Synergistic interaction between CCK and leptin to regulate food intake.
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Chronic Oxytocin Administration as a Treatment Against Impaired Leptin Signaling or Leptin Resistance in ObesityGut-Brain Cross-Talk in Metabolic Control.The neurohormonal regulation of energy intake in relation to bariatric surgery for obesityChanges in neurohormonal gut peptides following bariatric surgery.Neuroanatomical characterisation of the expression of the lipodystrophy and motor-neuropathy gene Bscl2 in adult mouse brainLow-affinity CCK-A receptors are coexpressed with leptin receptors in rat nodose ganglia: implications for leptin as a regulator of short-term satietyPeptide signals regulating food intake and energy homeostasis.Sixteen years and counting: an update on leptin in energy balanceIntegrative capacity of the caudal brainstem in the control of food intake.Pancreatic signals controlling food intake; insulin, glucagon and amylinVisceral response to acute retrograde gastric electrical stimulation in healthy humanBrain Fos expression during 48 h after cisplatin treatment: neural pathways for acute and delayed visceral sickness.Functional compensation between cholecystokinin-1 and -2 receptors in murine paraventricular nucleus neurons.Mechanisms of CCK signaling from gut to brain.CCK-58 elicits both satiety and satiation in rats while CCK-8 elicits only satiation.Functional organization of neuronal and humoral signals regulating feeding behavior.Gastrointestinal hormones and polycystic ovary syndrome.Synergistic metabolic benefits of an exenatide analogue and cholecystokinin in diet-induced obese and leptin-deficient rodents.Renaissance of leptin for obesity therapy.Overnight food deprivation markedly attenuates hindbrain noradrenergic, glucagon-like peptide-1, and hypothalamic neural responses to exogenous cholecystokinin in male rats.Effects of leptin on cat intestinal vagal mechanoreceptors.Effects of leptin on cat intestinal motility.Comparison of the metabolic effects of sustained CCK1 receptor activation alone and in combination with upregulated leptin signalling in high-fat-fed mice.Effects of tumor necrosis factor α on leptin-sensitive intestinal vagal mechanoreceptors in the cat.Infant satiety depends on transient expression of cholecystokinin-1 receptors on ependymal cells lining the third ventricle in mice.Peripheral ghrelin participates in glucostatic feeding mechanisms and in the anorexigenic signalling mediated by CART and CRF neurons.Is there a role for gastric accommodation and satiety in asymptomatic obese people?Electrophysiological evidence for distinct vagal pathways mediating CCK-evoked motor effects in the proximal versus distal stomach.Coinjection of CCK and leptin reduces food intake via increased CART/TRH and reduced AMPK phosphorylation in the hypothalamus.
P2860
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P2860
Synergistic interaction between CCK and leptin to regulate food intake.
description
2000 nî lūn-bûn
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2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Synergistic interaction between CCK and leptin to regulate food intake.
@ast
Synergistic interaction between CCK and leptin to regulate food intake.
@en
Synergistic interaction between CCK and leptin to regulate food intake.
@nl
type
label
Synergistic interaction between CCK and leptin to regulate food intake.
@ast
Synergistic interaction between CCK and leptin to regulate food intake.
@en
Synergistic interaction between CCK and leptin to regulate food intake.
@nl
prefLabel
Synergistic interaction between CCK and leptin to regulate food intake.
@ast
Synergistic interaction between CCK and leptin to regulate food intake.
@en
Synergistic interaction between CCK and leptin to regulate food intake.
@nl
P2093
P1433
P1476
Synergistic interaction between CCK and leptin to regulate food intake.
@en
P2093
P356
10.1016/S0167-0115(00)00153-1
P50
P577
2000-08-01T00:00:00Z