01

P356

Q34039180-568F8D8B-16E2-40D5-B8C2-4888F06CEF9E

P407

Q34039180-FAA006A0-303B-42CE-A85E-5022CF8568A9

P433

Q34039180-148DC0D2-696E-4B4B-BF63-08A6396F2DB9

P478

Q34039180-1D37BFB2-E2A3-446A-B7D1-0A3CA07E0D51

P50

Q34039180-6912A0F5-1EA5-4ABC-9835-D6732499BCD8

Q34039180-7AA2C687-1FE3-4C7A-BD57-3B78957C17F0

Q34039180-B042F00F-0A61-482D-A0BF-9EC94044F169

Q34039180-BA2891E6-AF6E-4FDA-87C4-23EA3DB607B5

Q34039180-E0CA41FF-6678-4609-8C0F-30C6285C0A52

P577

Q34039180-FD38F2E9-AF33-4D2B-B33F-2A16595FEBC3

P5875

Q34039180-F0D3899B-A513-474B-B34E-FED3CA112FDB

P698

Q34039180-BC0878D8-42B0-4699-8202-2082094C3480

P921

Q34039180-738995F2-7830-4424-BA3F-EAD5ADEA89C8

P932

Q34039180-F79C925B-435E-4849-9710-E2ECD236B06F

P356

JIMMUNOL.0903151

P1433

Journal of Immunology

P1476

IL-7 promotes T cell viability ...... nd improves survival in sepsis

@en

P2093

Andrew S Hoekzema

http://www.w3.org/2001/XMLSchema#string

Eizo Watanabe

http://www.w3.org/2001/XMLSchema#string

Jacqueline Unsinger

http://www.w3.org/2001/XMLSchema#string

Jacquelyn S McDonough

http://www.w3.org/2001/XMLSchema#string

Jared T Muenzer

http://www.w3.org/2001/XMLSchema#string

Johannes Tschoep

http://www.w3.org/2001/XMLSchema#string

Margaret McGlynn

http://www.w3.org/2001/XMLSchema#string

Michel Morre

http://www.w3.org/2001/XMLSchema#string

Thomas A Ferguson

http://www.w3.org/2001/XMLSchema#string

P2860

IL-7 administration to humans leads to expansion of CD8+ and CD4+ cells but a relative decrease of CD4+ T-regulatory cells

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis

Bim/Bcl-2 balance is critical for maintaining naive and memory T cell homeostasis

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

The pathophysiology and treatment of sepsis

Essential role of gamma interferon in survival of colon ascendens stent peritonitis, a novel murine model of abdominal sepsis.

Targeted adenovirus-induced expression of IL-10 decreases thymic apoptosis and improves survival in murine sepsis.

Apoptosis in sepsis: a new target for therapeutic exploration.

Sepsis syndromes: understanding the role of innate and acquired immunity.

P304

3768-3779

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4049/JIMMUNOL.0903151

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

184

http://www.w3.org/2001/XMLSchema#string

P50

Charles C. Caldwell

Jonathan E. McDunn

Richard S Hotchkiss

P577

2010-03-03T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

41722085

http://www.w3.org/2001/XMLSchema#string

P698

20200277

http://www.w3.org/2001/XMLSchema#string

P921

P932

2914630

http://www.w3.org/2001/XMLSchema#string