SOD2 protects against oxidation-induced apoptosis in mouse retinal pigment epithelium: implications for age-related macular degeneration. - Wikidata - awgldk - TriplyDB

SOD2 protects against oxidation-induced apoptosis in mouse retinal pigment epithelium: implications for age-related macular degeneration.

SOD2 protects against oxidation-induced apoptosis in mouse retinal pigment epithelium: implications for age-related macular degeneration.

http://www.wikidata.org/entity/Q34049463

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Lipid peroxidation: pathophysiological and pharmacological implications in the eye AMD Genetics in India: The Missing Links RPE necroptosis in response to oxidative stress and in AMD Mitochondrial function and redox control in the aging eye: role of MsrA and other repair systems in cataract and macular degenerations Association of superoxide anions with retinal pigment epithelial cell apoptosis induced by mononuclear phagocytes TGF-β1 induces senescence of bone marrow mesenchymal stem cells via increase of mitochondrial ROS production Validation of genome-wide association study (GWAS)-identified disease risk alleles with patient-specific stem cell lines.The effects of quercetin in cultured human RPE cells under oxidative stress and in Ccl2/Cx3cr1 double deficient mice.Enhanced HtrA2/Omi expression in oxidative injury to retinal pigment epithelial cells and murine models of neurodegeneration.The pathogenic role of the canonical Wnt pathway in age-related macular degeneration.Mitochondrial dysfunction in retinal diseases Inflammatory cytokines protect retinal pigment epithelial cells from oxidative stress-induced death.SOD2 contributes to anti-oxidative capacity in rabbit corneal endothelial cells.Confluent monolayers of cultured human fetal retinal pigment epithelium exhibit morphology and physiology of native tissue.An association between environmental factors and the IVS4+44C>A polymorphism of the DMT1 gene in age-related macular degeneration Research resource: nuclear receptor atlas of human retinal pigment epithelial cells: potential relevance to age-related macular degeneration miR-17-3p Exacerbates Oxidative Damage in Human Retinal Pigment Epithelial Cells.SOD2 protects neurons from injury in cell culture and animal models of diabetic neuropathy Peroxiredoxin 3 (PDRX3) is highly expressed in the primate retina especially in blue cones.Role of carnitine acetyl transferase in regulation of nitric oxide signaling in pulmonary arterial endothelial cells New era for personalized medicine: the diagnosis and management of age-related macular degeneration.Role of Nrf2 in retinal vascular development and the vaso-obliterative phase of oxygen-induced retinopathy Prevention of Oxidative Stress-Induced Retinal Pigment Epithelial Cell Death by the PPARgamma Agonists, 15-Deoxy-Delta 12, 14-Prostaglandin J(2).The effect of ranibizumab versus photodynamic therapy on DNA damage in patients with exudative macular degeneration.Coenzyme Q10 ameliorates oxidative stress and prevents mitochondrial alteration in ischemic retinal injury A case for neuroprotection in ophthalmology: developments in translational research.Dietary hyperglycemia, glycemic index and metabolic retinal diseases.Oxidation and age-related macular degeneration: insights from molecular biology.Consequences of oxidative stress in age-related macular degeneration.SOD3 Ameliorates Aβ25-35-Induced Oxidative Damage in SH-SY5Y Cells by Inhibiting the Mitochondrial Pathway.SOD3 Ameliorates H2O2-Induced Oxidative Damage in SH-SY5Y Cells by Inhibiting the Mitochondrial Pathway.The influence of sublethal blue light exposure on human RPE cells.Quantitative proteomic analysis of host responses triggered by Mycobacterium tuberculosis infection in human macrophage cells.Weikangning therapy in functional dyspepsia and the protective role of Nrf2.Association study of newly identified age-related macular degeneration susceptible loci SOD2, MBP, and C8orf42 in Han Chinese population.Protection of vascular smooth muscle cells by over-expressed methionine sulphoxide reductase A: role of intracellular localization and substrate availability.A Method for the Isolation and Culture of Adult Rat Retinal Pigment Epithelial (RPE) Cells to Study Retinal Diseases.Oxidative stress in dry age-related macular degeneration and exfoliation syndrome.

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P2860

SOD2 protects against oxidation-induced apoptosis in mouse retinal pigment epithelium: implications for age-related macular degeneration.

http://www.wikidata.org/entity/Q34049463

description

2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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SOD2 protects against oxidatio ...... -related macular degeneration.

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SOD2 protects against oxidatio ...... -related macular degeneration.

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SOD2 protects against oxidatio ...... -related macular degeneration.

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SOD2 protects against oxidatio ...... -related macular degeneration.

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SOD2 protects against oxidatio ...... -related macular degeneration.

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Investigative Ophthalmology & Visual Science

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SOD2 protects against oxidatio ...... -related macular degeneration.

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Emiko Kasahara

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Li-Ren Lin

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Venkat N Reddy

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Ye-Shih Ho

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P2860

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

Neuroprotectin D1: a docosahexaenoic acid-derived docosatriene protects human retinal pigment epithelial cells from oxidative stress

Oxidants, antioxidants, and the degenerative diseases of aging

Expression of metallothionein isoforms in human chorioretinal complex

Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

Effects of variation in superoxide dismutases (SOD) on oxidative stress and apoptosis in lens epithelium

Ocular pathology in mitochondrial superoxide dismutase (Sod2)-deficient mice

Increased mitochondrial oxidative stress in the Sod2 (+/-) mouse results in the age-related decline of mitochondrial function culminating in increased apoptosis

Manganese superoxide dismutase mediates the early release of mitochondrial cytochrome C and subsequent DNA fragmentation after permanent focal cerebral ischemia in mice

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