Inflammatory changes in the airways of mice caused by cigarette smoke exposure are only partially reversed after smoking cessation.
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Animal models of COPD: What do they tell us?Synergistic effect of bolus exposure to zinc oxide nanoparticles on bleomycin-induced secretion of pro-fibrotic cytokines without lasting fibrotic changes in murine lungsImpact of inflammation, emphysema, and smoking cessation on V/Q in mouse models of lung obstruction.Targeting prolyl endopeptidase with valproic acid as a potential modulator of neutrophilic inflammationOutcome of Smoking Cessation on Airway Remodeling and Pulmonary Inflammation in COPD PatientsIL-17A and serum amyloid A are elevated in a cigarette smoke cessation model associated with the persistence of pigmented macrophages, neutrophils and activated NK cellsCigarette smoke-induced lung emphysema in mice is associated with prolyl endopeptidase, an enzyme involved in collagen breakdown.Centrilobular emphysema combined with pulmonary fibrosis results in improved survival.Cigarette smoke and α,β-unsaturated aldehydes elicit VEGF release through the p38 MAPK pathway in human airway smooth muscle cells and lung fibroblasts.Effects of N-acetylcysteine in ozone-induced chronic obstructive pulmonary disease model.Cigarette smoke induces β2-integrin-dependent neutrophil migration across human endotheliumInflammation and pathological damage to the lungs of mice are only partially reversed following smoking cessation on subacute exposure to cigarette smoke.Cadmium-mediated toxicity of lung epithelia is enhanced through NF-κB-mediated transcriptional activation of the human zinc transporter ZIP8.Roflumilast inhibits the release of chemokines and TNF-α from human lung macrophages stimulated with lipopolysaccharide.Cigarette smoking behaviors and time since quitting are associated with differential DNA methylation across the human genome.Experimental progressive emphysema in BALB/cJ mice as a model for chronic alveolar destruction in humans.Probing Cellular and Molecular Mechanisms of Cigarette Smoke-Induced Immune Response in the Progression of Chronic Obstructive Pulmonary Disease Using Multiscale Network Modeling.Human adipose-derived stem cells ameliorate cigarette smoke-induced murine myelosuppression via secretion of TSG-6.Epigenetic biomarkers of T-cells in human glioma.Tobacco smoke induced COPD/emphysema in the animal model-are we all on the same page?Anti-Allergic Effect of Oroxylin A from Oroxylum indicum Using in vivo and in vitro Experiments.Pulmonary cytokine composition differs in the setting of alcohol use disorders and cigarette smokingEpigenomic association analysis identifies smoking-related DNA methylation sites in African Americans.Persistence of Th17/Tc17 cell expression upon smoking cessation in mice with cigarette smoke-induced emphysema.Everything prevents emphysema: are animal models of cigarette smoke-induced chronic obstructive pulmonary disease any use?Smoking and inflammation: evidence for a synergistic role in chronic disease.The matrikine PGP as a potential biomarker in COPD.Experimental animal models for COPD: a methodological review.Airway inflammation in chronic obstructive pulmonary disease (COPD): a true paradox.17β-estradiol dysregulates innate immune responses to Pseudomonas aeruginosa respiratory infection and is modulated by estrogen receptor antagonism.Compensatory lung growth after bilobectomy in emphysematous rats.Stimulated bronchial epithelial cells release bioactive lysophosphatidylcholine 16:0, 18:0, and 18:1.Measurement of airway function using invasive and non-invasive methods in mild and severe models for allergic airway inflammation in mice.Lung inflammatory effects, tumorigenesis, and emphysema development in a long-term inhalation study with cigarette mainstream smoke in mice.Activation of p38 mitogen-activated protein kinase in ovalbumin and ozone-induced mouse model of asthma.Animal models of cigarette smoke-induced chronic obstructive pulmonary disease.A comparison of fixation methods on lung morphology in a murine model of emphysema.Mesenchymal stem cells decrease lung inflammation during sepsis, acting through inhibition of the MAPK pathway.Increased zona pellucida thickness and meiotic spindle disruption in oocytes from cigarette smoking mice.Characterization of biochemical, functional and structural changes in mice respiratory organs chronically exposed to cigarette smoke.
P2860
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P2860
Inflammatory changes in the airways of mice caused by cigarette smoke exposure are only partially reversed after smoking cessation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Inflammatory changes in the ai ...... ersed after smoking cessation.
@ast
Inflammatory changes in the ai ...... ersed after smoking cessation.
@en
Inflammatory changes in the ai ...... ersed after smoking cessation.
@nl
type
label
Inflammatory changes in the ai ...... ersed after smoking cessation.
@ast
Inflammatory changes in the ai ...... ersed after smoking cessation.
@en
Inflammatory changes in the ai ...... ersed after smoking cessation.
@nl
prefLabel
Inflammatory changes in the ai ...... ersed after smoking cessation.
@ast
Inflammatory changes in the ai ...... ersed after smoking cessation.
@en
Inflammatory changes in the ai ...... ersed after smoking cessation.
@nl
P2093
P2860
P356
P1433
P1476
Inflammatory changes in the ai ...... ersed after smoking cessation.
@en
P2093
Aletta D Kraneveld
Frans P Nijkamp
Gert Folkerts
Paul A J Henricks
Saskia Braber
P2860
P2888
P356
10.1186/1465-9921-11-99
P577
2010-07-22T00:00:00Z
P5875
P6179
1039752873