Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
about
Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activityMitochondrial transplantation for therapeutic useRole of the MPTP in conditioning the heart - translatability and mechanismNeuroprotective role of ATP-sensitive potassium channels in cerebral ischemiaThe KATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidationModeling mitochondrial bioenergetics with integrated volume dynamicsMitochondrial reactive oxygen species production in excitable cells: modulators of mitochondrial and cell function.Activation of ion-conducting pathways in the inner mitochondrial membrane - an unrecognized activity of fatty acid?Mitochondrial matrix K+ flux independent of large-conductance Ca2+-activated K+ channel openingPotential therapeutic benefits of strategies directed to mitochondria.Desferoxamine and ethyl-3,4-dihydroxybenzoate protect myocardium by activating NOS and generating mitochondrial ROS.Cardioprotective effects of nitrite during exercise.Mitochondrial Ca2+-activated K+ channels more efficiently reduce mitochondrial Ca2+ overload in rat ventricular myocytes.The role of mitochondria in protection of the heart by preconditioning.Sarcolemmal and mitochondrial K(ATP) channels and myocardial ischemic preconditioning.Role of adenosine A1 and A3 receptors in regulation of cardiomyocyte homeostasis after mitochondrial respiratory chain injury.KATP Channels in the Cardiovascular System.Protection against cardiac injury by small Ca(2+)-sensitive K(+) channels identified in guinea pig cardiac inner mitochondrial membraneSelective opening of mitochondrial ATP-sensitive potassium channels during surgically induced myocardial ischemia decreases necrosis and apoptosisMitochondrial reactive oxygen species: which ROS signals cardioprotection?Mitochondria and big-conductance potassium channel openers.Interaction of mitochondrial potassium channels with the permeability transition pore.Mitochondrial potassium channels as pharmacological target for cardioprotective drugs.Crosstalk of mitochondria with NADPH oxidase via reactive oxygen and nitrogen species signalling and its role for vascular function.Calpeptin, not calpain, directly inhibits an ion channel of the inner mitochondrial membrane.Persistent mitoKATP Activation Is Involved in the Isoflurane-induced Cytotoxicity.Cellular and molecular events in ischemic preconditioning: potential therapeutic applications in cardioprotection.Mitochondrial ATP-sensitive potassium channels enhance angiotensin-induced oxidative damage and dopaminergic neuron degeneration. Relevance for aging-associated susceptibility to Parkinson's disease.Insights into the cardioprotective function of adenosine A(1) and A(3) receptors.epsilonPKC phosphorylates the mitochondrial K(+) (ATP) channel during induction of ischemic preconditioning in the rat hippocampus.Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heart.Effective protection by NHE-1 inhibition in ischemic and reperfused heart under preconditioning blockade.Expression of opioid receptor subtypes and their ligands in fibrillating human atria.Role of mitochondrial KATP channels and protein kinase C in ischaemic preconditioning.Sevoflurane depolarizes pre-synaptic mitochondria in the central nervous system.Functional distinctions between the mitochondrial ATP-dependent K+ channel (mitoKATP) and its inward rectifier subunit (mitoKIR).Attenuation of fatty acid-induced apoptosis by low-dose alcohol in neonatal rat cardiomyocytes.Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration.Pharmacological preconditioning of ATP-sensitive potassium channel openers on acute urinary retention-induced bladder dysfunction in the rat.Cardiac mitochondrial preconditioning by Big Ca2+-sensitive K+ channel opening requires superoxide radical generation.
P2860
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P2860
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
description
2000 nî lūn-bûn
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2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի օգոստոսին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
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2000年论文
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name
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@ast
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@en
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@nl
type
label
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@ast
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@en
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@nl
prefLabel
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@ast
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@en
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@nl
P356
P1476
Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen.
@en
P2093
P2888
P304
P356
10.1007/S003950070046
P577
2000-08-01T00:00:00Z
P5875
P6179
1019269266