Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.

Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.

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http://www.wikidata.org/entity/Q34048285
Generation of highly purified human cardiomyocytes from peripheral blood mononuclear cell-derived induced pluripotent stem cellsRole of sodium and calcium dysregulation in tachyarrhythmias in sudden cardiac death.Nonequilibrium reactivation of Na+ current drives early afterdepolarizations in mouse ventricleNeuronal Na+ Channels Are Integral Components of Pro-arrhythmic Na+/Ca2+ Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-adrenergic Stimulation.Cardiac arrhythmia in a mouse model of sodium channel SCN8A epileptic encephalopathyCalcium-dependent Nedd4-2 upregulation mediates degradation of the cardiac sodium channel Nav1.5: implications for heart failure.The role of Epac in the heart.Neuronal sodium channels: emerging components of the nano-machinery of cardiac calcium cycling.Intracellular calcium attenuates late current conducted by mutant human cardiac sodium channels.C-terminal phosphorylation of NaV1.5 impairs FGF13-dependent regulation of channel inactivation.Early effects of Epac depend on the fine-tuning of the sarcoplasmic reticulum Ca2+ handling in cardiomyocytes.Distribution of cardiac sodium channels in clusters potentiates ephaptic interactions in the intercalated disc.Reduction of SR Ca2+ leak and arrhythmogenic cellular correlates by SMP-114, a novel CaMKII inhibitor with oral bioavailability.Ranolazine antagonizes catecholamine-induced dysfunction in isolated cardiomyocytes, but lacks long-term therapeutic effects in vivo in a mouse model of hypertrophic cardiomyopathy.Late sodium current inhibitors to treat exercise-induced obstruction in hypertrophic cardiomyopathy: an in vitro study in human myocardium.Arrhythmogenic Mechanisms in Heart Failure: Linking β-Adrenergic Stimulation, Stretch, and CalciumAltered Ca and Na Homeostasis in Human Hypertrophic Cardiomyopathy: Implications for Arrhythmogenesis
P2860

Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.

Item
http://www.wikidata.org/entity/Q34048285
description
2014 nî lūn-bûn
@nan
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@ast
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@en
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@nl
type
label
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@ast
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@en
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@nl
prefLabel
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@ast
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@en
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P859
P932
P356
P1433
P1476
Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P859
P932