about
Downregulation of the tumor-suppressor miR-16 via progestin-mediated oncogenic signaling contributes to breast cancer developmentSmall-molecule antagonists of Myc/Max dimerization inhibit Myc-induced transformation of chicken embryo fibroblastsDeregulation of scribble promotes mammary tumorigenesis and reveals a role for cell polarity in carcinomaRapamycin potentiates transforming growth factor beta-induced growth arrest in nontransformed, oncogene-transformed, and human cancer cells.There is no correlation between c-Myc mRNA expression and telomerase activity in human breast cancerC-myc, not HER-2/neu, can predict recurrence and mortality of patients with node-negative breast cancerKnockdown of c-Myc expression by RNAi inhibits MCF-7 breast tumor cells growth in vitro and in vivo.Correlation of expression of BP1, a homeobox gene, with estrogen receptor status in breast cancerc-myc, not her-2/neu, can predict the prognosis of breast cancer patients: how novel, how accurate, and how significant?The well-accepted notion that gene amplification contributes to increased expression still remains, after all these years, a reasonable but unproven assumptionTo Know How a Gene Works, We Need to Redefine It First but then, More Importantly, to Let the Cell Itself Decide How to Transcribe and Process Its RNAsWeaknesses and Pitfalls of Using Mice and Rats in Cancer Chemoprevention StudiesNodal signaling promotes a tumorigenic phenotype in human breast cancerThe other side of the coin: the tumor-suppressive aspect of oncogenes and the oncogenic aspect of tumor-suppressive genes, such as those along the CCND-CDK4/6-RB axisTIP30 interacts with an estrogen receptor alpha-interacting coactivator CIA and regulates c-myc transcriptionSALL4 is a new target in endometrial cancerGenomic alterations in breast cancer patients in betel quid and non betel quid chewersParabens and Human Epidermal Growth Factor Receptor Ligand Cross-Talk in Breast Cancer CellsThe epigenetic modifier JMJD6 is amplified in mammary tumors and cooperates with c-Myc to enhance cellular transformation, tumor progression, and metastasisInferring gene dependency network specific to phenotypic alteration based on gene expression data and clinical information of breast cancerIdentification of condition-specific regulatory modules through multi-level motif and mRNA expression analysis.Prognostic and predictive significance of MYC and KRAS alterations in breast cancer from women treated with neoadjuvant chemotherapy.Polymorphisms cMyc-N11S and p27-V109G and breast cancer risk and prognosis.Global regulation of nucleotide biosynthetic genes by c-MycThe knockdown of c-myc expression by RNAi inhibits cell proliferation in human colon cancer HT-29 cells in vitro and in vivo.Biphenyl C-cyclopropylalkylamides: New scaffolds for targeting estrogen receptor betaClinicopathologic and prognostic significance of c-MYC copy number gain in lung adenocarcinomas.Expression of cell cycle proteins in male breast carcinoma.Mitochondrial p32 protein is a critical regulator of tumor metabolism via maintenance of oxidative phosphorylation.Molecular characterizations of Nop16 in murine mammary tumors with varying levels of c-Myc.The Wilms' tumor suppressor WT1 induces estrogen-independent growth and anti-estrogen insensitivity in ER-positive breast cancer MCF7 cells.Effectiveness and molecular interactions of the clinically active mTORC1 inhibitor everolimus in combination with tamoxifen or letrozole in vitro and in vivo.Molecular genetics complexity impeding research progress in breast and ovarian cancers.MET and MYC cooperate in mammary tumorigenesis.c-MYC functions as a molecular switch to alter the response of human mammary epithelial cells to oncostatin M.Heterogeneity of osteosarcoma cell lines led to variable responses in reprogrammingAdaptation to hypoxia and acidosis in carcinogenesis and tumor progressionTargeted silencing of elongation factor 2 kinase suppresses growth and sensitizes tumors to doxorubicin in an orthotopic model of breast cancerCommon low-penetrance risk variants associated with breast cancer in Polish women.Atbf1 regulates pubertal mammary gland development likely by inhibiting the pro-proliferative function of estrogen-ER signaling.
P2860
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P2860
description
2000 nî lūn-bûn
@nan
2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
c-Myc in breast cancer.
@ast
c-Myc in breast cancer.
@en
c-Myc in breast cancer.
@nl
type
label
c-Myc in breast cancer.
@ast
c-Myc in breast cancer.
@en
c-Myc in breast cancer.
@nl
prefLabel
c-Myc in breast cancer.
@ast
c-Myc in breast cancer.
@en
c-Myc in breast cancer.
@nl
P356
P1476
c-Myc in breast cancer.
@en
P2093
P304
P356
10.1677/ERC.0.0070143
P577
2000-09-01T00:00:00Z